Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “MICROSPORUM”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 325 records · Page 18Linked to original sources
PubMed1988

Ribosomal phosphoproteins in Microsporum canis.

Ribosomal phosphoproteins of Microsporum canis labelled in vivo were characterised by two-dimensional and SDS polyacrylamide gel electrophoresis. A small subunit protein, S6, was the only phosphoprotein identified in 40S and 80S in basic-acidic two-dimensional gels. Three different forms of phosphorylated S6 were also observed in 40S subunit. On SDS gels five phosphoproteins were identified in 80S; of these three were present in 40S and two in 60S. S6 was the only basic phosphoprotein, while the other four were acidic.

Electrophoresis, Polyacrylamide Gel↗
PubMed1987

Mycological studies on Microsporum equinum isolated in Finland, Sweden and Norway.

Sixteen isolates of Microsporum equinum from cases of equine dermatophytosis in Finland, Sweden or Norway were studied mycologically. All the isolates produced typical macroconidia and were negative in the hair perforation test in vitro and were urease-positive. The growth pattern of M. equinum on polished rice greatly resembled that of M. audouinii during the first 2-3 weeks of incubation. All the isolates were incompatible with Nannizzia otae, the teleomorph of M. canis, but fresh M. equinum isolates showed growth stimulation against the (-) tester strain of N. otae. No cleistothecia or pseudocleistothecia were seen.

Animals↗
PubMed1986

Spontaneous dermatophytosis due to Microsporum canis in rabbits.

Reports of naturally occurring Microsporum canis infection in the rabbit are rare. During the tenth week of a 91 day percutaneous toxicity study, 17 of 30 adult New Zealand white rabbits developed skin lesions varying from multiple papules to ringed lesions 1 X 2 cm in diameter. The lesions were not pruritic. Hair and scale samples taken from rabbits with lesions were cultured for dermatophytes. Based upon colony morphology and macroconidial characteristics, M canis identification was confirmed. At the time of necropsy, fluorescence was observed in three animals examined with a Wood's lamp, and of 10 rabbits that were positive on culture, seven were clinically normal. Microscopically, hair follicles contained spores and mycelia. The source of this outbreak was not determined. Tap water was cultured and found negative for pathogenic fungi. These findings document M canis infections in laboratory-housed New Zealand white rabbit, such an asymptomatic carrier state should be considered in this outbreak. The significance of dermatomycosis in laboratory animals is primarily as a zoonosis and a research complication.

Animals↗
PubMed1986

Dual infection of the toe web caused by Trichophyton rubrum and Microsporum canis.

A case of tinea pedis caused by a dual infection with Trichophyton rubrum and Microsporum canis is reported. It is of particular interest because infections of the toe webs caused by M. canis are uncommon and the combination of T. rubrum and M. canis has not been reported. In addition, a marked difference in response to treatment with an imidazole (bifonazole) was observed.

Adult↗
PubMed1979

Electron microscopic evidence of Phosfon D-induced alterations in the membranes of the dermatophytic fungus Microsporum cookei.

The dermatophytic fungus Microsporum cookei was cultivated for 24 h in the presence of subinhibitory and inhibitory concentrations (50 and 100 micrograms/ml) of Phosfon D, a growth retardant for higher plants also affecting fungal development, and its toxic effects were examined at the ultrastructural level. In both treatments, Phosfon D attacked the membranes, whose structural integrity was clearly compromised with damage of particular severity to mitochondria, nuclei and endoplasmic reticulum. In the instance of fungal growth suppression, the compound also caused plasmolytic and autolytic phenomena, sometimes accompanied by plasma membrane breakages. The submicroscopic effects observed confirm that Prosfon D is an antifungal compound which displays its toxic effects in the area of lipid metabolism, probably preventing the synthesis of fundamental components of the cellular membranes, such as unsaturated fatty acids and sterols.

Antifungal Agents↗
PubMed1978

[Microsporum persicolor, an easily misinterpreted dermatophyt].

Trichophyton persicolor was described by Sabouraud in 1910, and was mistaken by most mycologists for Trichophyton mentagrophytes. Recently the infection in wild rodents was discovered and the independence of this fungus proved by its perfect form (Nannizzia persicolor) which is called at the present time Microsporum persicolor.

Adult↗
PubMed1979

Taxonomy of Exophiala werneckii and its relationship to Microsporum mansonii.

