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At least 217 records · Page 12Linked to original sources
PubMedDate not supplied

[A treatment-prevention combination in the therapy of chronic parenchymatous parotitis in children].

A combined treatment-and-prevention complex includes infusion of ol. semeni fructus Rosae into the diseased gland via its duct, peroral administration of retinol acetate and gland electrophoresis with 1% ascorbic acid solution. Up to 10 years follow-up of 118 children confirmed high efficiency of the procedure in 84,75% of the cases. Treatment duration in cases of exacerbations of chronic parotitis was reduced by 7.9 days per year per 1 patient.

Child↗
PubMed1988

The swollen neck. Cervical lymphadenitis, parotitis, thyroiditis, and infected cysts.

Swelling of the neck can result from a variety of causes, both infectious and noninfectious. This article describes the major causes of neck swelling from the more common infectious processes in the neck--cervical lymphadenitis, parotitis, thyroiditis, and infected cysts. The etiology and pathogenetic considerations, clinical manifestations, differential diagnosis, therapy, and prevention are discussed for each of these conditions.

Cysts↗
PubMed1979

Acute bilateral suppurative parotitis due to Haemophilus influenzae. Report of two cases.

Acute bilateral suppurative parotitis developed in two patients with documented cirrhosis of the liver while they were in the hospital. Gram's stain and culture of purulent material obtained from the orifice of Stensen's duct disclosed Haemophilus influenzae as the sole isolate. Both patients were treated successfully with local care and antibiotics. To our knowledge, with one possible exception, infection of the parotid gland due to this organism has not been described previously.

Acute Disease↗
PubMed1985

[State of microcirculation and the system of hemostasis in Sjögren's disease, Sjögren's syndrome and chronic parenchymatous parotitis].

Microcirculation and hemocoagulation were examined in 88 patients with Sjögren's disease and Sjögren's syndrome (SD and SS) in 20 patients with chronic parenchymatous parotitis (CPP) without a "dry syndrome", and in 20 healthy donors by conjunctival angioscopy, capillaroscopy of the nail matrix, and histological study of the salivary glands. Hemostasis was explored with the use of 37 tests. SD and SS patients manifested significantly more pronounced microcirculatory disorders versus donors (P less than 0.001) and CPP patients (P less than 0.001). In patients with hypergammaglobulinemic purpura, microcirculatory disorders were found to be enhanced (P less than 0.05). An indirect relationship between these disorders and the degree of lacrimation lowering was established. Intravascular disturbances increased as the process activity rose and diminished under the effect of prednisolone therapy. It is assumed that microcirculatory and hemocoagulation disorders play an important role in the pathogenesis of SD and SS.

Adult↗
PubMed1979

[Suppurative parotitis in newborns (author's transl)].

Neonatal suppurative parotitis is described in 6 patients followed by a review of the literature. About 30-40% of the patients were premature, dehydration constitutes a predisposing factor. Infection occurs usually by bacteria ascending the duct. In 5 of our 6 patients infection had been caused by staphylococci; E. coli was found in 2 patients and pseudomonas in 1 patient. The parenteral therapy with antibiotics is usually sufficient.

Dehydration↗
PubMed1978

The surgical management of recurrent parotitis.

Twenty-eight parotidectomies were carried out upon 26 patients with chronic parotitis. The lesion considered is chronic inflammation of the parotid gland associated with such intraglandular defects as sialadenitis secondary to ductal obstruction by calculi, cellular debris, stenosis or infiltrating lesions, that is, Mikulicz's or Sjögren's syndromes and sialoangiectasis, either primary or secondary to obstruction of the duct. Seventeen near total parotidectomies were done without significant complications or a recurrence of symptoms. Eleven superficial parotidectomies were performed in which symptoms recurred on the 12th postoperative day in one patient. No permanent weakness of the facial nerve occurred in any of the 28 parotidectomies. Based upon this experience, near total parotidectomy with removal of the parotid duct can be performed safely and should be the procedure of choice in patients with chronic, relapsing parotid sialadenitis.

Chronic Disease↗
PubMed1982

[Surgical therapy of chronic parotitis].

A short survey of the therapeutical possibilities available in cases of chronic parotitis is followed by a discussion of the results achieved after surgical treatment. This discussion is based on the findings obtained in follow-up checks. The - very strict - indication for surgery was not made until all conservative measures had been exhausted and thus it involved only 2.4% of 380 surgical patients suffering from various diseases of the parotis gland. It was possible to achieve a sustained total absence of complaints in all cases, with partial or subtotal resection being sufficient in most instances. Although postoperatively partial loss of the N. facialis function occurred relatively frequently, permanent damage was not observed in any single case.

