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Treating the open bite.

Tongue thrust is involved in nearly all open bites. An open bite can be created by tongue thrust, tongue posture or mandibular posture. Tongue is an unusual muscle in that its contraction allows it to assume many shapes; its influence in the swallow can create an open bite in the area dictated by the contraction, thrust or rest position. Once space has been created between the upper and lower teeth by the tongue it continues to enter the space created, consequently enlarging the space. Skeletal open bites do not occur in patients whose masseter muscles are active during the swallow (an open bite can develop in these individuals due to habit or during orthodontic therapy). In these individuals the masseters fully contract during the swallow. The weaker the masseter muscles, the more likely an open bite will either be present or may develop during orthodontic treatment. Orthodontic movement will always result in occlusal interferences at some point in treatment. Occlusal interferences during orthodontic treatment make it difficult for patients to find a comfortable biting position. At this point they do not fully contract--"squeeze"--the masseters during the swallow to avoid traumatizing the teeth. Once the masseter squeeze is reduced, the tongue must contribute more to the swallow than when the masseters were more actively involved. The tongue becomes the cushion for the dentition during the swallow. The tongue now positions itself between the teeth during the swallow and the open bite during orthodontic treatment is born. Once the open bite occurs it is best to treat it immediately as the open bite thus created will only worsen with time. If the patient was originally a counterclockwise grower (brachycephalic), it is easier to correct. A clockwise grower (dolichocephalic) is hardest to correct, but can be corrected with patience, perseverance, exercise and a properly constructed tongue thrust appliance. While it is true that some skeletal configurations (i.e. clockwise growers, dolichocephalic facial types) lend themselves to the development of an open bite, the skeletal configuration itself seldom produces an open bite. The tongue is the main progenitor of open bites and it is also responsible for perpetuating the open bite. Dolichocephalic facial types lend themselves to open bite development as any orthodontic treatment which either distalizes molars or allows molars to extrude will tend to wedge the mandible and dentition open. (In this type of patient the masseter muscles develop insufficient force on closure to intrude the molars.) Once the bite opens, the tongue now enters the picture in order to complete the swallow. The open bite immediately worsens. The open bite thus created may require several months to correct. In the clockwise growth patient it is imperative that all precautions be taken during treatment to counteract open bite development. It is also imperative that an open bite, once detected, be treated immediately as the longer it persists the more difficult it becomes to treat. If left untreated, it becomes a habit and the more engrained a habit the more effort needed to change it.

Adolescent↗

Outcome prediction in open bite cases.

Some anterior open bites close spontaneously, others do not. It would be advantageous if the orthodontist had some means of predicting the spontaneous outcome in open bite patients. One-hundred-and-twenty-seven patients having open bite were recorded longitudinally for a minimum period of 3 years using cephalometric radiographs. The open bite closed spontaneously in 50 per cent of a prepuberal group aged between 7 and 10 years, in 49 per cent of a puberal group aged 11-14 years, and in 44 per cent of a post-puberal group aged between 14 and 21 years when first seen. Data from cephalometric analysis of the first film in each case was analysed using discriminant function analysis. The analysis correctly predicted the outcome in 88 per cent of the prepuberal group, in 74 per cent of the puberal group, and in 94 per cent of the post-puberal group. The accuracy of the prediction equation was checked in a new sample of 35 subjects with open bite observed over at least 2 years so that the spontaneous outcome was known. The equation correctly predicted the outcome in 79 per cent of prepuberal, in 80 per cent of puberal, and in 100 per cent of post-puberal patients in the second sample.

Adolescent↗

The anterior open bite problem (infraclusion).

Anterior open bite is a major orthodontic problem that is very difficult to treat (Fig. 1). It may not be the most frequent problem but it is a major functional problem. Quite often it is very difficult to correct an anterior open bite problem and even more difficult to treat in an adult after growth. Most of the time anterior open bite is a myofunctional problem related to a bad habit like thumb or finger sucking and/or is related to a breathing and swallowing problem. How can we understand the functional open bite problem? What are the available treatment options? This article will try to answer some of these questions.

Adolescent↗

Magnetic resonance images of the temporomandibular joints of patients with acquired open bite.

OBJECTIVES: Acquired anterior open bites were reported as the consequence of condylar collapse, which was associated with inflammatory TMJ disorders. However, we have seen such malocclusion patients whose condylar changes seemed to be related to TMJ degeneration associated with internal derangement. The aims of this study were to review the clinical history and to study the TMJ MRI of these patients. STUDY DESIGN: TMJ MRIs of patients, who had presented acquired anterior open bite at first visit, were retrieved from the image database for the analysis. Clinical histories focused on internal derangement were collected retrospectively. The soft tissue and hard tissue changes disclosed by MRI were also studied. RESULTS: All patients had experienced common signs/symptoms of TMJ internal derangement. All affected TMJs had anteriorly displaced disks and degenerative changes. Horizontally destructed condylar forms were seen significantly more frequently in these patients. CONCLUSION: TMJ degeneration associated with displaced disks might be a cause leading to the development of acquired anterior open bite.

