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At least 37 records · Page 2Linked to original sources
PubMed2002

Efficacy of mastoid cortex plasty for middle ear aeration in intact canal wall tympanoplasty for cholesteatoma.

OBJECTIVE: To determine the effect of closing the bone defect of the mastoid cortex using bone pate after mastoidectomy (mastoid cortex plasty) during the first-stage operation for the restoration of the mastoid cavity aeration. STUDY DESIGN: This was a prospective study. SETTING: Tertiary medical center. PATIENTS: Thirty-five patients with cholesteatoma invading the mastoid cavity. INTERVENTION: Seventeen patients received mastoid cortex plasty with scutum plasty and insertion of a Silastic sheet after removal of the cholesteatoma using a combined approach during the first-stage operation. The 18 control patients received only scutum plasty with the insertion of a Silastic sheet without mastoid cortex plasty during the first-stage operation. MAIN OUTCOME MEASURES: The restoration of the middle ear aeration was assessed with high-resolution computed tomography before both the first-stage operation and the second-stage operation. RESULTS: The range of middle ear aeration ameliorated significantly whether or not mastoid cortex plasty was performed. Scutum plasty with the insertion of a Silastic sheet without mastoid cortex plasty was significantly effective only for the amelioration of the epitympanum aeration. Mastoid cortex plasty with scutum plasty with the insertion of a Silastic sheet was significantly effective for the amelioration of the epitympanum and also the mastoid cavity aeration. In both groups, before the first-stage operation, approximately 70% of the patients had good mesotympanum aeration, and the mesotympanum aeration ameliorated further after the first-stage operation, although this amelioration was not significant. CONCLUSIONS: Mastoid cortex plasty is statistically effective for the amelioration of mastoid cavity aeration after the first-stage operation.

Adolescent↗
PubMed2002

Mastoid cortex plasty using bone pate.

OBJECTIVE: To describe the surgical technique of mastoid cortex plasty using bone pate and its clinical significance. STUDY DESIGN: Retrospective. SETTING: Tertiary medical center. METHODS: Bone pate collected while drilling the mastoid cortex was mixed with fibrin glue to make a pate plate. The mastoid cortex defect was closed with a pate plate cut to the desired shape and size. PATIENTS AND MAIN OUTCOME MEASURES: Mastoid cortex plasty was included in the first-stage operation in 49 patients who underwent staged tympanoplasty. The second-stage operation was an inspection to see whether the mastoid cortex was reconstructed and the ingrowth of fibrous soft tissue was prevented. RESULTS: In 35 patients, the mastoid cortex was reconstructed completely, and soft tissue ingrowth into the mastoid cavity was blocked completely. In the remaining 14 patients, a defect of varying size occurred at the site of drainage-tube insertion. This defect was small enough to prevent soft tissue ingrowth into the mastoid cavity. CONCLUSION: Mastoid cortex plasty is a simple and rapid procedure for preventing postoperative deformity of the mastoid process and is valuable for restoring mastoid cavity function.

Adolescent↗
PubMed2003

Acute mastoiditis in Southern Israel: a twelve year retrospective study (1990 through 2001).

