Search PubMed⌕ Search

SEARCH · Search PubMed

Results for “STRIDOR”

Search indexed PubMed citations on genomics, clinical trials, systematic reviews and public health. Explore titles, authors and supplied subject terms, then open the PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 253 records · Page 14Linked to original sources
PubMed2005

Stridor and Horner's syndrome, weeks after attempted right subclavian vein cannulation.

A 23-year-old woman presented with renal failure resulting from polycystic kidney disease (PKD) aggravated by tubulo-interstitial nephritis. Emergency haemodialysis was planned, and cannulation of the right subclavian vein was attempted, but failed. During this procedure, inadvertent arterial puncture occurred. Transient mild ischaemia of the right arm, and a transient Horner's syndrome were noted. Seven weeks later she presented with severe stridor with impending respiratory failure necessitating emergency intubation; the right-sided Horner's syndrome had recurred. CT imaging showed a large pseudo-aneurysm of the brachiocephalic artery resulting in severe compression of the trachea. Using a prosthetic graft, the operation for the pseudo-aneurysm was successful; there were mild neurological sequelae. Although her family history was negative, autosomal dominant PKD should be considered, and we discuss the possible role of a pre-existing PKD-associated aneurysm.

Adult↗
PubMed2005

The endotracheal tube cuff-leak test as a predictor for postextubation stridor.

BACKGROUND: The endotracheal tube (ETT) cuff-leak test (CLT) has been proposed as a relatively simple, noninvasive method for detecting the presence of laryngeal edema prior to tracheal extubation. OBJECTIVE: To determine the value of the CLT for predicting postextubation stridor (PES) among medical and surgical patients, and to assess the impact of certain variables on the incidence of PES. METHODS: We conducted a prospective, observational study in the intensive care unit at Washington Hospital Center, a 907-bed acute care hospital in Washington DC, with patients who were intubated for > 24 h. As part of respiratory therapy quality assurance, patients intubated for > 24 h are evaluated daily for extubation readiness, and CLT is conducted prior to extubation. The CLT results and the postextubation outcomes were prospectively recorded for 6 months. RESULTS: Of the 462 patients studied, 20 (4.3%) developed PES that required treatment; 7 of those 20 (1.5%) required reintubation. With patients who failed the CLT, defined by an absolute leak volume < or = 110 mL, the positive predictive value for PES was 0.12, the negative predictive value was 0.97, the sensitivity was 0.50, and the specificity was 0.84. Using different definitions for CLT failure did not improve the accuracy of CLT for predicting PES. Patients who had PES were more likely to be female (6.5% vs 2.4%, p = 0.04), to have a longer duration of translaryngeal intubation (6.5 + 4 d vs 4.5 + 4 d, p = 0.02), and to have a larger ratio of ETT size to laryngeal size (49.5 + 6% vs 45.5 + 6%, p = 0.01). CONCLUSIONS: Failing the CLT was not an accurate predictor of PES and should not be used as an indication for either delaying extubation or initiating other specific therapy. Female patients, those whose ratio of ETT size to laryngeal diameter was > 45%, and patients intubated for > 6 d were more likely to develop PES.

Adrenal Cortex Hormones↗
PubMed2004

Metabolic stridor: a case report.

The clinical features of severe hypokalaemia include constipation, ileus, ventricular and atrial tachycardias, weakness, hypotonicity and rarely an ascending motor paralysis with ventilatory failure. We describe a patient who presented with severe diarrhoea, hypokalaemic normal anion gap metabolic acidosis and hypophosphataemia. He developed stridor and respiratory failure following early correction of his hypokalaemia which was successfully managed with endotracheal intubation, mechanical ventilation and further correction of his fluid and electrolyte abnormalities.

Journal Article↗
PubMed1991

[Inspiratory stridor as the only symptom of esophageal foreign body].

An eleven month old infant girl presented with a two-month history of inspiratory stridor. Analysis of her symptoms indicated airway obstruction located in the middle of the trachea; a barium esophagogram revealed an esophageal foreign body with tracheal compression. By endoscopy, a chestnut shell was extracted from the esophagus. In infancy, esophageal foreign bodies may cause mainly respiratory symptoms.

Airway Obstruction↗
PubMed1991

[Episodic laryngeal dyskinesia: a functional cause of stridor].

We present five cases of episodic laryngeal dyskinesia. This term describes an entity associating acute dyspnea with inspiratory and/or expiratory stridor, sometimes ending in acute respiratory distress. The origin of this trouble comes from paradoxal contraction of the vocal cords, which is reversible and recurrent. The clinical presentation presents as a differential diagnosis for serious disorders such as bronchial asthma, laryngeal oedema, or stenosing lesions of the upper airways. The definitive diagnosis is made by excluding organic pathology, by visualising laryngeal spasm and by the inspection of flow volume curve. A comparison of all the published papers since 1974 reveals the benign nature of this "noisy" disorder and to bring out the diagnostic criteria thus avoiding too aggressive an approach in these patients.

Adolescent↗
PubMed1988

[Severe congenital laryngeal stridor. Endoscopic surgical treatment].

