Search PubMed⌕ Search
Home/Archive/PubMed

PubMed · 10369091

Content-specific confabulation.

P W Burgess·J E McNeil
View original source ↗Export metadata ↓

Abstract

This report describes a person who confabulated following an anterior communicating artery aneurysm. His confabulation was limited to one very circumscribed area of his life and remained stable for twelve weeks, eventually improving with rehabilitation. It is argued that a content-specific confabulation of this nature challenges current theories of confabulation, and an integrative explanation is given based on previous findings by Burgess and Shallice (1996b) of the mechanisms of autobiographical recollection in healthy people.

Explore related subjects

Cognition DisordersHumansIntelligence TestsIntracranial AneurysmMaleMemoryMemory DisordersMental ProcessesMiddle AgedNeuropsychological TestsCase ReportsJournal ArticleResearch Support, Non-U.S. Gov't

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

P W Burgess, J E McNeil. 1999. Content-specific confabulation.. https://doi.org/10.1016/s0010-9452(08)70792-5

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed2000

[New insights in frontotemporal dementia].

Frontotemporal dementia (FTD) is a neurodegenerative disorder characterised by progressive behavioural disturbance, aphasia and a decline in frontal cognitive functions. Frontotemporal atrophy on CT and MRI, and hypoperfusion of the frontal brain regions on single-photon emission computed tomography (SPECT), are characteristic findings. Neuropathological examination reveals deposition of abnormally phosphorylated tau protein in neurons and glial cells in a number of the sporadic and familial cases, while aspecific changes with neuronal loss, spongiosis and gliosis are found in the remaining cases. A familial form with an autosomal dominant pattern of inheritance is seen in 20% of FTD patients. Mutations in the tau gene have been identified in a number of families with deposition of abnormal tau protein in affected brain regions. Presymptomatic DNA testing is now available for relatives of patients with tau mutations, but must only be considered after extensive genetic counselling in a centre with neurogenetic expertise.

Cognition Disorders↗
PubMed2000

Intensive care unit syndrome: a dangerous misnomer.

The terms intensive care unit (ICU) syndrome and ICU psychosis have been used interchangeably to describe a cluster of psychiatric symptoms that are unique to the ICU environment. It is often postulated that aspects of the ICU, such as sleep deprivation and sensory overload or monotony, are causes of the syndrome. This article reviews the empirical support for these propositions. We conclude that ICU syndrome does not differ from delirium and that ICU syndrome is caused exclusively by organic stressors on the central nervous system. We argue further that the term ICU syndrome is dangerous because it impedes standardized communication and research and may reduce the vigilance necessary to promptly investigate and reverse the medical cause of the delirium. Directions for future research are suggested.

Cognition Disorders↗