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Results for “Parietal Lobe”

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Increased sensitivity to ipsilateral cutaneous stimuli following transcranial magnetic stimulation of the parietal lobe.

Transcranial magnetic stimulation of the sensorimotor cortex results in decreased sensitivity of threshold electrical stimuli to fingers of the contralateral hand. It has been suggested that one factor contributing to neglect contralateral to a unilateral parietal lesion is a release of the normal hemisphere from reciprocal interhemispheric inhibition by the damaged hemisphere. Consistent with this account, the current study demonstrated that transcranial magnetic stimulation over the parietal cortex results in increased sensitivity to cutaneous stimulation ipsilateral to the stimulation. The likely mechanism is a transcranial magnetic stimulation-induced transient dysfunction of the ipsilateral parietal cortex that then results in disinhibition of the contralateral parietal cortex.

Adolescent↗

Pre-movement parietal lobe input to human sensorimotor cortex.

Movement-related cortical potentials (MRPs) were recorded in an auditory dichotic selective attention experiment in patients with focal lesions centered in either posterior superior temporal gyrus (temporal) or in lateral parietal cortex (parietal). Controls and temporal patients generated comparable pre-movement negative shifts (NSs) and motor potentials (MPs), onsetting about 400 ms prior to movement and maximal in amplitude over scalp sites contralateral to button press. Unilateral parietal cortex lesions markedly reduced the NSs but preserved the MP component of the MRP. The results indicate that human parietal association cortex exerts modulatory input to sensorimotor cortex, beginning at least 400 ms prior to movement. The differential effect on the NSs and MPs by parietal lesions suggests that these MRP components may have independent intracranial generators.

Acoustic Stimulation↗