Search PubMed⌕ Search

PubMed · 2646608

Identifying the alcoholic client.

Abstract

Although there is no single sign or symptom that provides conclusive evidence of alcoholism, a thorough and thoughtful assessment can provide clues for early identification of the alcoholic person. The nurse's approach should include an accepting, matter-of-fact, nonthreatening attitude. Family members should be included in the assessment if possible. The assessment should include information about the client's drinking patterns, as well as about physiological, psychological, and behavioral signs and symptoms indicative of alcoholism. Astute observations and questioning about this combination of factors can result in early identification and intervention with alcoholic clients and their families.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

S H Tweed. 1989. Identifying the alcoholic client.. https://pubmed.ncbi.nlm.nih.gov/2646608/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

How does alcohol impair neuronal migration?

Maternal alcohol consumption during pregnancy can cause serious birth defects, of which fetal alcohol syndrome (FAS) is the most devastating. Recognized by characteristic craniofacial abnormalities and growth deficiency, this condition produces severe alcohol-induced damage in the developing brain. FAS children experience ataxia; deficits in intellectual functioning; and difficulties in learning, memory, problem solving, and attention. Multiple aspects of central nervous system development can be affected by alcohol exposure, but the most striking abnormalities are neuronal and glial migration. Little is known about cellular mechanisms by which alcohol affects the migration of immature neurons. Recently, it has been found that Ca(2+) signaling and cyclic nucleotide signaling are the central targets of the action of alcohol in neuronal cell migration. Most importantly, the aberrant migration of immature neurons caused by alcohol exposure is significantly ameliorated by controlling the activity of these second-messenger pathways. In this Mini-Review, we first describe how alcohol exposure impairs the migration of cerebellar granule cells and then discuss the signaling mechanisms involved.

Alcoholism↗

[Carbamazepine intoxication. Complication of alcohol detoxification with combined carbamazepine and tiapride].

The advantages of alcohol detoxification treatment with combined carbamazepine and tiapride compared to benzodiazepines or clomethiazole is a lower level of sedation and lack of addictive potential. We report a case of carbamazepine intoxication with serum levels up to 19 mg/l in an otherwise healthy 45-year-old alcohol-dependent male after treatment with 600 mg carbamazepine and 600 mg tiapride per day. Medication was discontinued immediately and a purgative was administered. We were able to combat the intoxication but the assumed good tolerance of the combined treatment with carbamazepine and tiapride for alcohol detoxification still has to be proven.

Alcoholism↗