PubMed · 1255207
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D White. 1976. Letter: Automatic midline echoencephalography.. https://doi.org/10.1136/jnnp.39.1.103
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The incidence of periventricular leukomalacia (PVL) in risk neonates was evaluated among 300 risk neonates in a prospective ultrasound study at the Pediatric Clinic of the Medical Faculty, University of Rostock. Perinatal risk factors for the development of PVL and gestational age were taken into consideration. Maternal infections during pregnancy were found to be a risk factor for development of PVL. A severe hyaline membrane disease was more frequently diagnosed in highly immature preterm neonates with postnatal PVL. The percentage of highly immature preterm neonates with postnatal PVL and severe hyaline membrane disease was higher in vaginal-born risk neonates. A correlation between 1-minute Apgar scores < or = 3 and postnatal PVL was found in more mature preterm neonates.
We report the outcome of 12 very low birth weight infants with macrocrania caused by subarachnoid fluid collections. By the age of 15 to 18 months, head growth had stabilized along a curve above and parallel to the 95th percentile. No infant required neurosurgical intervention, nor was cerebral palsy or mental retardation diagnosed in any of the infants.
Periventricular leukomalacia (PVL) is an important pathogenetic factor in cerebral palsy in premature infants. We studied 708 premature infants with gestational ages < 36 weeks, who had been admitted to the NICU of Matsudo City Hospital, examined by cranial ultrasonographic method repeatedly, and followed up for more than 1 year with cranial ultrasonography, magnetic resonance imaging (MRI), and CT scanning. We made a diagnostic protocol for PVL. All cystic lesions and periventricular echo densities (PVE) 3, and 14.9% of PVE 2 on ultrasonography were clinically diagnosed in PVL. Cranial MRI were consistent with clinical symptoms, and its complicated findings corresponded to the severity of PVL. This protocol was useful for the diagnosis of clinical PVL.