Abscess↗
PubMedDate not supplied

[The participation of prostaglandin F2 in the peroxidation damages and antiperoxidation protection of the parotid gland in acute nonepidemic parotitis].

Effects of prostaglandin F2 on the major components of the antioxidative system (levels of malonic dialdehyde and lipid peroxides, activities and content of glutathione enzymes and nonenzymes (glutathione peroxidase, glutathione reductase, and NADPH (NADH), oxidized and reduced glutathione) were studied in experiments with 60 dogs in which acute nonepidemic parotitis was modeled. A varying capacity of the cells of the parotid gland to resist the detrimental effects of the disease and specific features of prostaglandin F2 effect on this capacity were revealed: injection of prostaglandin F2 stimulated both lipid peroxidation and the activity of the antioxidative system and led to cellular injury in the presence of a relatively high level of cellular metabolism, preserving cellular viability on condition of inhibited rate of metabolic reactions, this being the principal factor for the parotid gland, among other factors determining the cytoprotective effect.

Acute Disease↗
PubMed1994

[Acute suppurative parotitis caused by Pseudomonas aeruginosa].

Acute suppurative parotitis is a rare infection which usually occurs in hospitalized patients. Risk factors are dehydration, old age, the postoperative state and immunocompromised states. We describe a 66-year-old woman with acute myeloid leukemia who developed this condition. Pseudomonas aeruginosa was cultured from the blood and the orifice of Stensen's duct. Recovery followed a combination of antistaphylococcal medication with clindamycin, ofloxacin and gentamicin. We stress the need to recognize Gram-negative bacteria as possible pathogens in this condition.

Acute Disease↗
PubMed1994

Intraductal tetracycline therapy for the treatment of chronic recurrent parotitis.

Chronic recurrent parotitis (CRP) is recurrent parotid inflammation with non-obstructive sialectasis. Therapies which produce acinar atrophy or remove the acini are effective in treating CRP. Parotidectomy, tympanic neurectomy, duct ligation, and radiation therapy have either a low success rate or a high risk of morbidity. Intraductal antibiotic instillation has been proposed as a possible method of treatment. We hypothesized that the cytotoxic effects of tetracycline could produce acinar atrophy. A double-blind experiment of intraductal tetracycline instillation was performed in ten rabbits. Acinar atrophy and acute inflammation were found in 40% of the tetracycline treated glands; controls had a complete absence of these histologic changes. These results support the use of tetracycline instillation to produce acinar atrophy and therefore, intraductal tetracycline may be an effective, low-risk therapy for CRP. The clinical features of CRP will be reviewed and therapeutic implications discussed.

Adult↗
PubMed1993

[Sialographic changes in chronic obstructive parotitis related to clinical and pathologic findings].

This study presents a study on 23 patients with chronic obstructive parotitis (COP) in whom the sialographic changes are related to the clinical and histopathological features. The sialographic changes are divided into 4 types. The stimulated parotid flow rate (SPFR) is normal in sialography Type I patients, while SPFR is significantly reduced in Type II, III, and IV patients, as compared with healthy persons. The changes under light microscope consist of irregular dilatation and chronic inflammation of main and branching ducts with saliva deposits in the enlarged luminal system. Ultrastructural changes include degeneration of ductal epithelial cells, marked proliferation of periductal fibrous tissue, and moderate electron-dense deposits in the lumen of the dilated peripheral ducts. The extent of the histopathologic changes largely parallel the severity of the sialographic changes. These findings indicate that COP is a common inflammatory disease and that the sialographic classification reflects the severity of the disease.

Adult↗
PubMed1993

[Chronic recurrent parotitis in children--diagnosis and therapy].

The author presents a report on a group of 11 children with chronic recurrent parotitis, treated at the ENT department in Cadca. The author emphasizes the importance of sialography for establishment of the diagnosis of this disease and draws attention to therapeutic possibilities.

Adolescent↗
PubMed1993

[Juvenile recurrent parotitis].

Juvenile recurring parotitis is a relatively rare condition. The clinical picture as illustrated by a characteristic case history is reviewed. The condition shows a tendency to remission around puberty. Sialographic examination is characterised by the configuration of the parotid gland said to resemble a bunch of grapes. Biochemical tests do not contribute to the diagnosis which is established by the clinical picture and the typical sialographic findings. No systemic disease can be demonstrated in the patients who are healthy in other respects. The cause of the condition appears to be congenital sialectic changes localised to the parotid gland with resultant secondary ascending bacterial infection. In treatment during the acute stages, attempts should be made to stimulate secretion of saliva with sour foodstuffs and chewing gum. Treatment with penicillin for one week is recommended. On account of the benign character of the condition and the good long-term prognosis, operative intervention should not prove necessary.