BACKGROUND: Acute mastoiditis is a serious complication of acute otitis media (AOM) and has been increasingly reported in the last decade. OBJECTIVES: To report the experience accumulated with acute mastoiditis at the Soroka University Medical Center, Beer-Sheva, Israel, in a period of increasing antimicrobial resistance with Streptococcus pneumoniae. PATIENTS AND METHODS: We reviewed the records of all children with acute mastoiditis hospitalized from 1990 through 2001. Acute mastoiditis was diagnosed when one or more of the physical signs of mastoiditis (swelling, erythema, tenderness of the retroauricular area and anteroinferior displacement of the auricle) were diagnosed in the presence of concomitant or recent (< or =4 weeks) AOM. RESULTS: One hundred sixteen episodes of acute mastoiditis occurred in 101 children age 2 months to 14 years (median, 25 months; 19% <1 year old). The average yearly incidence was 6.1 cases per 100 000 population <14 years old, with a significant increase in the number of cases during the study period. Acute mastoiditis was the first evidence of AOM in 10 (10%) patients. Fever >38 degrees C and >15,000 WBC/mm3 were present in 67 and 43% of cases, respectively. Irritability, retroauricular swelling, redness and protrusion of the auricle occurred more commonly in patients <3 years old (79, 90, 84 and 76% vs. 28, 42, 45 and 30%, respectively, in patients > or =3 years old; P < 0.002). Computed tomography scans were performed in 54 of 116 (47%) cases and revealed bone destruction in 38 (70%). Periosteal abscess and lateral sinus vein thrombosis were diagnosed in 8 and 2 patients, respectively. Simple mastoidectomy was done in 32 of 116 (28%) cases, after no response to intravenous antibiotics was observed; ventilation tubes were inserted in 12 patients. Cultures were obtained at admission in 83 (72%) episodes (71 by tympanocentesis and 12 from ear discharges). Overall 43 pathogens were isolated: 34 at admission; 14 at surgery; and 5 at both occasions. The most commonly isolated organisms were S. pneumoniae (14 of 43, 33%; 4 penicillin-nonsusceptible), Streptococcus pyogenes (11 of 43, 26%), nontypable Haemophilus influenzae (6 of 43, 14%), Pseudomonas aeruginosa (5 of 43, 12%) and Escherichia coli (4 of 43, 9%). All S. pneumoniae organisms were isolated between 1996 and 2001. CONCLUSIONS: (1) The incidence of acute mastoiditis in children in Southern Israel is greater than that reported in the literature; (2) a significant increase in the number of cases was recorded during the study period; (3) the clinical picture of acute mastoiditis was more severe in infants and young children; (4) the pathogen distribution in acute mastoiditis differs from that of AOM with significantly higher rates of S. pyogenes and lower rates of nontypable H. influenzae recovery; (5) penicillin-nonsusceptible S. pneumoniae played only a minor role in the etiology of acute mastoiditis in Southern Israel.

Acute Disease↗
PubMed2003

[Temporal bone study of residual mastoid inflammation in ears with normally appearing tympanic membrane].

We examined 395 temporal bones with an intact tympanic membrane to explore the relationship between residual inflammation in the middle ear cavity and development of mastoid pneumatization. Histopathological changes were studied in the middle ear cavity. Mastoid pneumatization was classified as good or poor based on the extent of mastoid tip development to the lateral semicircular canal. Specimens were 344 temporal bones with well-pneumatized mastoid and 51 with poorly-pneumatized mastoid. Otitis media was noted in 119 (34.6%) bones in the good group and 9 (17.6%) in the poor group. In well-pneumatized mastoid, chronic inflammatory changes were frequently observed at the lower portion of mastoid cells, the round window niche, and the tympanic sinus. In contrast, no such incidence of inflammatory change was noted in poorly-pneumatized mastoid. Our findings indicate that an intact tympanic membrane does not always mean freedom from mastoid inflammation, especially when the mastoid is well-pneumatized. This makes it important to check for possible remaining otitis media in patients with a well-pneumatized mastoid, even if the tympanic membrane appears normal.

Ear, Middle↗
PubMed1995

Mastoiditis in Kelantan.

Chronic mastoiditis and subperiosteal abscess are rarely seen nowadays in most countries. Thirty-four cases of mastoiditis were reviewed to find out the prevalence of chronic mastoiditis in the east coast of Malaysia. Twenty (58.82%) of these cases were a sequelae of chronic suppurative otitis media with cholesteatoma. All the patients with chronic mastoiditis were more than six years old. Forty-five percent of cases with chronic mastoiditis had a well pneumatized mastoid air cell on the unaffected side. The occurrence of chronic mastoiditis or cholesteatoma in a well pneumatized mastoid is not really as rare as was thought to be. X-ray of the mastoids is very useful in diagnosing patients with chronic mastoiditis and cholesteatoma. Mastoiditis is uncommon in adults and whenever a case is seen, an underlying pathology like cholesteatoma should be suspected.

Adolescent↗
PubMed1997

Evaluation of mastoid air cell system by three-dimensional reconstruction using sagittal tomography of the temporal bone.

