PubMed · 612821
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I Fukuda, T Yoshida, T Mozai. 1977. [Ramsay Hunt syndrome].. https://pubmed.ncbi.nlm.nih.gov/612821/
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In order to ascertain what can develop from cerebral infantile hypotonia, 98 infants suffering from hypotonic forms of cerebral palsy were reexamined after one year and some of them again after another year. It was presumed that hypotonia in infants with so-called hypotonic cerebral palsy is a sign which changes with time. In the ages between 2-4 years 50.2% of the hypotonias decreased and new syndromes appeared. In the ages up to 4-6 years, there was a decrease in 85.7% of hypotonias. The new syndromes were cerebellar in 56 cases, mental defectivity with developmental disintegration in 31 cases, and there were 26 without any cerebral symptomatology. Apart from these two main semeiological groups, there were spastic and dyskinetie syndromes, mostly combined with the cerebellar or mental group, and minor brain, mostly cerebellar, disturbances which developed out of infantile central hypotonias.
Arterial hypertension causes the formation of small lacunes, or ischemic brain infarcts, which may result in transient ischemic attacks, hemiparesis, sensory loss, ataxic hemiparesis, or dysarthria. Usually these effects are reversible with physical therapy, but multiple lesions may lead to dementia. Prevention of lacunar disease is possible with vigorous control of hypertension.