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Weiguo Chen

Publications and source records attributed to Weiguo Chen.

2 recordsLinked to original sources

Genetic mutations within tva receptor gene confer resistance to ALV-A and ALV-K infection in chickens.

Avian leukosis virus subgroup A (ALV-A) and the newly emerging subgroup K (ALV-K) are primary causative pathogens of avian leukosis in chickens and continue to pose a persistent threat to poultry industry worldwide. Susceptibility or resistance of chicken cells to ALV-A and ALV-K is determined by the tva receptor gene. Indeed, six ALV-A resistant alleles (tvar1-tvar6) have been identified within the tva gene. However, whether these tva alleles confer resistance to ALV-K infection remains unclear. In this study, we identified five tva resistant alleles, namely tvar2, tvar3, tvar4, tvar5, and tvar6, in Qingyuan partridge chickens (QYPC). In vitro analyses, including flow cytometry and quantitative real-time PCR, we demonstrated that the tvar2 allele confers complete resistance to both ALV-A and ALV-K infection. In contrast, the tvar3, tvar4, tvar5, and tvar6 alleles each significantly reduced susceptibility to ALV-K. Furthermore, these in vitro observations were corroborated by in vivo infection experiments, which confirmed that the tvar2 allele consistently conferred complete resistance to both ALV-A and ALV-K, while tvar3, tvar4, tvar5, and tvar6 alleles each resulted in a significant reduction in ALV-K susceptibility. Additionally, the tvar2-tvar6 alleles were broadly distributed across QYPC lines, with notably high frequencies of resistant genotypes observed in specific QYPC populations. Collectively, these findings not only expand our understanding of tva-mediated resistance to both ALV-A and ALV-K but also provide valuable genetic targets for selective breeding programs aimed at enhancing resistance to ALV-A and ALV-K in chickens.

ALV-A

QTL mapping for seed vigor-related traits under artificial aging in common wheat in two introgression line (IL) populations.

BACKGROUND: Seed vigor recognized as a quantitative trait is of particular importance for agricultural production. However, limited knowledge is available for understanding genetic basis of wheat seed vigor. METHODS: The aim of this study was to identify quantitative trait loci (QTL) responsible for 10 seed vigor-related traits representing multiple aspects of seed-vigor dynamics during artificial aging with 6 different treatment times (0, 24, 36, 48, 60, and 72 h) under controlled conditions (48 °C, 95% humidity, and dark). The mapping populations were two wheat introgression lines (IL-1 and IL-2) derived from recipient parent (Lumai 14) and donor parent (Shaanhan 8675 or Jing 411). RESULTS: A total of 26 additive QTLs and 72 pairs of epistatic QTLs were detected for wheat seed-vigor traits. Importantly, chromosomes 1B and 7B contained several co-located QTLs, and chromosome 2A had a QTL-rich region near the marker Xwmc667, indicating that these QTLs may affect wheat seed vigor with pleiotropic effects. Furthermore, several possible consistent QTLs (hot-spot regions) were examined by comparison analysis of QTLs detected in this study and reported previously. Finally, a set of candidate genes for wheat seed vigor were predicted to be involved in transcription regulation, carbohydrate and lipid metabolism. CONCLUSION: The present findings lay new insights into the mechanism underlying wheat seed vigor, providing valuable information for wheat genetic improvement especially marker-assisted breeding to increase seed vigor and consequently achieve high grain yield despite of further investigation required.

Triticum