The mastoid air cell system has been recognized as an important contributor to the pathophysiology of middle ear inflammatory diseases. Various methods of temporal bone imaging have been designed to investigate the correlation between middle ear disease and mastoid pneumatization. In this study, the mastoid air cell system was reconstructed three-dimensionally from sagittal tomographic images of the temporal bone on X-ray films, using a personal computer to evaluate the mastoid pneumatization in a total of 29 patients with chronic otitis media, adhesive otitis media, adhesive-type cholesteatoma, attic cholesteatoma and cholesterol granuloma, and in five normal subjects as controls. Reconstructed three-dimensional images of the mastoid air cell system and its volume were analyzed. The reconstructed images were helpful in recognizing the three-dimensional solid appearance of the mastoid air cell system. The volume of the reconstructed mastoid air cell system was significantly reduced compared with that in the controls in each of the patient groups. Mastoid pneumatization in the patients with adhesive-type cholesteatoma was significantly suppressed compared with that in the adhesive otitis media patients. Interestingly, the adhesive otitis media group showed cell development at the tip of mastoid process, whereas the group of adhesive-type cholesteatoma did not, suggesting a difference in the pathophysiology in the two diseases. We found that three-dimensional reconstruction of the temporal bone using sagittal tomographic images was useful in evaluating the state of mastoid air cell system development in individual cases and in investigating the pathophysiology in middle ear disease.

Adolescent↗
PubMed1997

Gas exchange function through the mastoid mucosa in ears after surgery.

Gas exchange function through the mastoid mucosa was investigated in ears after surgery using nitrous oxide. Increase in the mastoid pressure was assessed by a micropressure sensor placed in the mastoid cavity during the second-stage revision operation performed under general anesthesia using 67% nitrous oxide, 33% oxygen, and sevoflurane on 14 ears with chronic adhesive otitis media or cholesteatoma as well as on seven ears without inflammation as controls. All seven control ears showed pressure increase in the mastoid in various degrees. In the 14 postoperative ears, nine of the 10 ears on which the mastoid mucosa had previously been able to be preserved in various degrees showed pressure increase in the mastoid, but none of the remaining four ears, which had previously had mastoidectomy, showed any pressure increase. The presence or absence of the mastoid pressure increase of those ears was also found to be correlated well with the presence or absence of mastoid aeration on computed tomography examined just before the second-stage operation. These results appear to indicate that, in ears after surgery, recovery of both the gas exchange function and aeration in the mastoid is expected only when the mastoid mucosa can be preserved even partially.

Adolescent↗
PubMed2003

Mastoid and tympanic membrane as pressure buffers: a quantitative study in a middle ear cleft model.

HYPOTHESIS: The tympanic membrane (TM) and mastoid air cells are measurable pressure buffers of the middle ear (ME). BACKGROUND: Pressure homeostasis of the ME is maintained approximately atmospheric by mechanisms that neutralize (buffer) pressure fluctuations, two of which are the TM and mastoid. MATERIALS AND METHOD: Negative pressures were induced by volume changes in an artificial ME model. Those were recorded directly while using a rigid or a flexible TM with "mastoids" of various sizes. RESULTS: In the rigid TM model, the volume changes correlated linearly with the induced pressures and were confirmed to fit Boyle's law. In the flexible TM model, the pressure/volume correlation was nonlinear up to -50 mmH2O, where the TM was maximally displaced (approximately 25 mm3), became rigid, and constituted 75%, 41%, and 33% of the buffering gained in tandem with the "mastoid" in a model having a "mastoid" of 0, 5, and 10 mL, respectively. Altogether, a large "mastoid" required a greater volume change than a small one to induce the same pressure. CONCLUSIONS: The mastoid air volume "dilutes" pressure changes relatively to its size: the volume change required to alter a given pressure in an average (6 mL) mastoid is six-fold that which is needed in a small (1 mL) mastoid. ME volume reduction by TM retraction buffer negative ME pressures. This maximal ME volume change is constant for a "normal" TM. Therefore, it is the ME with the small mastoid that is most vulnerable to pressure changes and may develop compensatory buffering mechanisms, e.g., additional TM retraction (atelectasis) and/or ME volume reduction by fluid accumulation.

Acoustic Impedance Tests↗
PubMed1984

Role of the mastoid in tympanic membrane reconstruction.