Adolescent↗

Quantitative changes in the mRNA for contractile proteins and metabolic enzymes in masseter muscle of bite-opened rats.

To study the effects of bite opening on the fibre phenotypes of rat masseter, the mRNAs of four predominant myosin heavy-chain isoforms (MHC I, IIa, IId/x and IIb) and two alkali light-chain isoforms (LC1f and 3f) as well as those of two metabolic enzymes, carbonic anhydrase III (CAIII, oxidative enzyme) and glucose-phosphate isomerase (GPI, glycolytic enzyme), were measured in relation to the total RNA of masseter muscle by competitive, reverse transcriptase-polymerase chain reaction in control and bite-opened rats. Bite opening (2.8 mm increase in the vertical dimension for 1 week) significantly (P<0.05) increased the amount of MHC IIa mRNA but decreased (P<0.001) the amount of MHC IIb mRNA without changing the amount of MHC IId/x mRNA. No MHC I mRNA was found in any masseter studied. A significant (P<0.01) increase in the mRNA of LC1f associated with a decrease (P<0.05) in that of LC3f was observed after the bite opening. The CAIII mRNA increased significantly (P<0.001), while the GPI mRNA decreased (P<0.05) in association with the bite opening. These results strongly suggest that in 1 week of bite opening changes the rat masseter muscle from a glycolytic, MHC IIb-LC3f-dominant fibre to an oxidative, MHC IIa-LC1f-dominant fibre.

Adaptation, Physiological↗

An adolescent case of anterior open bite with masticatory muscle dysfunction.

In adolescence an open bite is likely to be induced by various causes, and most patients with open bite show masticatory muscle dysfunction. The orthodontic correction of an open bite may lead to the recovery of harmonious stomatognathic function. This article reports treatment for a 12-year and 10-month-old female patient with an anterior open bite. She complained of occlusal disturbances due to an anterior open bite. Before the eruption of the upper second molars, she recognized the presence of occlusal contacts between the upper and lower anterior teeth. Before treatment, the activity of the masticatory muscles was relatively low during clenching. An upper lingual arch with an accessory spring to eliminate the premature contact and to improve the anterior open bite plus conventional fixed appliances for tooth alignment was selected as the treatment plan. Immediately after improvement of the open bite, the muscle activity became slightly greater than that at the initial stage. The total treatment time was 13 months. After treatment, an acceptable occlusion was achieved, the masticatory muscles were working harmoniously, and their activity was much higher than that at the initial stage. After a 2-year retention period, an acceptable occlusion was maintained without recurrence of the open bite, indicating a long-term stability of occlusion. The result of this treatment indicated that correction of open bite is of great importance for prevention of skeletal impairment and recovery of harmonious stomatognathic function lost by an unstable occlusion.

Adolescent↗

[Open bite and interdental sigmatism].

The authors revealed very often open bite in preschool children with interdental sigmatism and vice versa. They investigated the effect of interdental sigmatism on the persistence of open bite. According to the authors findings open bite persists in 3-5 year-old children with open bite and interdental sigmatism after a 10-to-22 month follow-up significantly more often than in children of similar age with open bite but without interdental sigmatism. Interdental sigmatism is associated with already existing open bite. It is possible that at the time of their development open bite and interdental sigmatism have no causal relationship and the two defects associate accidentally. Only during subsequent development open bite persists more frequently in those children who suffer at the same time from interdental sigmatism, while in children without interdental sigmatism the open bite improves more often.

Child, Preschool↗

[Morphological study on open bite patients].

The purpose of the present study is to investigate the morphological features of open bite patients who have also disharmony of anteroposterior relations between maxilla and mandible. Lateral head films of 40 adult females with open bite were used. The samples were classified into three groups by ANB angle. Three groups were as follows. 1. Type I open bite (12 subjects, 1 degree less than or equal to ANB less than or equal to 4.5 degrees). 2. Type II open bite (13 subjects, ANB less than 4.5 degrees). 3. Type III open bite (15 subjects, ANB less than 1 degree) The normal samples was the longitudinal data of females in our department. Skeletal and dental variables were calculated and compared norm versus type I open bite, type I open bite versus type II open bite and type I open bite versus type III open bite, respectively. The following results were obtained. 1. Disharmony of the vertical dimensions in type I open bite was mainly due to long anterior lower facial height which was accompanied by large gonial angle as compared with norm. 2. There were no difference in anteroposterior position of the maxilla and nasal floor in all three open bite groups. 3. There were no difference in anterior and posterior facial height between type I and type II open bite. However, type II open bite showed the backward rotation of the mandible accompanied by over eruption of upper first molars. 4. Type III open bite showed longer anterior lower facial height which was caused by excessive mandibular growth than type I open bite. 5. Upper incisors showed labial inclination as compared with norm in all three open bite groups. However, lower incisors showed labial inclination in type II open bite and lingual inclination in type III open bite as compared with type I open bite. As a result, the inter-incisal angle was small in type II open bite and large in type III open bite as compared with type I open bite. These results suggested that the dentoalveolar factors were large in type II open bite and the skeletal factors were large in type III open bite as compared with type I open bite.

