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At least 199 records · Page 11Linked to original sources
PubMed2000

Sex pheromone mimicry in the early spider orchid (ophrys sphegodes): patterns of hydrocarbons as the key mechanism for pollination by sexual deception.

We investigated the female-produced sex pheromone of the solitary bee Andrena nigroaenea and compared it with floral scent of the sexually deceptive orchid Ophrys sphegodes which is pollinated by Andrena nigroaenea males. We identified physiologically and behaviorally active compounds by gas chromatography with electroantennographic detection, gas chromatography-mass spectrometry, and behavioral tests in the field. Dummies scented with cuticle extracts of virgin females or of O. sphegodes labellum extracts elicited significantly more male reactions than odorless dummies. Therefore, copulation behavior eliciting semiochemicals are located on the surface of the females' cuticle and the surface of the flowers. Within bee and orchid samples, n-alkanes and n-alkenes, aldehydes, esters, all-trans-farnesol and all-trans-farnesyl hexanoate triggered electroantennographic responses in male antennae. Most of the alkanes and alkenes occurred in similar patterns both in the bees and orchids. O. sphegodes leaf extracts contained mostly the same compounds but in different proportions. In behavioral tests with synthetic compounds, blends of alkenes triggered significantly more approaches and pounces of the males whereas alkanes were not more attractive than odorless dummies. Since alkanes and alkenes together were most attractive, we conclude they constitute the bees' sex pheromone as well as the pseudocopulation-behavior releasing orchid-odor bouquet.

Adaptation, Biological↗
PubMedDate not supplied

Munchausen by proxy syndrome: the triad of abuse, self-abuse, and deception.

The Munchausen By Proxy Syndrome (MbPS) is a complicated phenomenon whereby a peculiar threefold relationship between perpetrator, victim, and medical staff develops. This triad is based on abuse of the child, deception of the doctor, and, in many cases, self-abuse or passivity of the victim towards the perpetrator's deeds. We have analyzed the interrelationship between the perpetrator, the victim, and the medical authority, in light of the mother's pathology.

Child↗
PubMed1994

Deceptive imprinting in the immune response against HIV-1.

The clonal profile of anti-HIV-1 antibodies is established at the time of infection as part of a vigorous immune response against HIV-1, and remains stable during the infection process. This bias towards antibodies specific for the initially infecting clonal virus population, termed imprinting, is inappropriate for attempts of the infected host to control viral variants that subsequently emerge. Here, Heinz Köhler, Sybille Müller and Peter Nara argue that immunodominant epitopes on viral variants or recombinant proteins are selected that induce and maintain this deceptive state, and thereby remain unrecognized through a functional and cross-reactive hole in the B-cell repertoire.

Animals↗
PubMed1995

The biology of deception: the reluctance to accept the cognitive animal.

Cognitive ability did not appear de novo in man. Despite our ability to recognize limited cognitive behavioral characteristics in animals, there has been no outcry to proclaim this phenomenon. Man's image of himself solely possessing cognition has taken advantage of the illusory potential in intersubjectivity and placed him outside of reality. This deception, however, has positive survival value since it is man's self-proclaimed responsibility to excel beyond other simple animal species. However, at this point in evolution, we must allow our cognitive ability to reform itself and, in so doing, evolve with the benefit of the knowledge that this ability is itself creating. By recognizing that animals may have limited cognitive ability, we only enhance our self-esteem, not diminish it.

Animals↗
PubMed2005

Deception and the origin of honest signals.

Deceptive signals are a challenge to explain because on average, signals should be reliable. When being deceived is costly to the receiver, a coevolutionary struggle between senders and receivers can ensue. Recent work by Macías Garcia and Ramirez raises the intriguing possibility that through such a coevolutionary process, cheats can become honest.

Journal Article↗
PubMed2001

Eye blinks: new indices for the detection of deception.