Otologists have long debated the importance of the mastoid in determining the success or failure of tympanic membrane reconstruction. The pneumatic spaces within the mastoid represent an "air reservoir" which can be drawn upon during periods of eustachian tube dysfunction and buffer the middle ear against the development of detrimental negative pressures. Mastoid inflammatory disease, if untreated, may result in recurrent suppuration and graft failure. Small mastoid volume, aside from its well known association with chronic infectious middle ear disease, has been shown to effect adversely graft survival following myringoplasty. In 48 patients undergoing myringoplasty with simple mastoidectomy, neither small mastoid size nor inflammatory mastoid disease significantly decreased the rate of graft healing. This suggests that simple mastoidectomy is an effective means of repneumatizing the mastoid and eradicating mastoid sources of infection. The successful surgical creation of a pneumatized mastoid cavity in communication with the middle ear was confirmed by postoperative computerized tomographic (CT) scans. In failed cases, CT scanning predictably identified residual mastoid disease. Simple mastoidectomy is considered to be a safe and useful adjunct to myringoplasty in selected cases of chronic otitis media with perforation.

Hearing↗
PubMed2001

[Pneumatization of the mastoid process in medieval and contemporary skulls in light of radiologic and anthropometric measurements].

Pneumatization and morphogenesis of the temporal mastoid process take place concurrently and exhibit noticeable variability. The mastoid process is phylogenetically the youngest part of the skull. The aim of this work was to examine the pneumatization of the mastoid process in mediaeval and contemporary skulls using radiological and anthropometric methods. 85 male skulls from the second half of the 20th century were compared with 102 male and 50 female skulls from the Middle Ages. Bilateral X-ray images of mastoid processes according to Schuller were obtained and anthropometric measurements were done. Planimetry was used to obtain the air cell area of the mastoid processes. Correlations between several parameters of the temporal bone, facial bones, cranial bones and pneumatization of the mastoid process were studied. X-ray images revealed four types of the mastoid process: pneumatic, mixed, sclerotic, and apneumatic. The pneumatic mastoid process prevailed in every group of skulls. The mixed type was the least frequent in mediaeval female skulls and most frequent in contemporary male skulls. The apneumatic type was the rarest one in all groups of skulls. The largest surface area of mastoid air cell system was found in contemporary male and the smallest in mediaeval female skulls (Fig. 5). An asymmetry between left and right processes was observed in mediaeval and contemporary skulls, being more evident in the latter. No correlation was found between anthropometric parameters of the skull and the extent of pneumatization of the mastoid process.

Adult↗
PubMed1998

Middle ear pressure variations during 50% N2O anesthesia as a function of mastoid pneumatization.

HYPOTHESIS: Mastoid size is a factor in middle ear (ME) pressure regulation. BACKGROUND: In a study investigating ME pressure variations during nitrous oxide (N2O) anesthesia, particularly high values of ME pressure increase rate (PIR) were observed in four patients with sclerotic mastoids. The current study is aimed at systematically assessing this observation. METHODS: Middle ear pressure was measured periodically in 30 patients during 50% N2O anesthesia using tympanometry. For each patient, a curve representing ME pressure during anesthesia was plotted. From the curve steepness, the PIR was calculated. Extent of mastoid pneumatization was assessed planimetrically using mastoid x-rays. Ears then were divided by the median into two groups: ears with small mastoids and ears with large mastoids. The difference between the mean PIR of both ear groups was statistically analyzed. RESULTS: A significant difference between the PIR was found among the two groups. In ears with mastoids smaller than 9.475 cm2 (the median), the PIR was significantly higher than in ears with mastoids > 9.475 cm2. CONCLUSIONS: These findings support the concept that the mastoid has a ME pressure buffering capability: the larger its volume, the better its buffering capability. It is therefore suggested, that the mastoid plays a role in ME pressure regulation.

Acoustic Impedance Tests↗
PubMed2002

Mastoid air sinus abnormalities associated with lateral venous sinus thrombosis: cause or consequence?

BACKGROUND: Mastoiditis is a known cause of lateral venous sinus thrombosis (LST). We have encountered patients with LST associated with mastoid abnormality on MRI without any clinical signs of infection; the significance of these abnormalities is uncertain. This study examines the relationship of LST and mastoid air sinus abnormalities systematically. SUMMARY OF REPORT: We performed a retrospective clinical and radiological review of a series of 26 patients with cerebral venous thrombosis. Mastoid abnormalities were detected ipsilateral to 9 of 23 thrombosed lateral sinuses (39%) and 0 of 29 unaffected lateral sinuses (P<0.001). No patient had clinical evidence of mastoiditis. Eight of 9 patients with mastoid abnormalities were treated without antibiotics; all made uneventful clinical recoveries. Repeated MRI in 1 patient revealed reversal of the mastoid changes. CONCLUSIONS: The mastoid changes observed are likely to be due to venous congestion as a consequence of LST, not mastoiditis.