Cephalometry↗

Bilateral open bite in dicygotic twins. A combined orthodontic-prosthetic approach.

CASE REPORT: Dizygotic twins, male, 25 years of age, required treatment for an identical orthodontic diagnosis. DIAGNOSIS: Class III malocclusion with mesial molar relation and frontal edge-to-edge bite, lyrate upper dental arch, grouped cross-bite and bilateral open bite in the molar and bicuspid region, retention and lingual inclination respectively of the lower left second bicuspid, mesial inclination of both lower first molars. The severity of the malocclusion differed in the two brothers. THERAPY: Orthodontic treatment was successful concerning the transversal expansion and alignment of the maxillary dental arch, the functional relation of the anterior teeth, the transversally correct relation of the upper and lower dental arches and, following surgical removal of the lower second bicuspids, the reduction of crowding in the lower arch. An attempt was made to upright the molars in the mandibular arch and to close the lateral open bite by means of vertical elastics. However, the 10-month period of resistance to the therapy suggested, after a tongue protrusion habit had been ruled out, a diagnosis of ankylosis. Further orthodontic treatment was renounced and a prosthetic solution was pursued instead: the teeth in infraocclusion were treated with full ceramic overlays and, in the regions with residual gaps, with pontics (Empress II, Ivoclar, Schaan, Liechtenstein), after minimally invasive preparation (confined to removal of existing fillings). CONCLUSION: This case is particularly interesting because the infrapositioned molars in both brothers were very likely due to ankylosis, suggesting a genetic cause.

Adult↗

Vertical proportions: a guide for prognosis and treatment in anterior open-bite.

1. Most open-bites are deficient in growth of the upper face and in growth of the posterior face. However, some varieties have adequate posterior face height. A compensatory increase in lower anterior face height occurs in both types. 2. The ratio of upper anterior face height to lower anterior face height (UAFH/LAFH) is suggested as an aid in determining the prognosis for the orthodontic correction of anterior open-bite. 3. The treatments of eight patients were presented to illustrate this concept. The results were critically analyzed. 4. The validity of this index can be determined only after prolonged use by many orthodontists.

Adolescent↗

Nonsurgical management of the anterior open bite: a review of the options.

The open bite malocclusion has been described as being of 2 types: dental and skeletal. Proper differentiation is essential in determining the appropriate corrective measures. Dental open bites are generally more responsive to treatment with orthodontics alone, whereas skeletal open bites often require a combination of orthodontics and orthognathic surgery. Patient selection and treatment principles for nonsurgical open bite treatment are discussed, and a review of various methods of treatment for the skeletal open bite is presented. Posttreatment stability and retention concerns are addressed.

Adult↗

Skeletal morphologic features of anterior open bite.

Lateral cephalograms of sixty patients with open bite and sixty untreated subjects who were determined clinically to have Class I normal occlusions were studied. The following six values were recorded: (1) ratio of posterior to anterior face height, (2) ratio of upper to lower face height, (3) Sn GoGn angle, (4) gonial angle, (5) SN-PP angle, and (6) PP-GoGn angle. Results were compared with previous studies. The constancy of the ratios and angles with age was tested and an attempt was made to separate the open-bite sample into skeletal and dentoalveolar groups. Results were generally consistent with those reported previously, except for the Sn PP angle, which was not significantly different from the normal group. In the open-bite sample the study showed a decrease in the ratio of PFH/AFH, a decrease in the ratio of UFH/LFH, and an increase in the other angles measured. Upon further division of the sample into mixed- and permanent-dentition groups, it was found that the angles and ratios remained relatively constant with age. An attempt to divide the open-bite sample into skeletal and dentoalveolar groups resulted in more extreme values for all the measurements made. The difficulty in differentiating between the two groups and the variability of dentoskeletal patterns in open bite is discussed.

Adult↗

[Anterior open bite: a formula for the differential diagnosis].

BACKGROUND: The differentiation between dental and skeletal anterior open bite, both with distinct etiologies and characteristics, is necessary to establish adequate orthodontic and speech-language treatment procedures. AIM: To establish a formula for the differential diagnosis of dental anterior open bite and skeletal anterior open bite based on a cephalometric analysis of the angles that measure the mandibular plane inclination (NS.GoGn), the vertical and anteroposterior mandibular growth (NSGn), and the Facial Axis angle. METHOD: 78 lateral radiographies, of individuals with ages varying from 6 to 13 years, were analyzed and divided into two groups: dental anterior open bite and skeletal anterior open bite. RESULTS: There was a significant difference between the groups regarding the measurements of the angles: for the group with skeletal anterior open bite the NS.GoGn and the NSGn angles were bigger and the Facial Axis angle was smaller when compared to the group with dental anterior open bite. The adjustment to the linear function resulted in a formula that determines the type of anterior open bite with only 3.5% of error. CONCLUSIONS: It was possible to establish a high precision formula for the differential diagnosis between dental and skeletal open bite, based on the angle measurements of NS.GoGn NSGn and Facial Axes. The presented formula for the differential diagnosis has research and clinical applicability.

Age Distribution↗