Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “HEART ENLARGEMENT”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 37 records · Page 2Linked to original sources
PubMed1989

[Experience of valve replacement in 16 cases of heart enlargement].

From July 1985 to 1988 Feb. 16 patients with large heart (C-T ratio 65-85%) underwent valve replacements with mechanical valve. Isolated mitral valve replacement was performed in 15 cases and combined aortic and mitral valve replacement in 1. The valvular lesions was rheumatic in origin. The heart function (NYHA) was grade III in 5 cases (32.5%), and IV in 11 (67.5%), 60% of the cases were suffered from moderate or severe pulmonary hypertension. 1 patient died postoperatively. In the survivals the cardiac function was improved and the heart-size reduced distinctly. Some points about the operative results, selection of the time for operation and the postoperative management were discussed.

Adolescent↗
PubMed1976

[Heart enlargement and arterial hypertension in renal arteriovenous fistula].

In a patient hospitalized for progressive congestive heart failure the findings were hypertension, cardiomegaly and a mass in the left upper abdomen. A murmur was audible over the tumor. Clinical suspicion of a renal newgrowth with an arteriovenous fistula was confirmed by renal angiography. After nephrectomy the heart size became normal but the hypertension could only be controlled by drugs. The causes, symptoms and signs, and pathophysiology of arteriovenous shunts are discussed. Therapy is surgical. Surgery eliminates the congestive heart failure.

Adenocarcinoma↗
PubMed1984

[Survey echocardiography. A comparative evaluation of enlarged heart chambers in the echocardiogram and the plain x-ray].

Eighty patients with various types of heart disease were examined by radiography and sectional studies of the heart on the same day. These were compared with 2-dimensional examinations of the chambers during end-diastole. It was found that echocardiography may demonstrate dilated chambers due to a cardiomyopathy where the plain radiograph is entirely normal. Radiographic enlargement of the left side of the heart is not necessarily due to dilatation of the left ventricle, but can also be caused by gross hypertrophy. Echocardiography is indicated for the investigation of cardiac enlargement due to multivalvular disease, cardiomyopathy, combined left and right cardiac insufficiency and the exclusion of pericardial effusions.

Cardiomegaly↗
PubMed1977

[Formation of transversely oriented myofibrils in myocardiocytes in the presence of heart enlargement].

Myocardial hypertrophy was induced in rats and mice by training in the pressure chamber (high altitude hypoxia) or ligation of the apex of the heart. Transversely oriented myofibrils developed in the subsarcolemma area of some cardiomyocytes of the ventricles in a fortnight; the Z bands of the newly-formed myofibrils were connected with the T tubules as in the normal myofibrils. It is supposed that this phenomenon was due to the appearance under the sarcolemma of transversely directed mechanical forrces as a result of enlargement of the cell volume.

Animals↗
PubMed1990

The effects of the pericardium on function of normal and enlarged hearts.

In summary, the pericardium is a membranous structure that surrounds the heart. Its physical properties are such that it is distensible when the total intrapericardial volume is small and inextensible when total intrapericardial volume is large. The pericardium serves to limit acute cardiac distention and to modulate the phenomenon of ventricular interdependence. It is a dynamic structure that, when subjected to chronic stretching--either due to the gradual accumulation of intrapericardial fluid or to cardiac enlargement--will grow to accommodate its contents, such that the working range of pressures between it and the surface of the heart is low. The precise level of cardiac distention that is necessary to engage the pericardium is unknown, but studies in normal and dilated hearts suggest that, at the upper end of the normal volume range of the heart, the intrapericardial volume is just sufficient to reach the noncompliant portion of the tissue's length-tension relation. Further studies are needed to determine whether this behavior represents a hemodynamic protective mechanism in normal animals.

Cardiomegaly↗
PubMed2003

Heart enlargement after thrombolysis for unsuccessful resuscitation.

Thrombolysis has been recently reported for the treatment of unsuccessful out-of-hospital cardiopulmonary resuscitation. This case report presents the first case of mediastinal hematoma following thrombolysis in this setting. Chest X-ray and hemodynamic pattern mimicked pericardial effusion with cardiac-filling compromise. Diagnosis was established by transesophageal echocardiography and chest computerized tomodensitometry.

Aged↗