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PubMed · 7500784

Decrease in glutamate in the jugular vein after head injury.

A Kudo·M Suzuki·A Kitakami·K Kuroda·A Ogawa
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Craniocerebral TraumaGlutamic AcidHumansJugular VeinsLetter

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A Kudo, M Suzuki, A Kitakami, K Kuroda, A Ogawa. 1995-12-16. Decrease in glutamate in the jugular vein after head injury.. https://doi.org/10.1016/s0140-6736(95)91974-0

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PubMed1995

Excessive hypokalemia and hyperkalemia following head injury.

A sudden decrease of serum potassium below 2.5 mmol/l carries the risk of dangerous arrhythmias and requires immediate replacement therapy [6]. We refer to a patient with a brain stem compression after head injury, who developed a profound hypokalemia (K+ = 1.2 mmol/l) with life-threatening arrhythmias, probably due to a catecholamine induced intracellular potassium shift (beta-2-stimulation). Only by aggressive potassium replacement up to 80 mmol/h (610 mmol/16 h) could potassium levels be increased and cardiac arrhythmias terminated. Although replacement therapy was stopped when the serum K(+)-level increased to 2.4 mmol/l, 3.5 h later the patient became hyperkalemic (8.1 mmol/l). This was probably due to a secondary shift of potassium from intra- to extracellular space. In patients with severe head trauma and the potential risk of excessive catecholamine release special attention must be paid to changes in potassium balance.

Craniocerebral Trauma↗