Epithelial-mesenchymal interactions: mesenchymal specificity.

H C Slavkin; G N Trump; A Brownell; N Sorgente

Home/Archive/PubMed

PubMed · 198906

Epithelial-mesenchymal interactions: mesenchymal specificity.

H C Slavkin·G N Trump·A Brownell·N Sorgente

View original source ↗Export metadata ↓

Abstract

The source did not provide an abstract. Follow the original record for more information.

Explore related subjects

Ameloblasts Amelogenesis Animals Cell Communication Cell Differentiation Collagen Dental Enamel Proteins Dentinogenesis Epithelium Intercellular Junctions Mice Microbial Collagenase Odontoblasts Odontogenesis Rabbits Journal Article Research Support, U.S. Gov't, P.H.S.

Keep this discovery

Explore connections, maps & timelines

BibTeX RIS

H C Slavkin, G N Trump, A Brownell, N Sorgente. 1977. Epithelial-mesenchymal interactions: mesenchymal specificity.. https://pubmed.ncbi.nlm.nih.gov/198906/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed1979

Mineralization of the enamel of ovine permanent central incisor teeth using microhardness and histological techniques.

Explore the source record for details and available documents.

Ameloblasts↗

PubMed1979

[Neurohistologic studies on retained human teeth including the follicular cyst IV].

Explore the source record for details and available documents.

Ameloblasts↗

PubMed1979

Physiological cell death of secretory ameloblasts in the rat incisor.

The migration of the ameloblasts in the continuously erupting incisors of the rat is accompanied by cell loss. Ameloblasts degenerate near the mesial and lateral cemento-enamel junctions in the secretory zone and in the middle two thirds of the region of postsecretory transition, degeneration being most marked where these areas merge. These findings support the hypothesis that the prism decussation in the enamel results from alternating transverse rows of secretory ameloblasts sliding past each other whilst elaborating their rods. The distribution of the degenerating cells suggests, however, that the sliding cell rows are not exactly transverse but arcuate, with the opening facing incisally. The progress of structural alterations of the nuclei in the degenerating ameloblasts appears to follow the pattern earlier described in vinblastine-damaged ameloblasts.

Ameloblasts↗