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At least 289 records · Page 16Linked to original sources
PubMed1986

Paralysis with Ixodes cornuatus envenomation.

Ixodes cornuatus is a tick that is widely distributed in Victoria, Tasmania and southern New South Wales. Serious human envenomation has not been reported previously. The clinical syndrome that results from envenomation by Ixodes cornuatus in a three-year-old boy is presented. Bulbar palsy and respiratory failure necessitated endotracheal intubation and mechanical ventilation for five days. Canine tick antivenom was administered.

Animals↗
PubMed2006

Abducens nerve palsy following a tick bite: a case report.

Neuromuscular paralysis caused by salivary proteins of ticks is a well-known complication after tick bites in Australia, North America, and South Africa. Symptoms may include general weakness, difficulty walking, ascending paralysis, and bulbar paralysis with diplopia, culminating in respiratory failure. In Europe, toxin-mediated paralysis has rarely been noted. We report a case of cranial nerve paralysis with delayed onset after a tick bite in northern Germany.

Abducens Nerve Diseases↗
PubMed1989

Seasonal incidence of Karoo paralysis in relation to the infestation density of female Ixodes rubicundus.

The epidemiology of Karoo paralysis, induced by the tick Ixodes rubicundus, was investigated in a paralysis-enzootic area of the southwestern Orange Free State, Republic of South Africa. Sheep became paralysed during the last week of April and the first week of May during the 1985 and 1986 seasons, respectively. Paralysis cases were recorded up until July, but most paralysis cases occurred during the first week of May with a second but smaller peak occurring during June. Tick numbers on paralysed sheep closely followed peak periods in paralysis. A total of 65 and 50% of the naive sheep became paralysed during the 1985 and 1986 seasons, respectively. The seasonal pattern of tick infestation by female I. rubicundus was similar on both previously exposed and on naive sheep. The results of this study indicate that a certain number of ticks per kilogram host body mass is required before symptoms of paresis of paralysis are observed. In general the tick burden per kilogram host mass was significantly greater in paralysed compared to non-paralysed sheep. The range in the number of ticks per kilogram host mass may, however, overlap between non-paralysed and paralysed groups. Previously exposed or previously paralysed sheep are less susceptible to paralysis in the following year. Differences in susceptibility between individual sheep seems likely.

Analysis of Variance↗
PubMed1989

In-vivo effects of ivermectin on Rhipicephalus appendiculatus: the influence of tick feeding patterns and drug pharmacokinetics.

Sublethal effects seen amongst Rhipicephalus appendiculatus feeding on ivermectin-treated rabbits were diverse and dependent both on drug dose, pharmacokinetics and tick feeding patterns: changes in drug formulation, the time of infestation relative to treatment, and the tick instar used, profoundly influenced acaricidal activity. Death was a sequel to paralysis only if tick feeding was interrupted for sufficient time to produce irreversible dehydration. Concurrent pharmacokinetic investigations revealed that, for the larvae of R. appendiculatus, the mean critical lethal dose of ivermectin imbibed over a 5-day engorgement period was 3500 micrograms/kg. This quantity of ivermectin was achieved in the blood-meals of larvae feeding on rabbits treated subcutaneously with a single dose of Ivomec injection (MSD)* greater than or equal to 800 micrograms/kg, provided infestation took place within 24 h of treatment. At lower drug doses, or if larval infestations were delayed for greater than 24 h post-treatment, the quantity of circulating ivermectin (and thus imbibed by the tick larvae) fell below 3500 micrograms/kg and an increasing percentage of larvae successfully engorged and detached. More than 90% of such larvae moulted to the nymphal stage. Nymphae and larvae exhibited similar susceptibility to ivermectin on treated rabbits which could be explained by similar feeding patterns. However, adult female and male ticks were markedly less susceptible and interpretation of ivermectin-induced effects was more complex.

Animals↗
PubMed2006

[Eyelid tick bite].

CASE REPORT: We describe a patient who was bitten on his right upper eyelid by a common canine tick. The tick was impregnated with gasoline prior to being removed. DISCUSSION: Tick bites are uncommon in ophthalmologic practice. They occur more frequently in rural areas, especially in spring and summer. Correct identification of the problem and treatment is essential. The tick needs to be completely removed in order to avoid the embedding of different parts of the insect. Zoonoses, such as rickettsiosis, arbovirus and paralysis due to ticks must be ruled out.