Adolescent↗
PubMed2003

Assessment of mastoid air cell size versus sigmoid sinus variables with a tomography-assisted digital image processing program and morphometry.

We assessed the mastoid air cell size and variables of the sigmoid sinus in healthy ears and ears with chronic otitis media (COM). Thirty-eight patients with unilateral COM [15 with cholesteatoma (COM/+) and 23 without cholesteatoma (COM/-)], and 20 subjects with healthy ears, were included in the study. Assessment was performed using a quantitative digital image processing computed tomography (CT) program, and the volume of the mastoid bone was measured using the morphometric method of Cavalieri. In both COM/+ and COM/- patients the sigmoid to suprameatal spine distance and mastoid size were greater on the healthy side than on the diseased side ( p<0.05). The distance and area were significantly greater in the healthy control subjects than in either the healthy or the diseased ears of the patients with COM ( p<0.05). In the healthy ears of COM patients, there was significant correlation between the sigmoid to suprameatal spine distance and air cell size and mastoid volume ( p<0.05). In the diseased ears of COM patients, this correlation was absent ( p>0.05). The sigmoid sinus shape was of the half-moon type (62%), protrusive type (22%) and saucer type (16%). The digital image processing CT program allowed us to estimate the individual area of the air and soft tissue filled mastoid air cells. The mastoid size in both intact and disease ears of COM patients was smaller than in the healthy controls. The mastoid size may be determined genetically. However, environmental factors such as infection may also affect the mastoid size. Therefore, both genetic and environmental factors may be related to COM as far as the size of the mastoid air cells is concerned.

Adult↗
PubMed2003

Acute mastoiditis in children: Pseudomonas aeruginosa as a leading pathogen.

OBJECTIVE: Acute mastoiditis is a serious bacterial infection of the temporal bone and is the most common complication of otitis media. The goal of this study is to assess the clinical features, pathogens, management, and outcome of acute mastoiditis in children in northern Israel. METHODS: A systematic review of medical records of all children who were admitted with acute mastoiditis from January 1990 through December 2000. RESULTS: Fifty-seven children were included. Median was age: 36 months. In 26 patients (45.6%) mastoiditis complicated the first episode of acute otitis media (AOM). Twenty-five children (44%) received antibiotic treatment prior to admission. Frequent symptoms included mastoid area erythema in 54 children (94.7%), proptosis of the auricle in 52 children (91.2%) and fever in 43 children (75.4%). Middle ear, and subperiostal culture yielded growth of pathogen in 30 children (75%), two cultures yielded more then one pathogen. The most frequent pathogens were: Pseudomonas aeruginosa in 10 children (25%), Streptococcus pneumoniae in eight children (20%), Group A streptococcus in six children (15%). The highest incidence of Streptococcus pneumoniae was found in children who did not suffer from AOM before admission (35 vs. 5%). Fifty-two (91.2%) children were cured with antibiotic treatment alone. Seventeen children underwent computed tomography (CT) of the mastoid. Mastoid bone destruction was demonstrated in six children and subperiostal abscess in eight. Mastoidectomy was performed in five children. CONCLUSIONS: The diagnosis of acute mastoiditis can be made on clinical basis alone requiring CT only when complications are suspected. Half of the children admitted with acute mastoiditis had no previous history of recurrent AOM. In those children S. pneumoniae was the leading pathogen while P. aeruginosa was more prevalent in children with recurrent AOM. Most of the children recovered with medical therapy alone, without surgical intervention.

Acute Disease↗
PubMed2000

Pneumococcal mastoiditis in children.

