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Results for “Color Vision Defects”

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At least 343 records · Page 19Linked to original sources
PubMed1993

Influence of pathologic scotopization on the extended Rayleigh match.

Pathologic scotopization, an important symptom of retinal disease, can be studied by means of the Nagel II anomaloscope. This method is called the micro-screw method. The micro-screw method was performed in 14 congenital and 13 acquired colour vision defective individuals. The method proves to be useful in detecting symptoms of rod intrusion in colour vision under photopic conditions.

Color Perception Tests↗
PubMed1992

An analysis of colour vision in 10,000 patients.

Examination of a great number of patients resulted in the depth localisation theory. The combination of this theory with the fixation-eccentrisation theory is clinically useful: acquired colour vision defects can be subdivided by the fixation mode and by signs of receptor damage. There are indications that in multiple sclerosis the slowly progressive cases present with more receptor damage than the acute cases.

Adolescent↗
PubMed1978

Color matching and Stiles-Crawford effect in central serous choroidopathy.

Color matching and Stiles-Crawford effect measurement were performed in 3 patients with central serous choroidopathy, 2 normal and 1 deuteranomalous trichromats. The color matches in the affected eye of each patient were displaced to red and could be explained by the hypothesis that the visual photopigments are in reduced optical density due to receptor disorientation caused by serous elevation of the sensory retina. The Stiles-Crawford effects of the affected eyes was abnormal confirming the hypothesis of receptor disorientation. The type III color defect accompanied by pseudo-protanomaly ascribable to receptor disorientation as occurs in central serous choroidopathy may be differentiated from the type III defect without pseudo-protanomaly.

Absorption↗
PubMed1988

[Chronic alcohol-tobacco poisoning: towards an early detection of optic neuropathy].

Forty-two patients (33 men, 9 women) with normal visual acuity admitted to an alcoholism rehabilitation programme were evaluated for colour vision using the automatized Fransworth-Munsell test (ChromopsR) and for central visual field using Friedmann's analyser. An early colour blindness without axis, i.e. a lack of colour discrimination, was often found. At the same time, in the more intoxications perimetric defects appeared in the 30 degrees area first centrally, then followed by arcuate superior scotomas.

Adult↗
PubMed1991

Velhagen Pfügertrident pseudoisochromatic plates in screening congenital red-green vision defects.

Red-green colour vision defects were screened in a group of 425 trade school students using Velhagen Pflügertrident pseudoisochromatic plates. Thereafter, the students were examined with the Nagel anomaloscope. Of the 425 students, 31 (7.3%) were found to be colour defectives. Deuteranomalous defects were found in 4.9% of cases; deuteranopic defects, in 0.2%; and protanomalous defects, in 2.1%. There were no protanopic students in the study group. The Velhagen plates found 19 of the 31 defectives (sensitivity, 61.3%); none of the students with normal colour vision were suspected of being colour defectives (specificity, 100%). The sensitivity of the Velhagen plates is not as high as that of other pseudoisochromatic tests. However, the Velhagen Pflügertrident test is easy to use when screening of pre-school-aged children is needed.

Adolescent↗
PubMed1991

Dominant cone dystrophy starting with blue cone involvement.

The results of ophthalmological and colour vision studies are reported on 13 patients from a family with a dominant cone dystrophy spanning seven generations. The onset of visual deterioration occurred in the third or fourth decade. In the early stages of the disease, when visual acuity is still close to normal, a severe defect in the blue sensitivity is already present, as measured by spectral sensitivity curves and other tests suitable for the detection of tritan defects. In our opinion this condition represents a distinct entity with autosomal dominant inheritance.

Adult↗
PubMedDate not supplied

Colour-blindness in Colombia.

The frequency of defective colour vision was studied in two neighbouring villages in the Andes Mountains of Colombia using AO H-R-R Pseudoisochromatic plates. The frequency of the red-green colour-blindness in males is almost the same in both villages (2.36-2.53%), being similar to frequencies reported for other mestizo' populations in Latin America. In one of the communities, families in which colour-blindness occurred were wealthier (P is less than 0.05) than non-colour-blind families, but there were no significant differences by colour vision class in numbers of surviving children nor mother's marriage age. These findings are consistent with the idea that in societies at the agricultural level, colour-blindness is selectively neutral. The association of colour-blindness with higher socio-economic status is expected given the history of European conquest in the New World, and suggests that the major cause of varying rates of the defect in Latin American populations is socio-economic heterogeneity and by inference different degrees of European-Amerindian admixture.

Age Factors↗
PubMedDate not supplied

Familial appraisal of colorblindness in school children of an Indian population.

A total 2000 unrelated school children were screened for colorblindness in Vishakhapatnam, India. Whether the protan and deutan defects are the result of mutations at one locus or at two loci has not been completely resolved, although the evidence favors two discrete loci. The investigation was extended to the families of the 40 color vision anomalous children to study the descendance patterns of these two loci. The importance of these observations are discussed.

Adolescent↗
PubMed1977

[Acquired colour-vision-deficiencies caused by side-effects of pharmacotherapy (author's transl)].

Acquired colour-vision deficiencies are an early indicator for drug-induced retinopathy as well as drug-induced retrobulbar neuritis. Koellner's rule, which says, that damage of the retina induces a tritan-defect, and damage of the optic nerve induce a red-green-defect is also valid for defects secondary to drug-toxicity. Pseudoisochromatic plates, anomaloscope and other tests (Panel D-15-test) have to be selected correspondingly to use them as screening-methods.

Antipsychotic Agents↗
PubMed1976

A consideration of the racial incidence of congenital dyschromats in males and females.

The female incidence of congenital dyschromats corresponds to the square of the males in the Northern European populations, but this relation is not always true in the non-white races. The above-mentioned facts in the non-white races which are theoretically strange are considered to be caused by an anti-glare factor of pigment epithelium in their eyes, on the assumption that the abnormal color sense of color defectives may be a special form of visual dysfunction glare pointed out by IINUMA.

Asian People↗