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At least 199 records · Page 11Linked to original sources
PubMed1986

Ultrasound evaluation of polyhydramnios and twin pregnancy.

We analyzed the ultrasound examinations and medical records of 75 pairs of twins who were delivered between January, 1983, and December, 1984, to study the relationship between increased amniotic fluid volume, fetal abnormalities, and preterm labor. Ten of these 75 twin pregnancies demonstrated elevated amniotic fluid volume that persisted throughout pregnancy. Total intrauterine volumes were elevated in these cases, and nine of the ten pregnancies were abnormal. In addition, it was noted that elevation of the amniotic fluid volume alone did not explain the high rate of preterm labor and delivery in twin gestations.

Congenital Abnormalities↗
PubMed1990

Pathophysiology of congenital diaphragmatic hernia: I. Renal enlargement suggests feedback modulation by pulmonary derived renotropins--a unifying hypothesis to explain pulmonary hypoplasia, polyhydramnios, and renal enlargement in the fetus/newborn with congenital diaphragmatic hernia.

Survival of newborns with congenital diaphragmatic hernia (CDH) is largely dependent on the severity of pulmonary hypoplasia (PH) present at birth. Intrathoracic compression by the herniated abdominal viscera is thought to be the primary factor involved in the pathogenesis of the PH in CDH. Humoral and/or amniotic pulmonary growth factors (PGF) have been hypothesized to play a role in normal fetal pulmonary development and may be involved in the pathogenesis of CDH as well. The hypothesis of this paper is that growth of the fetal lung is stimulated by a PGF produced by the kidneys, which is modulated by a feedback signal from the lungs, a pulmonary derived renotropin (PDR). In the fetus with CDH, the lungs may be unable to respond to PGF due to compression by the herniated abdominal viscera. Theoretically, PH associated with CDH would maximally stimulate this feedback loop to release more PDR, resulting in continual stimulation of the kidneys and renal enlargement. If such a scheme plays a role in the in utero pathophysiology of CDH, then newborns with CDH should have enlarged kidneys. To investigate this hypothesis, we reviewed 30 autopsy cases of newborns with CDH and analyzed their kidney weights versus body weights, using historical data as control. Kidney weights in CDH cases were greater than the control population in 77% of the cases; 57% of kidney weights were more than one standard deviation above control values. Adjusted group mean kidney weights were 29.8 g (+/- 1.0 SE) in CDH cases and 25.9 g (+/- 1.5 SE) in the control population (P less than .04). These data support our hypothesis and demonstrate that in newborns with CDH and morphologically normal kidneys, there is significant renal enlargement associated with CDH. The presumed mechanism of this renal enlargement, as well as its relationship to normal and aberrant pulmonary growth and regulation are discussed. If such a selective PGF exists, its therapeutic implications for fetuses and newborns with PH are considerable.

Analysis of Variance↗
PubMed1983

Ultrasound observations of a malignant cervical teratoma of the fetus in a case of polyhydramnios: case history and review.

A case report is given concerning an extremely rare case of malignant teratoma of the neck with mature and immature metastatic lesions in the lungs in an immature fetus. The ultrasound observations are reported. The differential diagnosis of masses in the fetal neck, as can be observed by ultrasound, is discussed, with a review of the literature on this subject.

Female↗
PubMed1984

[Ultrasonic diagnosis of fetal ascites with polyhydramnios resulting from partial small intestine atresia and secondary intestinal wall perforation].

A primigravida was admitted to hospital in the 31st week of pregnancy after normal pregnancy course, with premature rupture of the amnion. Ultrasonography revealed a marked fetal ascites as well as polyhydramnion. Pregnancy was terminated via Caesarean section because of threatening intrauterine asphyxia. The asphyctic, hypotonic infant, which did not show any visible malformation, was intubated and the ascites was punctured. After a brief recovery, the infant died 24 hours post partum. Autopsy revealed partial atresia of the small intestine with secondary perforation of the intestinal wall, as well as a fibrinous-purulent peritonitis. Pathogenesis of the foetal ascites, as well as the possibilities of diagnosis via sonography in case of fetal malformations, are discussed on the basis of the present case and previous literature.

Adult↗
PubMed1991

[Obstetric and ethical-legal problems in a twin pregnancy with anencephalus and polyhydramnios of the leading twin beyond the 28th week of pregnancy].

Malformations are found twice as often in twins than in singletons. In the following case, we report on a twin pregnancy with anencephaly and polyhydramnion of the leading twin I, which was first diagnosed after 28 weeks of pregnancy. The foetal disease of twin I led to an appreciable dyspnoea of the mother that exacerbated to an incipient respiratory failure by the thirty-second week of pregnancy. The projected termination of pregnancy by Caesarean section based on a maternal indication was not performed, but instead a selective foetocide of the diseased twin. The live twin was delivered spontaneously without complications after thirty-eight weeks of pregnancy. The obstetric and legal ethical problems are discussed.

Anencephaly↗
PubMed1988

[Capillary hemangioma of the umbilical cord as a cause of polyhydramnios].

The authors report on an encapsulated, fibrous capillary hemangioma, 4 cm in diameter, located at the insertion of the umbilical cord, which caused a polyhydramnion in the 26th week of gestation. The diagnosis of capillary hemangioma was confirmed by multiple sonographic examinations. In the 27th week of gestation, 700 ml of amniotic fluid were drained by a single transabdominal amniocentesis. The child had to be delivered by cesarean section in the 36th week of gestation owing to a breech presentation in a primigravida mother and signs of respiratory placental insufficiency. No malformations of any kind were found in the newborn. The importance of the capillary hemangioma as the cause of a polyhydramnion is discussed with regard to differential diagnosis.

Adult↗
PubMed1997

Polyhydramnios.

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Amniocentesis↗