PubMed · 13830982
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J de KLERK. 1960-11-12. [Bladder neck stenosis].. https://pubmed.ncbi.nlm.nih.gov/13830982/
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Pulmonary hypertension associated with increased pulmonary vascular resistance occurring in the setting of portal hypertension, referred to as "portopulmonary hypertension", is a complication of chronic liver disease, and occurs in 2% to 3% of patients with portal hypertension. Portal hypertension is a relatively common finding in patients with chronic myeloproliferative disorder (CMPD). Pulmonary hypertension is also an occasional finding in CMPD patients. Latent myeloproliferative disorder, on the other hand does not fulfill the diagnostic criteria of classical CMPD and is characterized by younger age of onset, slow disease progression, a high risk of thrombosis, platelet dysfunction, and normal or increased platelet count in spite of the presence of splenomegaly. We report findings in a 50-year-old woman with portal hypertension for which there were three major etiological findings-increased splenic blood flow, infiltration of hematopoietic cells in the liver, and thrombosis in the portal or hepatic vein-over a 25-year clinical course, during which there was also reversible stenosis of the portal vein. Twenty-three years after her first admission, her condition was diagnosed as latent myeloproliferative disorder, and she developed pulmonary hypertension. Her clinical history and data indicated that the portopulmonary hypertension was due to the latent myeloproliferative disorder.
Penile implant surgery has a high rate of success and a very high patient satisfaction rate. Compression of the external iliac vein by the reservoir of the penile implant is an extremely rare occurrence. We describe a case of deep vein thrombosis due to compression of the external iliac vein, which required replacement of the penile implant reservoir.
Congenital nasal pyriform aperture stenosis is a rare and an unusual cause of airway obstruction in infants. It has been described as a clinical entity only since 1989. Presenting symptoms in infants include cyclical cyanosis, feeding difficulty or sudden total obstruction. Embryologically, an overgrowth of maxillary ossification at the area of the nasal process of the maxilla may be responsible for this deformity. Definitive surgical management may be accomplished via a sublabial approach with magnification, drilling and post-operative stenting of the airway. Two new cases are presented. The diagnosis, embryology and technique of surgical correction are discussed.