Adult↗
PubMed2003

Increasing the paralyzing ability of a laboratory colony of Dermacentor andersoni Stiles.

Field-collected Rocky Mountain wood ticks Dermacentor andersoni Stiles from British Columbia caused limited paralysis in hamsters at high doses, but not at lower doses, while ticks collected from Alberta failed to cause paralysis. This prompted development of a laboratory strain of ticks with a more consistent ability to cause paralysis. Progeny of ticks that paralyzed a cow near Walhachin, British Columbia were reared and assayed for paralyzing ability. A high-virulence colony was selected for four generations using female ticks that cause paralysis singly on hamsters. A low-virulence colony was concurrently selected using female ticks that failed to cause paralysis at four ticks per hamster. Logistic regression indicated that the paralyzing ability of the high-virulence colony was consistently higher than that of the low-virulence colony, but that the paralyzing ability of both colonies increased through four generations of selection. Ticks required storage at 10 degrees C to terminate behavioral diapause. The effects of prolonged storage at 10 degrees C on the paralyzing ability of ticks was estimated and was small compared with differences between colonies and across generations. Single ticks from the high-virulence colony caused an increasing proportion of paralysis with each generation, but only limited paralysis with no increase across generations for the low-virulence colony. Single ticks from the high-virulence colony that caused paralysis were similar in weight and percent weight gain compared with single ticks that failed to cause paralysis. Engorged weight and egg mass production after four generations of rearing in the laboratory were similar to that measured for field-collected adults. Results indicated that laboratory selection could increase paralysis, but not eliminate it. This was likely a result of an inability to select male ticks.

Animals↗
PubMed1988

An undescribed Rhipicephalus species associated with field paralysis of Angora goats.

Paralysis of Angora goat kids caused by adult ticks, which most probably belong to an undescribed species of the Rhipicephalus pravus group, is described. Confirmed cases of paralysis occurred in the south-western Orange Free State between the second half of September and the first half of November as well as during the first half of February. The mean female tick burdens (mean = 21.4) of paralysed Angora kids were significantly higher than those of healthy kids (mean = 4.4). The predilection attachment site of the adult ticks was the ears of the goats. The elephant shrew Elephantulus myurus is a preferred host of the immature stages whereas the scrub hare Lepus saxatilis serves as an important host for the adult ticks.

Animals↗
PubMed1986

Immunological responses and the role of the paralyzing toxin in rabbits infested with Rhipicephalus evertsi evertsi.

Twenty-nine rabbits were exposed to 4 infestations with 20, 50, and 100 adult red-legged ticks (Rhipicephalus evertsi evertsi). Upon challenge, 16 rabbits (55%) contracted paralysis, and 14 of these (88%) died. Most deaths occurred in rabbits challenged with 100 ticks. Immunity to paralysis and subsequent resistance to tick feeding developed in the hosts that survived the initial challenge. Antibody titers to a salivary gland antigen increased progressively as determined by the following tests: passive hemagglutination, agar gel double immunodiffusion, and enzyme-linked immunosorbent assay. Rabbits challenged with 20 ticks displayed declining titers with advanced frequency of challenge and few of the rabbits in this group developed paralysis. There was no decline in antibody titers in groups of rabbits exposed to 50- and 100-tick loads. It is suggested that a certain quantum of antigen injected by the ticks is necessary for induction of immunity to paralysis.

Animals↗
PubMed1979

Tick toxicosis in North America.

This is a case presentation and review of an uncommon disorder, tick toxicosis. The history, epidemiology, pathophysiology, and treatment are discussed. This disorder was mentioned in diaries from the early 1800s and has been reported in 18 states and the District of Columbia. A review of 70 cases reveals that the typical patient is a female child who develops leg weakness, irritability, or clumsiness. The exact site at which the toxin induces the paralysis is unknown. Removal of the tick usually reverses the paralysis within hours. Confusing tick toxicosis with other disorders may occur, and death has resulted. This article will remind physicians to consider tick toxicosis when seeing patients with acute ataxia or ascending paralysis and to, perhaps, prevent death from an easily treatable disorder.

Adolescent↗