Eyeblink variables were investigated while subjects performed a guilty knowledge test (Experiment 1) and a dual modality attention task (Experiment 2). In both experiments, the temporal distribution of blinks was analyzed using an automatic video based blink analysis system [Matsuo and Fukuda, Jpn. J. Physiol. Psychol. Psychophysiol., 14 (1996), 17]. In experiment 1, the blink rate pattern discriminated between relevant and irrelevant stimuli. In experiment 2, the blink rate peak after the auditory stimulus disappeared during visually attended tasks whereas the blink rate peak after the visual stimulus was significant during auditory attended tasks. It was suggested that eye blinks could be related to the selective attention and that eye blinks could provide an additional index for the detection of deception.

Acoustic Stimulation↗
PubMed1998

P300 correlates of simulated malingered amnesia in a matching-to-sample task: topographic analyses of deception versus truthtelling responses.

Two experiments using a P300-enhanced Forced Choice Procedure (P3FCP) investigated simulated amnesia in a matching-to-sample task. In Experiment 1, successful manipulation of subjects towards different behavioral hit rates (75-80% vs. 85-90%) did not adversely affect the diagnostic sensitivity of match-mismatch Pz-P300 amplitude analyses, allowing detection of 69% of simulators. P300 amplitudes of simulators (Malinger group) were as large as those of truth-tellers (truth group, a control), indicating no dual task-related (Malingering) reduction across different behavioral hit rates. Experiment 2 found no main effect of oddball type, match vs. mismatch, on P300 (P3) amplitude with a mismatch-rare variant of the P3FCP. This study also revealed larger Pz-P3s in the Malingering (vs. Truth-telling) condition. Subsequent topographic analyses suggested different Truth and Malinger scaled P3 amplitude topographies in both these sets of P3FCP data and in those from a previous autobiographical memory paradigm. Further analysis yielded preliminary evidence for a common deception-related P3 amplitude topography across different paradigms/conditions.

Adult↗
PubMed1986

Deceptive patterns of uremic pulmonary edema.

Three uremic patients had chest x-ray changes due to pulmonary edema, but mimicking lobar pneumonia and fungal or metastatic disease. It is important to be aware of these deceptive radiographic patterns. Otherwise, a potentially disastrous delay in fluid removal, the mainstay of therapy, may result.

Adult↗
PubMed1996

The evolutionary biology of self-deception, laughter, dreaming and depression: some clues from anosognosia.

Patients with right hemisphere strokes sometimes vehemently deny their paralysis. I describe three new experiments that were designed to determine the extent and depth of this denial. Curiously, when asked to perform an action with their paralyzed arm, they often employ a whole arsenal of grossly exaggerated 'Freudian defense mechanisms' to account for their failure (e.g. 'I have arthritis' or 'I don't feel like moving it right now'). To explain this, I propose that, in normal individuals, the left hemisphere ordinarily deals with small, local 'anomalies' or discrepancies by trying to impose consistency in order to preserve the status quo. But when the anomaly exceeds threshold, a 'devil's advocate' in the right hemisphere intervenes and generates a paradigm shift, i.e. it results in the construction of a new model using the same data. A failure of this process in right hemisphere stroke would partially explain anosognosia. Also, our model provides a new theory for the evolutionary origin of self-deception that is different from one proposed by Trivers. And, finally, I use anosognosia as a launching-off point to speculate on a number of other aspects of human nature such as Freudian defense mechanisms, laughter, dreams and the mnemonic functions of the hippocampus.

Aged↗
PubMed1997

Early infantile Krabbe disease: deceptively normal magnetic resonance imaging and serial neurophysiological studies.

Early infantile Krabbe disease is a progressive neurodegenerative disease caused by deficiency of lysosomal enzyme galactocerebroside beta-galactosidase, with onset before the age of 6 months. We present serial clinical, radiological and neurophysiological findings of a patient with early infantile Krabbe disease, presenting at the third day of life with hypotonia, macrocephaly and neonatal seizures. The patient had a deceptively normal initial magnetic resonance imaging examination at the age of 3 months, with progression of the white matter disease over the following 9 months, showing a clinical picture of profound hypotonia with pyramidal and pseudobulbar signs, as well as mild optic atrophy. Assay of galactocerebroside beta-galactosidase activity in leukocyte culture disclosed a marked deficiency of the enzyme (0.00 nmol/mg protein per h with normal values > 0.7 nmol/mg protein per h), thus confirming the diagnosis of Krabbe disease. Nerve conduction velocity and evoked potential studies, as well as the electroencephalogram, were abnormal at the age of 6 months, while serial neurophysiological studies at the age of 12 and 18 months demonstrated the progressive nature of the disease.