OBJECTIVE: To determine the impact of antibiotic resistance on the frequency, clinical features, and management/outcome of mastoiditis attributable to Streptococcus pneumoniae. DESIGN: Retrospective review of the medical records of children with mastoiditis caused by S pneumoniae from September 1993 through December 1998. PATIENTS: Infants and children with pneumococcal mastoiditis cared for at 8 children's hospitals in the United States. RESULTS: Thirty-four children with pneumococcal mastoiditis were identified. The median age of the children was 12 months (range: 2 months-12.5 years); 28 (82%) were </=2 years old. Six children had recurrent otitis media. A subperiosteal abscess was noted in 13 children (37%). The mastoids were abnormal in all 25 patients on whom computed tomography was performed. There was no trend toward increasing numbers of cases per year despite increasing proportions of pneumococcal isolates, which were nonsusceptible to penicillin. Serogroup 19 accounted for 57% of isolates, serogroup 23 for 14.3% of isolates, and serotype 3 for 10. 7% of isolates. Except for receipt of less antibiotic therapy in the previous 30 days, children with penicillin-susceptible isolates had similar demographic features and clinical findings and surgical treatment as did children whose isolates were nonsusceptible to penicillin. CONCLUSIONS: Pneumococcal mastoiditis occurs primarily in children <2 years of age and usually is not associated with a history of recurrent otitis media. The number of cases of mastoiditis caused by S pneumoniae occurring among 8 children's hospitals has remained stable despite increasing rates of antibiotic-resistant S pneumoniae. Serogroup 19 is the leading serogroup associated with pneumococcal mastoiditis. Streptococcus pneumoniae, mastoiditis, serotypes, resistance.

Cephalosporin Resistance↗
PubMed1982

[Area and topography of the air cells of the mastoid and temporal squama in persons of different ages].

Age changeability of the area and topography of the mastoid air cells and squama temporalis projections have been studied in 276 skull roentgenograms of human beings at the age of 1-78 years to estimate pneumatization areas; common planimetric techniques have been applied. The quantitative material obtained have been treated biometrically. In order to study topography of the mastoid air cells projections on th mastoid process, the trepanation triangle contrasted prior to roentgenography has served as the reference point. Pneumatization of the mastoid process and the squama temporalis has been stated to occur in 85-90% of people regardless of their age. In children before 7 years of age, the pneumatization process proceeds slowly. The projection area of the mastoid air cells and squama temporalis in children at the age of 1 year, 2-3 and 4-7 years has no statistically significant differences. The mastoid process pneumatization is intensive in children at the age of 8-13 years, that of squama temporalis--during the same age interval and in adolescence. In all age groups the projection area of the mastoid air cells demonstrates great individual variations. There are not statistically significant differences between average values of the projection area of the mastoid air cells on the left and right sides in all age groups studied. Projection of the mastoid air cells on the trepanation triangle has certain age variations. In children at the age of 8-13 years, it does not significantly differ from that in mature persons.

Adolescent↗
PubMed1989

Acute and latent mastoiditis.

The diagnosis 'acute' mastoiditis is not an unambiguous entity. It contains both 'classical' and 'latent' mastoiditis. 'Classical' mastoiditis was often seen before the antibiotic era, had serious complications and was cured by mastoidectomy. After the introduction of antibiotics, the number of cases of 'classical' mastoiditis decreased and was replaced by a more prolonged condition called 'latent' mastoiditis. Lately in Oslo, we have seen an increase in numbers of 'classical' mastoiditis and at the same time a decrease in the incidence of 'latent' mastoiditis. A four year study of patients with 'classical' mastoiditis is described.

Acute Disease↗
PubMed1999

The Yung percutaneous mastoid vent: a medium-term follow-up study.

BACKGROUND: I designed a percutaneous mastoid vent to provide permanent ventilation to the middle ear. The vent consists of an outer titanium tube that osseointegrates with the mastoid bone and an inner Teflon tube that protrudes into the mastoid antrum. OBJECTIVE: To follow up all patients who had the mastoid vent inserted since 1995. STUDY DESIGN AND SETTING: Retrospective study of 14 patients with mastoid vents inserted at the ear, nose, and throat clinic of a district general hospital. PATIENTS: All patients had ventilation problems of the ear that failed to respond to conventional treatment. Three patients had persistent otitis media with effusion; 10 had completely collapsed eardrums; and 1 had failed tympanoplasty with recollapsed eardrum. INTERVENTION AND OUTCOME MEASURE: The mastoid vent extrusion rate, surrounding skin reaction, patency of the vent, and functional results were assessed with a follow-up period of 9 to 36 months. RESULTS: Only 1 vent was extruded in a patient who had a previous cortical mastoidectomy. There was no dermatitis around any of the vents. All vents remained patent, and 9 of the 14 ears underwent successful ventilation. Four ears had adhesions within the mastoid antrum, mainly due to a previous cortical mastoidectomy. Six of 8 ears with intact ossicles also had improved hearing. CONCLUSION: The percutaneous mastoid vent can provide medium-term ventilation to the middle ear.

Adolescent↗