Congenital laryngeal stridor or laryngomalacia is a congenital disease causing an usually shrill and solitary inspiratory noise, sometimes associated with disorders of deglutition and dyspnea when crying. Most often, the symptoms spontaneously disappear before the age of two. However, some cases are very severe, with permanent dyspnea, leading to tracheal intubation or tracheotomy. To avoid the risks of prolonged tracheotomy in infants, a new surgical technique has recently been described, beside epiglottectomy and hyomandibulopexy, now abandoned. This is simple endoscopic section and resection of the ary-epiglottic folds. Preliminary results in 15 patients seem to show the superiority of this so-called epiglottoplasty technique. Except in one case with supraglottic oedema attributed to a major gastro-esophageal reflux, all patients recovered from their dyspnea with this procedure. Extubation usually was rapidly feasible and the post-operative period was uneventful. The patients are kept in hospital for 2 to 5 days, and an antibiotic and anti-reflux treatment is recommended. This procedure is advocated as a treatment of choice of "laryngomalacia" with severe dyspnea.

Dyspnea↗
PubMed1986

Stridor due to cricoarytenoid arthritis in pauciarticular onset juvenile rheumatoid arthritis.

A 2-year-old girl developed severe inspiratory and expiratory stridor 2 months after onset of pauciarticular juvenile rheumatoid arthritis (JRA). Direct laryngoscopy demonstrated that both vocal cords were immobile and approximated to each other in the midline secondary to arthritis of the cricoarytenoid joints. High dose corticosteroid therapy resulted in clinical and laryngoscopic improvement and tracheostomy was avoided. Cricoarytenoid arthritis can be a life threatening complication in JRA. Early institution of corticosteroids appears to be the treatment of choice.

Arthritis, Juvenile↗
PubMed1985

[Vocal cord paralysis with stridor caused by interarytenoid fibrosis--a complication of intensive care].

Translaryngeal long-term intubation in intensive-care patients was followed in three cases by immobilisation of the vocal cords and severe stridor, caused by a fibrotic scar between the processus vocales and the posterior commissure. Different surgical approaches are described. Endoscopic incision of the scars is recommended. Most important is a bolting technique for several consecutive days.

Adult↗
PubMed1982

[Inspiratory stridor due to vocal cord paralysis in children with myelomeningocele and hydrocephalus].

Inspiratory stridor in combination with myelomeningocele and increased intracranial pressure is caused by bilateral vocal cord paralysis in children with Arnold-Chiari-deformation. The diagnosis of bilateral vocal cord paralysis can be established by direct laryngoscopy performed without general anesthesia. As emergency measures naso-tracheal intubation, tracheostomy and immediate ventricular puncture are recommended. Reduction of intracranial pressure has always to be performed within 24 hours. The bilateral vocal cord paralysis is totally reversible if the inracranial pressure is decreased timely. The bilateral vocal cord paralysis becomes irreversible when degeneration of the nucleus ambiguus occurs secondary to peripheral lesions of the nervus vagus.

Arnold-Chiari Malformation↗
PubMed1980

Supglottic coccidioidomycosis presenting with persistent stridor.

Localized laryngeal coccidioidomycosis has been rarely reported and usually is associated with disseminated disease. This paper has described localized subglottic coccidioidomycosis in a 13-month old white boy who presented with persistent stridor. Medical management including intravenous amphotericin B resulted in complete eradication of the subglottic lesion.

Amphotericin B↗
PubMed1993

Stridor in the infant and child. Assessment, treatment.

The health care team caring for the child with stridor must be familiar with congenital abnormalities of the airway and airway problems resulting from infections and foreign bodies and their typical presentation and endoscopic findings. Teamwork and communication are the keys to safe diagnosis and management of these children.

Child↗
PubMedDate not supplied

[Intermittent functional stridor as a manifestation of hysterical neurosis].

In hysteric episodes, one can observe breathing problems as a result of paradoxical joining of the vocal cords while inhalation. The paper provides data which may help to make differential diagnosis between true laryngeal stenosis treated by tracheostomy and intermittent functional stridor in hysteria treated psychotherapeutically.

Adolescent↗
PubMed1996

Congenital stridor.

Congenital stridor is a common problem that has many very different etiologies. A complete history and physical examination will allow the astute clinician to direct the laboratory evaluation efficiently. Establishing a relationship with a specialist will be useful for consultation when considering a referral in questionable situa.

Gastroesophageal Reflux↗
PubMed1998

[A case of adenoid cystic carcinoma presenting with stridor and which was treated by reversed gamma type stent placement].

A 66-year-old man was admitted to Saiseikai Suita Hospital with stridor and dyspnea. The flow-volume curve showed central airway obstruction pattern. Although a P-A chest roentogenogram was normal, chest tomogram demonstrated a tumor shadow in the lower portion of the trachea. Chest CT examination demonstrated a tumor arising from the posterior wall of the lower portion of the trachea and stenosis with wall thickening extending from the lower portion of the trachea to both main bronchi. Bronchofiberscopic examination revealed polypoid tumors obstructing the lower portion of the tracheal lumen, and biopsy was subsequently performed. On the night immediately after the bronchofiberscopic examination, the patent suddenly choked, and emergency intubation was performed. Adenoid cystic carcinoma was diagnosed on biopsy and we decided to place a spiral Z-stent. A reversed gamma-type stent placement was used. The first spiral Z-stent extended from the trachea to the right main bronchus and the second spiral Z-stent was placed in the left main bronchus through the first stent. Following the spiral Z-stent placement radiotherapy was performed. Spiral Z-stent placement resulted in dilation of the stenotic trachea and both main bronchi.

Aged↗