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Biomedical subjects

Y Darbois

Publications and source records attributed to Y Darbois.

At least 73 records · Page 4Linked to original sources
PubMed1986

[Facial paralysis of obstetrical origin. Etiology, evolution].

In the case of 17 obstetrical facial paralyses occurring in a total of 20,409 births, the authors have studied the circumstances of their appearance and the prognosis for this lesion. It is a rare lesion favoured by asymmetric purchase when using articulated forceps. The authors insist that use of Thierry's forceps do not harm the facial nerve. They emphasise the possibility that progression may not always be favourable.

Birth Injuries↗
PubMed1985

[Obstetrical injuries of the newborn infant. Retrospective study apropos of 20,409 births].

We have been able to find 291 cases of obstetric trauma when we analysed the 20,409 deliveries which took place in the Pitié maternity between 1971 and 1980. It has been shown that bony lesions did well in the long and short term but nerve lesions had a far less good prognosis. This pathology should become far less when it is possible to recognize better when the fetus is large and when such traumatising manoeuvres as high forceps and assisted deliveries of large breech babies are stopped.

Birth Injuries↗
PubMed1983

Multiple acyl-CoA dehydrogenase deficiency occurring in pregnancy and caused by a defect in riboflavin metabolism in the mother. Study of a kindred with seven deaths in infancy: Value of riboflavin therapy in preventing this syndrome.

Seven infants in one kindred died: one was stillborn; the others, who were floppy at birth and were breast-fed, developed a disorder with the odor of sweaty feet and died in early infancy. In two further pregnancies, 3-hydroxvisovaleric, glutaric, and C6-C10-dicarboxylic acids were demonstrated in the mother's urine during the seventh month. Riboflavin therapy in the last trimester of pregnancy and a riboflavin-rich diet given the infants prevented this syndrome. The presence of abnormal erythrocyte glutathione-reductase activity in the mother while she excreted normal amounts of riboflavin in her urine indicates a probable disorder of riboflavin metabolism resulting in multiple acyl-CoA dehydrogenase deficiency.

Acyl-CoA Dehydrogenases↗