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At least 253 records · Page 14Linked to original sources
PubMed1982

[Growth dyssymmetry of Shigella flexneri 6(762 S) cells].

Dyssymmetry in the growth of the initial and secondary shigella cells at early stages of the formation of microcolonies has been revealed by the method of dynamic morphometry in serial photographs obtained by time-lapse microfilming. Electron-microscopic autoradiography has allowed revealing dyssymmetry in the assimilation of exogenous H3-thymidine, H3-threonine, H3-asparaginic acid by different halves of shigella cells in the process of their growth and preparation for mytosis. These results suggest that the shigellae are divided into two unequal cells: the "mother" cell and the "daughter" one.

Aspartic Acid↗
PubMed1982

[Microcirculation in the liver in normal blood oxygenation and hypoxemia].

Vital contact microscopy and microfilming were used to measure the blood flow velocity in hepatic sinusoids 11 +/- 1 microns in diameter. The white blood cells moving in the axis stream of the red cells were used as the markers to measure linear flow velocity. In normoxia it was equal to 0.29 +/- 0.01 mm/sec and the volumetric flow rate in a liver sinusoid as calculated from this value was about 1.65 . 10(-3) microliters/min. Breathing with the mixture of nitrogen and 7% oxygen evoked a decrease of arterial blood pressure down to 80-70 torr and oxygen tension in a. femoralis and v. porta down to 45 +/- 3 torr and 18 +/- 3 torr, resp., as compared with 77 +/- 3 and 41 +/- 2 torr in normoxia. The mean sinusoidal blood flow velocity increased by nearly 12% only (p less than 0.01). This suggests that the increased oxygen extraction is the main compensatory mechanism in liver tissue hypoxia.

Animals↗
PubMed1995

[Application of patient card technology to health care].

The potential benefits of patient card technology in improving management and delivery of health services have been explored. Patient cards can be used for numerous applications and functions: as a means of identification, as a key for an insurance payment system, and as a communication medium. Advanced card technologies allow for the storage of data on the card, creating the possibility of a comprehensive and portable patient record. There are many types of patient cards: paper or plastic cards, microfilm cards, bar-code cards, magnetic-strip cards and integrated circuit smart-cards. Choosing the right card depends on the amount of information to be stored, the degree of security required and the cost of the cards and their supporting infrastructure. Problems with patient cards are related to storage capacity, backup and data consistency, access authorization and ownership and compatibility. We think it is worth evaluating the place of patient card technology in the delivery of health services in Israel.

Delivery of Health Care↗
PubMed1996

[The antiulcer effect of corticosteroids and the blood flow in the gastric mucosa].

Intravital microfilming by means of dark-field contact epiobjective was used for investigation of the role of gastric microcirculation in antiulcerogenic action of corticosteroids in rats. Microvascular volume blood flow velocity was calculated from measurements of red cell velocity and vessel diameters. There was compared gastric submucosal and mucosal microcirculation as well as gastric ulceration after water-immersion stress in rats with normal function of hypothalamo-pituitary-adrenocortical (HPA) axis and in rats with insufficiency of corticosteroid production, induced by blockade of the HPA axis. The 3h stress induced gastric ulcerations, dilatation of mucosal microvessels and a significant decrease of volume blood flow in submucosal microvessels. These changes were greater in rats with blockade of HPA axis than in animals with normal corticosteroid production. Replacement corticosterone therapy decreased the effects of HPA axis blockade. Thus corticosteroids which are produced during stress restricted the reduction of volume blood flow in submucosal microvessels, the dilatation of mucosal microvessels as well as gastric ulcerations. These findings indicate that corticosteroids exert their antiulcerogenic action via the mechanism of improvement of gastric microcirculation.

Adrenal Cortex Hormones↗
PubMed1996

[Corticosteroids limit the decrease in blood flow velocity in the microvessels of the gastric submucosal layer during stress in rats].

