Search PubMed⌕ Search

PubMed · 3283611

Static exercise--physiologic dangers and proper training techniques.

Abstract

Static exercise is commonly encountered occupationally and in normal activities of daily living. In addition, weight training, a form of static exercise, is one of the fastest growing physical activities in the United States. Static exercise is a stressor to the cardiovascular system. At high intensities it produces immediate and profound systolic and diastolic hypertension, moderate tachycardia and enhanced contractility resulting in a moderate increase in cardiac output without an increase in stroke volume. Static exercise is well tolerated in healthy people. People with cardiovascular disease appear to tolerate static exercise of low intensity if they have normal left ventricular fraction and reserve. However, in people with impaired left ventricular function and reserve, the exercise stress causes tachycardia, a hypertensive state, increased systemic vascular resistance, and left ventricular end diastolic pressure. If the exercise stress persists, stroke volume and ejection fraction decline. This article helps nurse practitioners in wellness and preventive patient education. An analysis of current research covers the relationship between client variables and the cardiovascular effects of static exercise. Nurse practitioners learn how to identify and advise clients who should avoid static exercise. The proper selection of a health fitness club and the proper components of a basic weight-training program are comprehensively reviewed.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

J L Stopford. 1988. Static exercise--physiologic dangers and proper training techniques.. https://pubmed.ncbi.nlm.nih.gov/3283611/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

Diffusion tensor magnetic resonance imaging mapping the fiber architecture remodeling in human myocardium after infarction: correlation with viability and wall motion.

BACKGROUND: Diffusion tensor magnetic resonance imaging (DT-MRI) provides a means for nondestructive characterization of myocardial architecture. We used DT-MRI to investigate changes in direction-dependent water diffusivity to reflect alterations in tissue integrity (trace apparent diffusion coefficients [ADCs] and fractional anisotropy [FA]), as well as indicators of remodeling of fiber helix angles, in patients after myocardial infarction. METHODS AND RESULTS: Thirty-seven patients (35 men, 2 women; median age, 59) after acute myocardial infarction (median interval from onset, 26 days) were enrolled. DT-MRI was performed at the midventricular level to measure trace ADC, FA, and helix angles of myofibers. Helix angles were grouped into left-handed helical fibers, circumferential fibers, and right-handed helical fibers. Measurements were correlated with viability and regional wall motion assessed by contrast-delay-enhancement and cine MRI, respectively. The infarct zone showed significantly increased trace ADC and decreased FA than the remote zone. The percentage of left-handed helical fibers increased from the remote zone (mean +/- SD, 13.3 +/- 5.8%) to the adjacent zone (19.2 +/- 9.7%) and infarct zone (25.8 +/- 18.4%) (MANOVA, P = 0.004). The percentage of right-handed helical fibers decreased from the remote zone (35.0 +/- 9.0%) to the adjacent zone (25.5 +/- 11.5%) and infarct zone (15.9 +/- 9.2%) (P < 0.001). Multiple linear regression showed that the percentage of left-handed helical fibers of the infarct zone was the strongest correlate of infarct size and predictor of ejection fraction. CONCLUSIONS: In vivo DT-MRI of postinfarct myocardium revealed a significant increase in trace ADC and a decrease in FA, indicating altered tissue integrity. The redistribution of fiber architecture correlated with infarct size and left ventricular function. This technique may help us understand structural correlates of functional remodeling after infarction.

Cardiovascular Physiological Phenomena↗

Development of a mathematical model of the human circulatory system.

A mathematical lumped parameter model of the human circulatory system (HCS) has been developed to complement in vitro testing of ventricular assist devices. Components included in this model represent the major parts of the systemic HCS loop, with all component parameters based on physiological data available in the literature. Two model configurations are presented in this paper, the first featuring elements with purely linear constitutive relations, and the second featuring nonlinear constitutive relations for the larger vessels. Three different aortic compliance functions are presented, and a pressure-dependent venous flow resistance is used to simulate venous collapse. The mathematical model produces reasonable systemic pressure and flow behaviour, and graphs of this data are included.

Cardiovascular Physiological Phenomena↗