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[Heredofamilial allergic predisposition in favism].
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Favism in Cyprus; analysis of 67 cases admitted to Nicosia General Hospital during the last 3 years.
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[Ictero-hemoglobinuric favism as immunological hemolytic anemia; serological findings on atypical antibodies].
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[Blood colloid changes in favism].
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[A case of late-onset favism].
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[Favism in Polish families].
Four cases of fawism are presented. The disease was seen in one male patient, one homozygote and in 3 carriers of G6PD deficit. Diagnostic procedures, course of the haemolytic crisis in these patients, and possibility of prophylaxis in the families with fawism are discussed.
[Epidemiologic study of acute favism cases (G6PD deficiency) in the Autonomous Community of Murcia during 1983-1987. Report of a case].
This study reminds primary care teams of the fundamental importance of epidemiological histories for diagnosing broad bean disease in rural areas in which broad beans are regularly harvested and the patient's nationality lead one to suspect this illness. A retrospective study of the consumption of broad beans in the region of Murcia is made for the period 1983-1987, the variables of which are: Age, sex, geographical distribution, epidemiological history, diagnosis based on a clinical analysis, need for transfusion, treatment, progress and family case study. Among the conclusions, the greatest number of cases occur in males ranging from 0 to 10 years of age, residents basically of Murcia and the surrounding districts, with a prior history of having ingested broad beans, showing symptoms, revealed through a clinical analysis, which are compatible with are compatible with an hemolytic anemia, for which it was necessary to provide transfusions for a considerable number of cases, achieving good progress and a treatment based on the procedure of fundamentally eliminating broad beans from their diet and certain medicines, and lastly, a later study of the members of these patients' families.
Recent advances in the biochemistry of favism.
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[Erythrocyte antioxidant enzymatic systems and serum beta-glucuronidase in subjects with favism. Preliminary results].
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The erythroenzymopathies. With particular reference to glucose-6-phosphate dehydrogenase deficiency and favism.
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[Glucosephosphate dehydrogenase deficiency in erythrocytes and leukocytes of patients with congenital nonspherocytic hemolytic anemia and favism].
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Prevention of favism in the rural areas.
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[Favism, hemoglobin H, alpha-thalassemia and hemoglobinopathy in various Sardinian families. Studies on glucose-6-phosphate dehydrogenase].
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Glucose-6-phosphate-dehydrogenase deficiency and favism.
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