To evaluate the influence of stress and corticosteroids on blood flow velocity in gastric submucosal microvessels it was used intravital microfilming by means of a dark-field contact epiobjective. The plasma filled "gap" in the continuous erythrocyte flow were used as the markers to measure linear blood flow velocity. In unstressed rats the linear blood flow velocity was equal to 2.0 +/- 0.08 mm/sec (156 microvessels, 6.4 +/- 0.14 micron in diameter), calculated volume blood flow--(6.9 +/- 0.48) x 10(-5) microliters/sec. Stress (water immersion and restraint) led to a decrease of blood flow velocity in microvessels to more than double. A stress-induced decrease of blood flow velocity was more greater in rats with insufficient corticosteroid production. The linear velocity of blood flow of these rats was lowered to 0.6 +/- 0.06 mm/sec, volume blood flow to (1.6 +/- 0.21) x 10(-5) microliters/sec. Replacement corticosterone therapy increased the blood flow velocity. It was concluded that stress decreases blood flow in microvessels of gastric submucosa. The results revealed that glucocorticoids take part in normalization of blood flow of gastric microvessels during stress and this action can provide antiulcerogenic effect of corticosteroids which was shown by us previously.

Adrenal Cortex Hormones↗
PubMed1995

[The effect of stress and corticosteroids on the rate of blood flow in the microvessels of the gastric muscular coat of rats].

In order to evaluate the influence of stress and corticosteroids on blood flow velocity in microvessels of muscular coat of the stomach we applied intravital microfilming by means of a dark-field contact epiobjective. The plasma filled "gap" in the continuous erythrocyte flow were used as the markers to measure linear flow velocity. In unstressed rats the velocity was equal to 1.76 +/- 0.07 mm/sec (182 microvessels, 3.7-8.7 mu in diameter). Stress (water immersion and restraint) led to a decrease of blood flow velocity in microvessels to more than double. A stress-induced decrease of blood flow velocity was greater in rats with insufficient corticosteroid production. The velocity of blood flow in these rats was lowered to 0.62 +/- 0.04 mm/sec. Replacement corticosterone therapy increased the blood flow velocity. It was concluded that stress decreases blood flow in microvessels of muscular coat of the stomach as well as in microvessels of mucosa. The results revealed that glycocorticoids take part in normalisation of blood flow of gastric microvessels during stress.

Adrenal Cortex Hormones↗
PubMedDate not supplied

[The evaluation of the mechanism of thrombocytopoiesis from the viewpoint of the phenomenon of emperipolesis].

Cytolytic aggressiveness of bone marrow leucocytes towards megakaryocytes was documented with the aid of a phase contrast Zeitraffer microfilming of alive cells technique. The difference was found between the process above and cell interaction through emperipolesis. The author comes to the conclusion that leucocytes play an active part in platelet production regulation. The data obtained enable new pathognomic methods of treatment to be developed and preventive measures to be devised against blood platelet cytosis involved in the morbid course of quite a number of medical conditions, and further studies to be made.

Animals↗
PubMedDate not supplied

Summaries of clinically relevant studies of dental materials from the 1995 meeting of the American Association for Dental Research.

The object of this review is to provide clinical research information. Each year, several hundred dental materials research papers are presented at the AADR meeting. For additional information about these studies, consult the abstracts published in J Dent Res 1995;74(special issue):12-257. Papers presented to the AADR Dental Materials Group are published in microfilm and microfiche, available in dental school libraries.

Composite Resins↗
PubMed1996

[The adaptation-protective effect of corticosteroids on the rat gastric mucosa and its mechanism].

The influence of stress-induced corticosteroid production on gastric ulceration, blood flow velocity in gastric microvessels and blood pressure was studied in rats. The role of plasma corticosteroids was investigated by means of blockade of the pituitary-adrenocortical system (PACS) and following corticosterone replacement therapy (400 mu/100 g b.w.). The blockade which was induced by Fi. hydrocortisone administration (7 days before stress, 30 mg/100 g b. w.) resulted in an insufficient corticosteroid production. To evaluate the influence of corticosteroids on blood flow velocity in gastric microvessels of muscular, submucosal and mucosal coats it was used intravital microfilming by means of a dark-field contact epiobjective. Stress (water immersion + restraint) induces an ulceration, a decrease in the systemic arterial blood pressure (3 h after stress onset) and a decrease in blood flow velocity in the gastric microvessels (3 h after stress onset). In rats with insufficient corticosteroid production stress-induced ulceration, a decrease in blood pressure and gastric blood velocity were more greater than in rats with intact PACS. Replacement corticosterone therapy corrected all parameters. The results revealed that antiulcerogenic effect of stress-induced glucocorticoid production is realised owing to normalisation of gastric blood supply which is provided by an increase in systemic blood pressure.

Adaptation, Physiological↗