Variable convergence adaptive filter and its application to cardiac action potentials.
Explore the source record for details and available documents.
Action Potentials↗
Biomedical subjects
Z Bin
Publications and source records attributed to Z Bin.
[Histological observation on gingival fibers of upper molar region].
The purpose of this study was to investigate the structures and functions of the gingival fibers extending in the mesiodistal direction (mesiodistal fibers) in the human gingiva of the upper molar region. They were observed by light and scanning electron microscopes. The findings are as follows: 1. There is a large number of mesiodistal fibers in the buccal gingiva of the upper molar region. 2. In addition to the factor of the space, there exists a close relation between the development of the mesiodistal fibers of the buccal gingiva and the function in its region. 3. The mesiodistal fibers are made up of primary and secondary fiber bundles. 4. The bundles of the mesiodistal fibers compose the three dimensional network and are twisted.
Gingiva↗
[Physiologic delayed puberty].
Explore the source record for details and available documents.
Adolescent↗
Inhibition by glaucocalyxin A of aggregation of rabbit platelets induced by ADP, arachidonic acid and platelet-activating factor, and inhibition of [3H]-PAF binding.
Glaucocalyxin A is a new diterpenoid isolated from the ethereal extract of the leaves of Rabdosia japonica (Burm f) Hara var glaucocalyx (Maxim) Hara (Labiatae) collected in the northeastern China. When it was incubated with washed rabbit platelets, glaucocalyxin A inhibited ADP- or arachidonic acid-induced platelet aggregation with IC50 values of 4.4 mumol/l, 14.1 mumol/l respectively. Glaucocalyxin A also inhibited PAF-induced aggregation of rabbit platelets which were refractory to ADP and arachidonic acid with an IC50 value of 13.7 mumol/l. Analysis of [3H]-PAF binding showed that glaucocalyxin A prevented [3H]-PAF binding to intact washed rabbit platelets with an IC50 value of 8.16 mumol/l, which was consistent with its inhibition of PAF-induced platelet aggregation.
Adenosine Diphosphate↗