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Results for “ASTHENOPIA”

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At least 37 records · Page 2Linked to original sources
PubMed1979

Restricted lees screen fields in patients with asthenopia, with and without psychogenic disorders.

Lees screen restrictions of non-specific origin, including apparent restrictions of the SR, IR, and LR muscles, were reported last year by Labow et al. In this complementary study, the authors examined eight apparently healthy subjects who had no history of ocular or psychiatric problems, eight psychiatric patients, and eight who had had a CNS disorder or had suffered cerebral trauma. Comparison of the Lees screen findings, visual fields, and accommodative defects of these patients revealed significant restriction of the Lees screen fields in only those patients who had symptoms of asthenopia (including all in the third group). Thus, such restriction does not depend on emotional state.

Adolescent↗
PubMed2001

Effect of cumulative nearwork on accommodative facility and asthenopia.

PURPOSE: To investigate quantitatively in young adults the relationship between long-term cumulative nearwork, degradation of dynamic accommodative ability and the presence of asthenopic symptoms. METHODS: Subjects consisted of 87 young students and office workers between 18 and 31 years of age with uncorrected visual acuity of 20/30 or better in each eye. The amounts of nearwork, dynamic accommodative facility, and level of asthenopic symptoms were measured for each subject. RESULTS: Total cumulative nearwork time was negatively correlated with accommodative facility and positively correlated with the number of asthenopic symptoms. Furthermore, significant correlations were found between total nearwork time and blurred vision, and blurred vision and reduced accommodative facility. Moreover, the sub-category of "hours spent reading over the years" was found to be significantly correlated with decreased accommodative facility. CONCLUSIONS: The correlations suggest a relationship between cumulative amount of nearwork, decreased accommodative facility and asthenopia.

Accommodation, Ocular↗
PubMed1981

Adaptation to prism-induced heterophoria in subjects with abnormal binocular vision or asthenopia.

In a previous study we investigated how normal subjects adapted to prism-induced heterophoria. In this study we have continued this work by investigating this phenomena in 15 patients with abnormal binocular vision and/or asthenopia. It was found that the majority of subjects lacked or had a deficient adaptation system to base-in and/or base-out prisms, but all except two subjects examined adapted normally to vertical prism. These results suggest that it may be possible to treat the horizontal adaptation system independently from the vertical one. Prism vergences, heterophoria measurements, and the presence of fixation disparity gave little indication of the adaptation ability of the subjects. Symptoms correlated well with adaptation ability.

Accommodation, Ocular↗
PubMed1989

[Asthenopia and objective ophthalmological findings in a population of 2058 VDT operators in Lombardy].

A large population of 2058 VDT operators from all compartments in Lombardia of a large national company was studied. An "ergophthalmological" questionnaire was administered to all subjects, followed by an ophthalmological-orthoptic examination. Ametropias showed a prevalence of 51.9%; the most common refractive defect was myopia followed by astigmatism and hyperopia. Heterophorias with bad or mediocre compensation had a prevalence of 4.1 and 11% respectively. The study of symptoms showed a frequency of 23.5% of intense asthenopic complaints and 21.1% of less severe and less frequent symptomatology. Asthenopia was significantly correlated (chi square) with sex, VDT exposure, refraction, presbyopia, and decompensated heterophorias.

Adult↗
PubMed1997

Bilateral striate cortex contusion presenting with cerebral asthenopia rather than cortical blindness.

We report a 22-year-old female with a history of contusion of bilateral occipital lobes at the age of 1 year. Magnetic resonance imaging confirmed severe lesion in bilateral striate cortices which should have resulted in cortical blindness. However, she displayed a fairly good visual function presenting only cerebral asthenopia and the restriction of the visual field. A positron emission tomography revealed that no areas other than the primary visual cortices responded to visual stimuli. Thus, her residual vision may be attributable to plasticity of the visual cortex in humans.

Adult↗
PubMed1980

[Asthenopia in clinical practice: a comparative study of complaints, clinical findings and results of therapy in asthenopic patients (author's transl)].

A study was carried out by seven ophthalmologists practising in the Berne region on 473 patients complaining of eye strain. Medical histories were taken by questionnaire, refraction was done and phoria measured, followed by therapy in accordance with the physician's opinion. In 380 cases, glasses were prescribed, including prismatic glasses for 14 patients. In 123 cases, local medical therapy was instituted. The statistica evaluation of pre-treatment findings and results of therapy, was carried out using a computer. The principial complaints before treatment were eyestrain and visual disturbances, followed by conjunctival irritation and headache. Women complained more about headaches, men more about visual disturbances. A majority of the patients with visual disturbances needed minus lenses, while for the treatment of headaches mostly plus lenses were needed. In patients with conjunctival irritation local medical treatment resulted in additional relief of symptoms. Cylindric correction had only a minor influence on the result of therapy. Heterophoric patients did not have more complaints than orthophoric ones, and results with spectacles without prisms were the same as in orthophoric patients.

Adolescent↗
PubMed1982

Asynchronous cortical input and asthenopia.

Abnormal delays in retinal-cortical transmission are typically associated with optic nerve disease. Clinically optic nerve diseases produce visual field abnormalities, decreased visual acuity, and color vision defects. Disease processes causing only interocular differences in retinal-cortical transmission properties are not easily detected. Patients with diseases causing interocular latency differences may complain of visual discomfort and perceptual distortions yet perform normally in standard clinical tests. A simple procedure to minimize differences in interocular latency and provide relief from visual disturbances arising from this abnormality is described.

Asthenopia↗