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Results for “CATTLE DISEASES”

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At least 271 records · Page 15Linked to original sources
PubMed1993

A photometric method for recording hoof diseases in cattle, with special reference to haemorrhages of the sole.

After cleaning and trimming, the sole surface and interdigital space of the hooves of cows are photographed in a standardized way. The colour slides are projected on to a screen and the haemorrhages of the sole (reflecting Pododermatitis aseptica diffusa) and the lesions of Dermatitis interdigitalis and Erosio ungulae are scored according to their severity and extent by 2 people who do not know the identity of the cows. In a repeatability test the correlation coefficients between 2 assessments of the haemorrhages in the sole ranged between 0.78 and 0.88 for assessments of the front hooves, the hind hooves and all the hooves together.

Animals↗
PubMed1995

[Ultrasonographic findings of perivascular jugular vein diseases in cattle].

Ultrasonographic examinations were used to characterise and describe perivascular changes of the jugular vein like abscess, hematoma and periphlebitis. Well circumscribed masses with irregular, multiple, hypoechogenic areas, corresponding to fluids and necrotic tissues, suggested an abscess. A fresh hematoma, adjacent to the wall of the vein was shown as a clearly delineated structure with a hyperechogenic content. Cases with periphlebitis were characterised by a diffuse anechoic accumulation of fluids infiltrating the perivascular tissue. The diagnoses were proven by needle aspiration biopsy using ultrasound control.

Abscess↗
PubMed1989

Serological comparison of cytopathogenic and non-cytopathogenic bovine viral diarrhea-mucosal disease viruses isolated from cattle with mucosal disease.

Ten cattle that died with mucosal disease were examined for bovine viral diarrhea-mucosal disease (BVD-MD) viruses. Both cytopathogenic (CP) and non-cytopathogenic (NCP) BVD-MD viruses were isolated concomitantly from 9 of them, and only a CP virus was recovered from the other. Then each pair of CP and NCP viruses was compared serologically by a serum neutralization test. Each pair of CP and NCP viruses from the same cattle was found to be serologically indistinguishable, although a minor antigenic difference was observed among the groups of the paired viruses. These results seem to support the hypotheses that mucosal disease occurs in persistently infected cattle which were induced by in utero infection with NCP virus when they are superinfected with CP virus, and the antigenic homology in CP and NCP BVD-MD viruses may be an important factor in the pathogenesis of the disease.

Animals↗
PubMed1977

Pathology of Degnala disease in cattle and buffaloes.

Degnala disease, which is believed to be a mycotoxicosis, has clinical syndrome similar to chronic ergotism and is characterised by development of oedema, necrosis and gangrene of the legs, tail, ears, etc. The gross and histopathologic studies revealed thickening of the wall of blood vessels, associated with thrombus formation, and eosinophilic infiltration in the subcutaneous connective tissue of the affected parts of the body.

Animals↗
PubMed1996

Experimental mucosal disease in cattle: changes in the number of lymphocytes and plasma cells in the mucosa of the small and large intestine.

Changes in the number of lymphocyte and plasma cell subtypes were investigated in the lamina propria and in the epithelium of the small and large intestine of cattle with mucosal disease. Mucosal disease had been induced experimentally in seven out of 13 animals persistently viremic with non cytopathogenic BVD-virus by inoculation with a matching cytopathogenic BVD-virus. For comparison, six clinically healthy, persistently viremic cattle were used. IgA+, IgM+ and IgG1+ plasma cells, BoCD4+, BoCD8+ and gamma delta + T-lymphocytes, and the antigen of the cytopathogenic BVD-virus were demonstrated in tissue sections by immunohistochemistry. Distribution of cellular subtypes in the controls was consistent with data reported from non infected cattle. In cattle with mucosal disease, a decrease in the number of plasma cells which was significant for IgA+ and IgM+, but not for IgG1+ plasma cells was found in the lamina propria. The number of BoCD4+ T-lymphocytes was reduced in the small intestine, whereas their number per mm2 of mucosa was increased in the large intestine. Numbers of intraepithelial BoCD8+ and gamma delta + T-lymphocytes were severely decreased. Antigen of the cytopathogenic BVD-virus was detected predominantly in epithelial cells of the crypts. Overall there is a severe loss of effector cells which are essential components of the humoral and cell mediated immune protection of the mucosal barrier. The decrease of immunoregulatory cells in the lamina propria and epithelium may contribute to the transformation of mucosal architecture in mucosal disease.

