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At least 361 records · Page 20Linked to original sources
PubMed1986

Somatosensory evoked potentials to muscle percussion in humans: upper extremity.

Cortical as well as presumably subcortical potentials were recorded from 20 normal subjects in response to mechanical percussion of the thenar eminence. Nuchal-to-frontal recordings detected an initial negative component at 21 msec, followed by positivities at 29 and 36 msec. C-prime to frontal recordings produced prominent positivities at 29 and 44 msec. Recordings over the peripheral nerve did not reveal consistent potentials. Control experiments indicate that potentials are initiated by fast-adapting muscle receptors. If additional control experiments continue to support these findings, yet another constituent of electrically evoked somatosensory potentials--which may prove useful in the definition of muscle innervation disorders--will have been identified.

Adolescent↗
PubMedDate not supplied

Distal, "Tinel-like" percussion paresthesiae, with proximal nerve lesions.

Can proximal nerve lesions cause a distal "Tinel-like" sign? We present the case studies of two patients with documented cervical root lesions who exhibited percussion paresthesiae distally in the absence of any clinical or nerve conduction evidence of distal pathology. The possible mechanisms underlying such manifestation, which has been previously reported in the literature, are explored, and a case is made for the phenomenon being related to the "distortion" of a normal sensory signal when it encounters an area of spatial inhomogeneity proximally along the nerve.

Adult↗
PubMed1996

Fluid-percussion brain injury adversely affects control of vascular tone during hemorrhagic shock.

To test the hypothesis that brain injury impairs control of vascular tone during compensation from hemorrhagic shock, Sprague-Dawley rats underwent fluid-percussion brain injury (or sham injury control) followed by a stepwise hemorrhage period to 1/2 baseline mean arterial pressure (1/2 MAP), a shock period holding at 1/2 MAP for 30 min, and a resuscitation period. Aortic blood flow (ABF) was measured and vascular conductance (ABF/MAP) was calculated. No differences occurred between groups during the stepwise hemorrhage period. During the 30 min shock period, controls decreased conductance from .2 +/- .07 to .16 +/- .04 and required repeated additional hemorrhage (3.4 +/- 1.3 cc) to maintain 1/2 MAP. In contrast, brain-injured animals increased conductance from .21 +/- .07 to .24 +/- .06 (p < .05) during the shock period and required repeated fluid replacements (3.0 +/- 1.3 cc lactated Ringer's (LR), p < .05) to maintain 1/2 MAP. Following resuscitation, conductance appropriately increased to .31 +/- .05 in controls but did not change (.25 +/- .04, p < .05) in brain-injured animals. We conclude that brain injury adversely affects control of vascular tone during shock and resuscitation in this model.

Animals↗
PubMed1997

[The development of a fluid percussion device for brain injury of animals].

This paper discusses a fluid percussion device for brain injury. The injury is produced by striking the cork with a pendulum dropped from a known fall height to produce transient high pressure through a fluid transfer system. Experimental results show the device has the advantages of making a controllable, repeatable, and precise injury, and it can produce different levels of injury on different animals.

Animals↗