Search PubMed⌕ Search

PubMed · 11084108

Pacemaker automaticity.

Abstract

The automated measurement of the main electrical parameters of pacemakers, such as battery voltage, current drain, pacing impedance, sensing levels, and pacing thresholds enables a continuous monitoring of the adequate functioning of the implanted device. New technologies in device interrogation, data transfer, and patient alert functions will enhance therapy safety significantly by the immediate detection of all, including transient, device malfunctions. The introduction of this technology will result in major changes in follow-up of patients with pacemakers. Technically we are very close to the goal of a fully automated pacemaker, although clinical acceptance lags behind. Clinical studies are necessary to demonstrate the beneficial effects of automated device functions, with regard to the main rationales for pacemaker automatization: increased patient safety, improved quality of life, and an improvement in the cost-benefit ratio in pacemaker therapy.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

J Neuzner, T Schwarz, J Sperzel. 2000-11-02. Pacemaker automaticity.. https://doi.org/10.1016/s0002-9149(00)01191-7

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

Sudden cardiac death, genes, and arrhythmogenesis: consideration of new population and mechanistic approaches from a National Heart, Lung, and Blood Institute workshop, Part II.

This is Part II of a 2-part article dealing with malignant ventricular arrhythmias, which are the leading mechanism of death in common cardiac diseases. Genetic population studies directed at discovering common proximal sources of inherited molecular risk most directly linked to arrhythmia initiation and propagation would appear to have considerable potential in helping reduce cardiovascular mortality.

Arrhythmias, Cardiac↗

Sudden cardiac death, genes, and arrhythmogenesis : consideration of new population and mechanistic approaches from a national heart, lung, and blood institute workshop, part I.

Malignant ventricular arrhythmias are the leading mechanism of death in patients with acute and chronic cardiac pathologies. The extent to which inherited mutations and polymorphic variation in genes determining arrhythmogenic mechanisms affect these patients remains unknown, but based on recent population studies, this risk appears significant, deserving much greater investigation. This report summarizes a National Heart, Lung, and Blood Institute workshop that considered sources of genetic variation that may contribute to sudden cardiac death in common cardiac diseases. Evidence on arrhythmogenic mechanisms in recent population studies suggests a significant portion of the risk of sudden cardiac death in such broad populations may be unrelated to traditional risk factors for predisposing conditions such as atherosclerosis, hypertension, and diabetes and instead may involve unrecognized genetic and environmental interactions that influence arrhythmic susceptibility more directly. Additional population and genetic studies directed at discovering the sources of inherited molecular risk that are most directly linked to arrhythmia initiation and propagation, in addition to studies on previously well-described risk factors, would appear to have considerable potential for reducing premature cardiovascular mortality.

Arrhythmias, Cardiac↗