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At least 289 records · Page 16Linked to original sources
PubMed2004

Postoperative stiffness and adhesion formation around repaired and immobilized Achilles tenotomies are prevented using a model of heat shock protein induction.

BACKGROUND: Tendon repair and subsequent immobilization is frequently complicated by postoperative stiffness secondary to inflammation and peritendinous adhesions. Thermal preconditioning is known to reduce inflammation by inducing formation of cytoprotective heat shock proteins. This study evaluates the role of thermal preconditioning following complete division and repair of the Achilles tendon, with subsequent immobilization, mimicking the typical clinical scenario. MATERIALS AND METHODS: Twenty-four New Zealand White rabbits were used in the study. The treatment group underwent thermal preconditioning, by elevating their core temperature to 41.5 degrees C for 20 minutes. The Achilles tendon of the hindlimb was divided and repaired 18 hours following thermal preconditioning. The animals were sacrificed following 3 weeks of immobilization. Range of movement of the ankle, tendon gliding, quantity of adhesions, and weight of repaired tendons were assessed. RESULTS: Loss of range of movement at the ankle was significantly less in the treatment group versus controls (P = 0.02). The quantity of adhesions and weight of the repaired tendons were significantly reduced in the treatment group (P = <0.001 and P = 0.005, respectively). Tendon gliding relative to the surrounding soft tissue was also significantly improved in the treatment group (P = 0.002). CONCLUSION: Preconditioned animals demonstrated a significantly better range of ankle movement, decreases in adhesion formation and in the gliding, and dimensions of tendons. Thermal preconditioning therefore has the potential to improve clinical results in tendon surgery following repair and immobilization.

Achilles Tendon↗
PubMed2002

Stylomandibular tenotomy in the transcervical removal of large benign parapharyngeal tumours.

We used a simple modification of the transcervical approach in a selected group of nine patients with large benign parapharyngeal space tumours, all of whom met the following inclusion criteria: The tumour was benign on fine needle aspiration, the encapsulated tumour was not attached to skull base or great vessels in the parapharyngeal space on imaging. Adequate exposure was achieved by just dividing the stylomandibular ligament and retracting the mandible anteriorly. It was possible to remove the tumours successfully and safely in all nine patients without the need for mandibulotomy or superficial parotidectomy. In seven cases, the tumour crossed the midline. There were no major perioperative neurological or vascular complications. On subsequent follow up, there were no clinical or radiological signs of residual or recurrent tumour.

Humans↗
PubMed1994

Tubular networks in soleus muscle fibers of the rat following tenotomy. I. Spatial organization.

Complex systems of tubules, 30-40 nm in diameter, were observed in myofibers of the soleus muscle of Wistar rats following experimental transection of the Achilles tendon. The appearance of these systems varied remarkably, depending upon the plane of sectioning. The spacial arrangement of the tubules was reconstructed by comparing several sections through individual systems. Such reconstructions revealed that they are networks of tubular elements arranged in an hexagonal pattern. A three-dimensional wire model was prepared, illustrating the spatial organization of the tubular systems. The model consists of four groups of lattices with a honeycomb-like arrangement. The lattices of each group are parallel to one another, and intersect those of the other groups at an inclination of 60 degrees.

Animals↗
PubMed2004

Stress-shielding as a cause of insertional tendinopathy: the operative technique of limited adductor tenotomy supports this theory.

The aetiology of tendinopathy is poorly understood. A new hypothesis proposed argues that tendinopathy may not be purely a tensile injury, rather that altered mechanics such as compression or stress-shielding may be important. Both tendon compression and a decrease in tendon load (stress-shielding) will induce change in a tendon similar to that seen in an insertional tendinopathy. Stress-shielding as a cause of tendinopathy is supported by the clinical success of operative release of adductor longus. This surgery releases the superficial section of the normal adductor longus tendon at a point distal to the insertion. This may have the effect of transferring stress from the superficial section of the tendon to the stress-shielded deeper portion, and the induction of normal loads in both the deeper and superficial portions of the tendon may assist in tendon recovery. This interesting hypothesis and clinical intervention require further investigation

Athletic Injuries↗
PubMed1990

Increased association of ribosomes with myofibrils during the skeletal-muscle hypertrophy induced either by the beta-adrenoceptor agonist clenbuterol or by tenotomy.

Ribosome distribution in skeletal-muscle myofibres was investigated by immunohistochemistry and microdensitometry by using anti-(60 S ribosomal subunit) antibodies. Administration of the beta-adrenoceptor agonist clenbuterol caused an increase in the staining of the myofibrillar region with this antibody relative to that found in the subsarcolemmal cytoplasm. A similar effect was observed during hypertrophy of the plantaris muscle following severance of the tendon to the gastrocnemius. The results suggest that increased association of ribosomes with the myofibrils occurs during muscle hypertrophy.

Animals↗