Age Factors↗
PubMed1996

Salivary factors in children with recurrent parotitis. Part 1: Salivary flow rate, buffering capacity and inorganic components.

The parotid flow rate, buffering capacity and inorganic components in saliva from 17 children with juvenile recurrent parotitis (JRP) in periods free of symptoms and in healthy controls of the same number, sex and age were analysed after gustatory stimulation with 1%, 2% and 6% citric acid. There was a great individual variation in all analysed variables in the JRPs, as well as in the controls. The flow rate, the buffering capacity of saliva and the ion concentration of phosphate were significantly lower for the JRP-children than for the controls, while the concentration of chloride, sodium and copper were raised (p < 0.01-0.001). No statistical differences were found for the pH, the calcium, the potassium, the magnesium and the zinc concentrations. The results suggest a longterm effect on the parotid saliva in the JRP-cases due to the disease.

Adolescent↗
PubMed1996

Parotidectomy for the treatment of chronic recurrent parotitis.

OBJECTIVE: To assess the success of superficial parotidectomy in the treatment of chronic parotitis (a potentially debilitating disease heralded by recurrent painful swellings) and purulent sialorrhea. DESIGN: A retrospective review. METHODS: Ten patients who were managed by superficial parotidectomy after failure of all conservative measures at the Department of Otolaryngology, McGill University were studied. RESULTS: Eight had complete resolution of their disease, and two had minor persistence on follow-up. None of them had permanent facial nerve injury. CONCLUSION: We therefore advocate superficial parotidectomy for this condition on failure of conservative measures.

Adolescent↗
PubMed1996

[Ultrasonography: a complement to sialography in recurrent chronic childhood parotitis].

The objective of this research is to compare the ultrasonographic (USG) and sialographic contributions for the diagnosis of Recurrent Parotitis in Childhood (RPC). Bilateral USG of the parotid gland was performed among 16 children with RPC and 16 healthy children. The image of the normal parotid glands was compared with the image of the parotid gland with RPC. A pathologic USG pattern for RPC was obtained which consisted in a homogeneous, micronodular parenchyme with a diffuse increase density, sometimes with multiples hypoechogenous areas, that were considered solid hypoechogenous nodules suggesting inflammatory nodes. A glandular contour finely granulated was described with normal peri-parotidareas and with aponeurotic-muscular planes. Then 16 USG of the parotid gland of the children with RPC were compared with 16 Sialography previous obtained. The possibilities of diagnosis of the disease with both techniques was not different (p > 0.05). When the clinic was correlated with the USG it was possible to obtain parameters of severity of the glandular damage and to visualise the degree of changes of the parenchyma of each child with RPC. In some cases, bilateral damage was observed in children with clinical unilateral symptoms. The USG was performed in children in the acute phase of disease, visualising the microabscess in the gland and confirming the previous diagnosis. Finally, it was possible to conclude the utility of USG in the study of the disease. Being a not invasive exam, harmless, unpainfully echography, was easily accepted by the child. Also, we obtain information not only about the diagnosis of the disease, but also about the severity, prognosis, treatment and follow-up of the infant patients.

Abscess↗
PubMedDate not supplied

Clinical and immunological features of the HIV infection associated with chronic hypertrophic parotitis in children.

The study refers to children of 0-15 years of age, infected with HIV and who developed a chronic hypertrophic parotitis (CHP), admitted to the "Colentina" Clinic of Infectious Diseases--Paediatrics in Bucharest, between January 1, 1990 and May 15, 1993. Among the total number of 579 HIV infection cases hospitalized in the above-mentioned period, 135 were associated with CHP, hence an incidence of 23.3%. The HIV infection was defined by two ELISA-positive assays, confirmed by a Western-blot test. No specific laboratory test for the diagnosis of CHP in the course of HIV infection was available. The detection of a uni- or bilateral painless parotid enlargement, without signs of skin inflammation in HIV-infected children, was conclusive for the diagnosis of CHP. IgG type anticytomegalovirus antibodies were detected in 41.17% (7/17) and anti-Toxoplasma antibodies in 50% of the tested cases (4/8). The immunogram performed in 85 children showed increased IgG values in 92.94% of cases (79/85) and increased IgM values in 85.88% (73/85). There was recorded a significant increase in the levels of immunoglobulins, especially of IgM, which exceeded 13 times the normal values. The CD8 cells were frequently normal or increased (94.44%, respectively 34/36). CHP appeared before a marked deterioration of CD4 cells, simultaneously with the CD8 cells proliferation. CHP developed at a stage of the HIV infection when the medium-term prognosis was still considered favourable.

Adolescent↗