Biomedical subjects

Yehoshua Almog

Publications and source records attributed to Yehoshua Almog.

4 recordsLinked to original sources

PubMed2004

Plotting visual fields using contralateral fixation: a novel honeycomb device.

A device that maintains steady fixation while plotting the visual field of an eye with a large central defect is presented. The device is composed of multiple adjacent pinhole tubes mounted on an eye shield and placed in front of the seeing fellow eye. The device constricts the visual field of the fixating nontested eye to preserve only the central 3 degrees, thus not interfering with the visual field of the tested eye. We tested 3 healthy volunteers with simulated visual field defects and more than 100 patients with unilateral loss of central vision. The use of this device allowed us to plot visual field defects with clear-cut borders while no meaningful results could be obtained without it. We found this device to be efficient, simple to use, and suitable for all varieties of perimetry.

Aged↗

PubMed2003

Visual outcome in eyes with asymptomatic optic disc edema.

BACKGROUND: Asymptomatic optic disc edema may last for months before conversion to anterior ischemic optic neuropathy (AION). Alternatively, the optic disc edema may resolve with preservation of normal vision. The conversion rate of asymptomatic optic disc edema to AION has not been prospectively studied. We prospectively followed patients with asymptomatic disc edema to determine this conversion rate. METHODS: The cohort was followed from 1991 to 2000 at a single ophthalmology clinic in Israel. There were 23 patients aged 47-74 years with asymptomatic disc edema and no signs of optic nerve dysfunction in whom the disc edema had been incidentally discovered on routine fundus examination performed for diabetes, hypertension, or follow-up after AION in the fellow eye. RESULTS: In 9 (36%) eyes, optic disc edema progressed to overt AION with a mean latency of 16.8 weeks (range 2-80 weeks). In 16 (64%) eyes, optic disc edema resolved without loss of vision with a mean latency of 15.5 weeks (range 4-44 weeks). The conversion rate to AION was 40% in patients who had had AION in the fellow eye, 31% in patients with diabetes, 43% in patients with diabetic retinopathy, and 0% in four amiodarone-treated patients. CONCLUSION: Asymptomatic disc edema generally resolves with no visual loss, but one third of patients progress to full-blown AION. Diabetes mellitus is common in patients with asymptomatic optic disc edema. Perhaps patients diagnosed as having diabetic papillopathy actually have an impending AION that does not progress to overt disease.

Aged↗

PubMed2002

[Bilateral acute angle closure glaucoma following topiramate treatment].

We describe a case of bilateral acute angle closure glaucoma associated with oral topiramate therapy. A 64 year old woman developed bilateral acute angle closure glaucoma two weeks after beginning topiramate therapy for peripheral diabetic neuropathy. A topical and systemic anti-glaucomatous treatment were given and laser peripheral iridotomies were performed and, the intraocular pressure were stabilized. However, the anterior chambers remained very shallow and peripheral ciliochoroidal detachment was observed and confirmed echographically. Assuming that the mechanism of acute bilateral simultaneous angle closure glaucoma differs from the common pupillary block, and is related to external cause, the topiramate therapy was discontinued, and the patient was treated with cycloplegic agent and steroids. A gradual deepening of the anterior chamber and resolution of the choroidal edema were accompanied by improvement of visual acuity and corneal clarity. The presumed mechanism of drug related acute bilateral angle closure glaucoma is choroidal effusion and detachment associated with forward budging of the iris-lens diaphragm. This, in turn, causes a shallow anterior chamber and blockage of the ocular draining system.

Cornea↗

PubMedDate not supplied

Adie's tonic pupil-induced angle-closure glaucoma.

A 52-year-old woman was diagnosed as having Adie's tonic pupil in her right eye. She reported few episodes of blurring of vision in her right eye in the recent few months. On one of the routine follow-up visits, right intraocular pressure (IOP) was 70 mm Hg and on gonioscopy the angle was closed 360 degrees. Medical treatment resulted in IOP reduction and laser iridotomy was then performed. This is, to our knowledge, the first description of intermittent angle-closure glaucoma attacks induced by a tonic pupil. The possibility of angle-closure glaucoma should be considered in patients with a tonic pupil, especially with symptoms of blurred vision or ocular pain.

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