Aging↗
PubMed1995

How shall a deceptive thing be called?

Thirty-six three-, four- and five-year-olds were asked to select labels for deceptive stimuli (e.g. for an eraser that looked like a pencil). Three types of labelling were investigated--simple (e.g. 'is an eraser'); appearance-predicated (e.g. 'looks like an eraser'); and reality-predicated (e.g. 'is really and truly an eraser'). An age-related appearance--reality shift was observed in simple labelling (e.g. older children were more likely than younger ones to accept eraser and reject pencil as simple names for the pencil-eraser). This trend was robust over method and semantic domain, though weaker with object than with colour labels. As in previous research, older children were more likely than younger ones to map different appearance- than reality-predicated labels onto an item (e.g. to accept that the pencil-eraser looks like a pencil, but is really and truly an eraser); however, all age groups were reluctant to extend more than one name to a stimulus via a common predicate (e.g. to accept two reality-predicated labels for the same object). This one-label-perpredicate pattern was observed more frequently within reality than within appearance predicates; more frequently with colour than with object names, and with questions blocked by predicate than by name. It is argued that younger children maintained this pattern because of inflexible encoding, but that older ones did so because of better understanding of the appearance-reality distinction, greater reality dominance, and a Mutual Exclusivity bias.

Child↗
PubMed2004

Hastening death by selective disclosure of treatment options--beneficence or "euthanasia by deception"?

In this paper I make a radical claim regarding selective non-disclosure of treatment options that have some hope of prolonging a patient's life. I suggest that selective non-disclosure under such circumstances is tantamount to what might be called "euthanasia by deception." I offer a case to test the validity of my claim and to demonstrate how the failure to offer or, at least, to discuss renal dialysis in this case (and, by inference, any other form of treatment which has some hope of prolonging a patient's life) qualifies as paternalism in its most egregious form. I discuss the actions of the health care team and try to find some plausible reasons why they acted as they did. I conclude that there must be greater emphasis placed on teaching clinicians how better to incorporate frank, open and on-going discussion about the central elements of the therapeutic relationship with patients long before they lose decisional capacity.

Aged↗
PubMed1983

Inadequacy of voice recognition as a demonstration of self-deception.

Gur and Sackeim (1979) argued that subjects deceived themselves when they failed to recognize their own voices on playback from a tape recorder. This claim is based primarily on the observation that subjects showed a heightened galvanic skin response when their own voices were present regardless of whether recognition took place. The authors suggest that even though subjects may not consciously recognize their own voices, a heightened physiological response implies that true recognition did in fact occur at some other level of cognitive processing. This article describes an experiment demonstrating that results similar to those arrived at by Gur and Sackeim can also be produced when subjects attempt to recognize the voice of a familiar "other." These results suggest that self-deception is not the main factor operating to produce the heightened physiological response.

Female↗
PubMed2004

Are eyes windows to a deceiver's soul? Children's use of another's eye gaze cues in a deceptive situation.

Three experiments examined 3- to 5-year-olds' use of eye gaze cues to infer truth in a deceptive situation. Children watched a video of an actor who hid a toy in 1 of 3 cups. In Experiments 1 and 2, the actor claimed ignorance about the toy's location but looked toward 1 of the cups, without (Experiment 1) and with (Experiment 2) head movement. In Experiment 3, the actor provided contradictory verbal and eye gaze clues about the location of the toy. Four- and 5-year-olds correctly used the actor's gaze cues to locate the toy, whereas 3-year-olds failed to do so. Results suggest that by 4 years of age, children begin to understand that eye gaze cues displayed by a deceiver can be informative about the true state of affairs.

Child, Preschool↗
PubMed2002

Does the conscious exaggeration scale detect deception within patients with chronic pain alleged to have secondary gain?

