Potentiation of effects of L-dopa on conditioned avoidance behavior by inhibition of extracerebral dopa decarboxylase.
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Animals↗
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Amantadine versus L-2 dopa and amatadine plus L-dopa.
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Amantadine↗
Determination of DOPA, dopamine, and 5-S-cysteinyl-DOPA in plasma, urine, and tissue samples by high-performance liquid chromatography with electrochemical detection.
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Animals↗
Optical resolution of N-acyl-DL-amino acids by preferential crystallization procedure. Preparation of L-DOPA and L-alpha-methyl DOPA.
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Crystallization↗
Separation of aromatic acids, DOPA, and methyl-DOPA by capillary electrophoresis with dendrimers as buffer additives.
Polyamidoamine starburst dendrimers with terminal carboxylate groups are used as a pseudo-stationary phase in electrokinetic chromatography for separating the selected aromatic amino acids phenylalanine, phenylglycine, homophenylalanine, and tyrosine and the catecholamines 3-(3,4-dihydroxyphenyl)alanine and 3-(3,4-dihydroxyphenyl)-2-methylalanine at different pH levels. A significant difference in analyte selectivity is observed between dendrimers of different generations and at different pH levels. At pH 7.0, the utility of the dendrimers for separating these analytes is limited by relatively low selectivity and a noisy baseline. However, good separation and selectivity are obtained with low generation dendrimers (G0.5 and G1.5) at low pH. Strong association of the solute with dendrimers is observed for high generations (G2.5, G3.5, and G5.5).
Biocompatible Materials↗
Inability of murine melanoma "tyrosinase" (dopa ozidase) to oxidize tyrosine in the presence or absence of dopa or dihydroxyfumarate cofactor.
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Animals↗
Changes in the formation of 3 H-catecholamines from 3 H-DOPA and 3 H-tyrosine induced by unlabelled DOPA.
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Brain Chemistry↗
Studies on the metabolism of D- and L-isomers of 3,4-dihydroxyphenylalanine (DOPA). I. Autoradiographic study on the distribution of 14 C-labeled D- and L-DOPA and dopamine after intravenous administration in rats.
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Adipose Tissue, Brown↗
The plasma levels of dopa and catecholamines after oral administration of L-dopa.
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Administration, Oral↗
Studies on the metabolism of D- and L-isomers of 3,4-dihydroxyphenylalanine (DOPA). II. Autoradiographic study on the distribution of 14 C-labeled D- and L-DOPA after oral administration in rats.
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Animals↗
Studies on the metabolism of D- and L-isomers of 3,4-dihydroxyphenylalanine (DOPA). V. Mechanism of intestinal absorption of D- and L-DOPA-14C in rats.
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Animals↗
Studies on the metabolism of D- and L-isomers of 3,4-dihydroxyphenylalanine (DOPA). 3. Absorption, distribution and excretion of D- and L-DOPA-14C in rats following intravenous and oral administration.
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Administration, Oral↗
Studies on the metabolism of D- and L-isomers of 3,4-dihydroxyphenylalanine (DOPA). IV. Urinary and tissue metabolites of D- and L-DOPA-14C after intravenous and oral administration to rats.
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Administration, Oral↗
Studies on the metabolism of D- and L-isomers of 3,4-dihydroxyphenylalanine (DOPA). VI. Metabolism of D-DOPA in rat kidney.
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Animals↗
High-performance liquid chromatographic determination of L-3,4-dihydroxy-phenylalanine (L-DOPA) and its metabolites in the urine of patients with Parkinson's disease, control patients and normal subjects after oral administration of L-DOPA.
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Adult↗
[Hypertension: 3 H-catecholamine metabolism in the adrenal glands and brain and tissue metabolism of 3 3 H-DOPA during reserpine, alpha-methyl DOPA and ST-155 administration in spontaneously hypertensive rats].
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Adrenal Glands↗
Stimulatory effect of the dopa-decarboxylase inhibitor Ro 4-4602 on prolactin release; inhibition by l-dopa, metergoline, methysergide and 2-Br-alpha-ergocryptine.
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Administration, Oral↗
[Clinical significance of long-term treatment of L-dopa with bromocriptine in parkinsonian patients who showed declining efficacy during L-dopa therapy].
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Adult↗