Study of the neotype culture of Exophiala werneckii (Cladosporium werneckii), as well as additional isolates, has shown that the yeast-like and hyphal conidiogenous cells are annellides. E. werneckii cannot be accommodated in Cladosporium, a genus characterized by catenulate holoblastic coanidia. E. werneckii and the dematiaceous hyphomycete that Castellani misidentified as Microsporum mansonii (Cladosporium mansonii) are two entirely different fungi. M. mansonii is considered to be a synonym of the Malassezia furfur since both names were used to describe in tissue the etiologic agent of pityriasis versicolor.

Cladosporium↗
PubMed1982

Isolation of an extracellular proteinase (keratinase) from Microsporum canis.

Proteolytic activity was demonstrated when a strain of Microsporum canis was cultured in broth with human hair, but not in the medium without hair. The extracellular proteinase (keratinase) was isolated and purified by chromatography. Disc electrophoresis showed one protein band of extracellular proteinase, and the antibody against this enzyme gave a single precipitin line in agar diffusion. The IgG fraction completely neutralized the proteinase activity. The proteinase of M. canis may play a role in infection caused by this fungus, by affecting keratinized tissue such as stratum corneum and hair.

Chromatography, Gel↗
PubMed1982

The changing clinical picture of Microsporum canis infections in Sweden.

All 22 cases of infections with Microsporum canis verified by culture during a five-year period were surveyed. Only tinea corporis was found. The clinical picture was varied and in more than half of the cases the preliminary diagnosis was other than tinea. The classical tinea capitis was not found in any case. Direct KOH-mounts showed poor correlation to the cultural findings (positive in 33%). It is concluded that mycological screening of certain dermatological disorders should be performed by culturing.

Adolescent↗
PubMed1978

Microsporum canis infection: first epidemic in Israel.

We report here on the first epidemic of Microsporum canis infection in Israel. All 78 cases were diagnosed in the port city of Eilat between August, 1975 and May 1976. The epidemic has apparently been controlled with Griseofulvin therapy and public health measures.

Adolescent↗
PubMed1984

Partial characterization of the extracellular keratinase from Microsporum canis.

The extracellular keratinase of Microsporum canis released peptides from alpha-type fibrous protein and the membranous fraction isolated from human stratum corneum. Inhibition of the enzyme by phenylmethyl-sulfonylfluoride and its weak inhibition by N-ethylmaleimide and etheneglycol tetra-acetic acid indicated that it is probably a serine proteinase.

Egtazic Acid↗
PubMedDate not supplied

Favus in a fighting cock caused by Microsporum gallinae.

The first diagnosis of favus in chickens in Costa Rica was made in a 1-year-old fighting cock that had lesions surrounding the comb. The etiologic agent was isolated and identified as Microsporum gallinae. The rooster recovered during topical treatment with tolnaftate and oral treatment with griseofulvin.

Animals↗
PubMed1982

Lipid composition of Microsporum gypseum.

Lipid composition of Microsporum gypseum grown in Sabouraud's liquid media on a rotary shaker were analyzed. The organism contains 6.07% lipid (dry weight basis), of which 75.88% is neutral lipids, 0.9% phospholipids and the rest are glycolipids and pigments. Phospholipids of M. gypseum contain phosphatidyl choline (44.21%), phosphatidyl ethanolamine (17.8%), polyphospho inositide (12.13%), phosphatidyl inositol (8.24%), phosphatidic acid (4.61%) and cardiolipin. Phosphatidyl serine is absent. The neutral lipid composition (expressed as mg/g dry weight of mycelia) of M. gypseum is monoglycerides (1.35), diglyceride (2.87), triglycerides (35.79), free fatty acids (1.00), sterols (3.95) and sterol esters (1.13).

Glycolipids↗
PubMed1981

A glabrous Microsporum canis in Greater London.

A variant of Microsporum canis, which produces glabrous colonies on primary culture, has been frequently isolated from cases of scalp and body ringworm by this department. The records of this laboratory, which undertakes a diagnostic service covering most of the Greater London Area, show that the prevalence of M. canis among scalp infections has increased during the past ten years, and this species is presently responsible for the majority of cases diagnosed by culture. The glabrous form now represents one third of the scalp isolates of M. canis and it has been found most commonly in the West and South of Greater London.

Dermatomycoses↗