OBJECTIVES: The illness behavior questionnaire (IBQ) is a test battery developed by Pilowsky to detect what he has termed abnormal illness behavior, which includes malingering. The IBQ has been widely utilized in patients with chronic pain (PWCP). Clayer developed a 21-item scale out of the IBQ, which he termed the conscious exaggeration (CE) scale. He proposed that the CE scale could detect conscious deception, i.e., malingering. The purpose of the present study is to test the CE scale in PWCP alleged to have secondary gain and thereby at greater risk for poor pain treatment outcome. It was postulated that the CE scale should generate scores in these groups significantly different from a comparison group and should predict treatment outcome in the secondary gain groups. DESIGN: A total of 96 PWCP completed the CE scale at admission and after completion of a 1-month pain facility treatment regimen. Other relevant pain variables, such as pain, depression, and anxiety, were measured and the data collected at admission and treatment completion. Work status was determined at 1, 3, 6, 12, 18, 24, and 30 months posttreatment. PWCP secondary gain subgroups (Workers' Compensation patients, patients in litigation, patients having a lawyer) were compared to the comparison group (no secondary gain factors) for treatment CE scale change scores. In order to control for the effects of pain, an analysis of covariance with pain level statistically removed was performed on admission and discharge CE scores utilizing the above patient subgroups. Pearson product correlations were utilized to determine the relationships between CE scores and psychological variables. Stepwise regression analyses were utilized to predict return to work with the CE scale score as a potential predictor. SETTING: Pain facility. PATIENTS: PWCP treated for 1 month in a pain facility. RESULTS: Overall, the analyses did not support the main hypothesis. For example, CE scale scores did not predict return to work. There was a significant degree of correlation between the variables of pain, depression, anxiety, and CE scale scores. CONCLUSIONS: PWCP characterized by the alleged secondary gain variables of Workers' compensation status, litigation, and having a lawyer did not differentially respond to the CE scale versus the comparison group. The CE scale, therefore, does not appear to be a valid instrument for identifying exaggeration in PWCP.

Journal Article↗
PubMed2003

Children's use of gaze and limb movement cues to infer deception.

A sample of 96 children from kindergarten, 2nd, 4th, and 6th grades judged the truthfulness of peers who varied in gaze and limb movement while providing verbal communications. Results indicated that children attributed greater lying to the peers who displayed indirect rather than direct gaze and active rather than nonactive limb movement. The use of these cues was more evident in 4th- and 6th-grade children than it was in kindergarten and 2nd-grade children. Pilot studies indicated that adults and children as young as 5-6 years of age associated indirect gaze and active limb movement with anxiety. The findings are discussed with respect to children's theory of mind, concepts of lying, understanding of display rules, and learning of physiological cues associated with deception.

Anxiety↗
PubMed1993

Detection of deception: a transactional analysis perspective.

I have attempted to integrate two divergent fields in applied psychology, the psychophysiological detection of deception (polygraph testing) and the transactional analysis theory. Transactional analysis theory provides a useful framework for understanding the examiner-examinee interaction in the pretest interview of the widely used but highly controversial control-question polygraph test. This research is relevant to the ongoing debate about the plausibility of the rationale underlying this technique.

Arousal↗
PubMedDate not supplied

Avoiding deceptive imprinting of the immune response to HIV-1 infection in vaccine development.

Lymphocyte clonal restriction is caused by priming the immune system with an antigen and has been referred to infectious disease study as "original antigenic sin" (OAS), described first for influenza by Francis. OAS is a dominant feature of a normal immune response. Benefits of OAS come from the initial contact with the pathogen, which induces immunological memory. Memory is achieved by priming B and T cells of an immunologically naïve host, and confers protection against infection with the antigen-related pathogen. Thus, a restricted antibody response to viral or parasite antigens is not per se pathogenic. However, the interplay between a "locked-in" immune response and the high genetic variation of the pathogenic agent can result in a deception of the immune system. In the following, clonal restriction of the immune response to HIV is described by giving examples of restricted anti-HIV antibody formation in maternally infected children. Clonal restriction results in host resistance of infected individuals to emerging HIV variants and quasispecies. The problems of classical approaches of vaccine design in AIDS and the lack of protection in vaccinated patients is reviewed.

AIDS Vaccines↗