Electrical injury: electrical engineer's perspective and an historical review.
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Anastomosis between the first to the fourth dorsal metacarpal arteries was observed in 43 hands of adult corpses. It was found that 86% of the first dorsal metacarpal artery arises from the radial artery, 14% is replaced by the main artery of thumb or superficial branches of the radial arteries; eighty-six percent of the second dorsal metacarpal artery originates from the dorsal carpal artery. Sixty-seven percent of the third and the fourth dorsal metacarpal arteries is formed by anastomosis of the dorsal carpal artery and its deep palmar arch. The first to the fourth dorsal metacarpal arteries have 4 to 8 cutaneous branches and proximal and distal anastomoses. It is concluded that the dorsal metacarpal arteries are characterized with multiple sources and abundant anastomoses around the metacarpophalangeal joints, which can be used with advantage in the repair of soft tissue defects of the area.
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Electrical injury is uncommon, and cardiac involvement occurs in a minority of patients who are injured. Cardiovascular complications in the electrically injured patient, however, can be devastating. Because the presentation of electrical injury to the heart can be atypical in nature, delayed in onset, or obscured by other trauma, caution must be exercised in the interpretation of initial symptoms, ECGs, monitor strips, and cardiac enzymes. Whenever there is doubt concerning the presence of cardiovascular injury, the patient should be monitored. When cardiac complications are in evidence, expectant management is generally sufficient. When specific management of cardiac arrest, other dysrhythmias, myocardial necrosis, hypertension, or conduction abnormalities is required, standard therapeutic regimens are generally appropriate. In managing apparent acute myocardial infarction, however, the possibility that the injury is not of ischemic origin must be considered. Reperfusion techniques should be applied only when occlusive coronary thrombosis is strongly suspected or angiographically confirmed. The risk of developing chronic cardiac disease after electrical injury to the heart is unknown. Patients who sustain cardiovascular injuries should be followed for at least 12 months and avoid elective surgery for 6 months after the incident.
An analysis of 45 victims of fatal lighting strike revealed the incident occurred most frequently in the early afternoon during midsummer in a field of one form or another. The scene of death, damage to clothing, and alterations of metallic objects on the victim are described. A terminal cardiac rhythm of ventricular fibrilation was recorded in half for whom data were available, and asystole was found in 40%. All but four had cutaneous injuries, and nearly one-third had pathognomonic patterns of erythematous arborization. When examined, tympanic membranes were found to be ruptured in > 80%. This study also revealed that craniocerebral injury and cardiac contusion can be serious direct consequences of lightning strike. A correlative approach to the investigation and autopsy of lightning victims is suggested.
OBJECTIVE: To describe the epidemiology of electrical fatalities in Western Australia between 1976 and 1990. DATA SOURCES: Data for the study were gathered from hospital records, autopsy reports and findings from State Energy Commission and coronal investigations. DATA EXTRACTION: Information that was abstracted included age, sex, occupation, voltage, history of incident and autopsy findings. DATA SYNTHESIS: There were 104 victims. Death occurred most frequently in young men exposed to low voltage current during summer, and nearly half the fatalities occurred in the workplace. Water was present in up to 52% of fatalities. Without exception, all victims developed a lethal arrhythmia at the time of exposure to the electric shock, which usually resulted in death at the scene. CONCLUSION: When a victim is exposed to a fatal electric shock, the lethal arrhythmia occurs at the time of electrocution and is just as likely to occur at home as in the workplace. Electrical fatalities can be prevented largely by the use of residual current devices.
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High-energy electrical injury, whether from lightning strike or electrical shock, occurs primarily in the workplace. Neurological dysfunction can be a devastating complication of electrical injury. A review of the literature was undertaken to develop a better understanding of the epidemiology, mechanisms of injury and neuropathology associated with this type of injury. The numerous challenges inherent in the management of these complex cases were illustrated by three case studies.
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