Animals↗
PubMed1988

A cellular analysis of immunosuppression in cattle with mucosal disease.

Eleven cattle with mucosal disease (MD) were investigated to determine the constitution and function of the cells participating in the immune response. The total numbers of blood mononuclear cells varied within the normal range but a poor response of the lymphocytes to mitogen stimulation indicated a state of immunosuppression in the cattle with MD. Identification of subpopulations of the mononuclear cells revealed that eight of the cattle with MD had an increased proportion of B cells. However, the proportions of monocytes and T cells were in the normal ranges, resulting in a low proportion of cells not identified as being either B or T cells (null cells). Three of the cattle with MD had an increased proportion of cells with receptor for the Fc part of IgG (Fc gamma + cells). Bovine Fc gamma + cells have previously been shown to exert suppressor activity, as was also demonstrated in the present study by increased mitogen induced lymphocyte stimulation following removal of the Fc gamma + cells. The effect of depletion of Fc gamma + cells was at least five-fold greater in the two cattle with MD tested than the two controls. It appears that a diminished number of null cells and, or, increased suppressor cell activity might contribute to the observed immunosuppression in cattle affected with MD.

Animals↗
PubMed1990

beta-Mannosidosis in a Salers calf: a new storage disease of cattle.

A storage disease characterised by widespread vacuolation of cells throughout the body was diagnosed in a newborn calf of the Salers breed. Extraction and analyses of water soluble material showed that the storage material was oligosaccharide in nature and was probably derived from the core region of the heterosaccharide moiety of glycoproteins. Glycosidase analyses showed that the disease was due to a deficiency of acidic beta-mannosidase.

Journal Article↗
PubMed2001

[Clinical picture and differential diagnosis of foot and mouth disease in cattle].

Foot-and-mouth disease (FMD) in cattle is characterized by the formation of vesicles and erosions in the mouth, teats and feet. Despite extremely high morbidity, mortality in adults is usually very low, however up to 50% of calves dies due to cardiac involvement or secondary infections. This paper describes the signs of FMD in cattle as well as those of other diseases which causes similar lesions in this species.

Age Factors↗
PubMed1982

Akabane disease in cattle: congenital abnormalities caused by viral infection. Experimental disease.

Nonpurulent encephalomyelitis and polymyositis were primary lesions of cattle and goats experimentally infected with Akabane virus. Two caprine fetuses, two months old, were infected placentally and examined 11 days after inoculation; twin caprine fetuses, three months old, were inoculated intramuscularly through the dam's uterus and examined nine days after inoculation. Both lesions were seen in each fetus. Reactive proliferation of immature endothelial cells was a significant encephalitic change. Myositic changes included parenchymal degeneration and cell infiltration in fetuses in the myotubule phase and the beginning of the myofiber phase. Only nonpurulent encephalomyelitis was seen in six calves 14 days to one year old, inoculated intracerebrally and examined six to 47 days after inoculation. Nerve-cell degeneration, neurological mobilization, and perivascular cuffs were typical encephalitic changes in the calves. Five fetuses were infected transplacentally and had polymyositis alone. The four bovine fetuses, two to six months old, were examined nine to 18 days after inoculation, and one caprine fetus, one month old, was examined 11 days after inoculation. Neither encephalomyelitis nor polymyositis was seen in four calves under one year old that were inoculated intravenously.

Animals↗