Cogan and Verhoeff: a friendship of genius.
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Gender differences in fear were examined in 693 Chinese children and adolescents. Subjects were asked to rate their own fears, the fears of their best friends, and the fears of "other" classmates using the Fear Survey Schedule for Children--Revised (Ollendick, 1983). Consistent with previous investigations in Western and Eastern countries, girls rated themselves as more fearful than boys. In addition, both girls and boys rated their best friends as similar in number, content, and intensity of fears. However, girls rated their classmates as less fearful than themselves or their best friends, while boys rated their classmates as more fearful then themselves or their best friends. Findings are discussed in terms of gender role expectations and similarity-attraction hypotheses.
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Using a sample of 1227 Swedish adolescents we examined peer relations across contexts and for multiple peer targets (three "Very Important Persons", VIPs). Specifically, we examined the relations between antisocial behaviour and the types of relationships individuals had with their VIPs (e.g. friend, romantic partner), the contexts in which they had met, and where they spent time (e.g. school, neighbourhood, club). Additionally, we tested an "additive homophily" hypothesis, or the idea that youths would show unique similarities to multiple peers. Results showed that individuals who nominated romantic partners as their first VIPs were the most antisocial (both boys and girls), and individuals who had met and spent time with their first VIPs in the neighbourhood were also the most antisocial. Similar results were found for the antisocial behaviour of the first VIP. Finally, results supported the additive homophily hypothesis, showing that significantly more variance in individual behaviour is explained when including second and third VIPs.
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There is a growing body of evidence suggesting that bacterial signaling molecules are involved in cross-kingdom communication with eukaryotic cells. Bacterial signaling molecules can enter eukaryotic cells and appear to modulate immune responses to bacterial pathogens. Recently, an enticing study demonstrated that mammalian airway epithelial cells have the ability to inactivate these bacterial signaling molecules, suggesting that this activity might have a role in the host innate response against bacterial infections.
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