[Practical application of histotopochemical methods in determining subclinical psoriasis].
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Diagnosis, Differential↗
Biomedical subjects
W Wohlrab
Publications and source records attributed to W Wohlrab.
[Mechanical separation of epidermis and cutis].
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Histological Techniques↗
[Modification of the specificity of the histotopochemical demonstration of 5'-nucleotidase].
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Adenine Nucleotides↗
[Hyaluronidase content of malignant melanoma].
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Humans↗
[Cytophotometric measurements on the vitro cultivated epidermis-cells of skin of patients with psoriasis vulgairs].
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Culture Techniques↗
[On the distribution and activity of hydrolytic enzymes in clinically healthy epidermis of psoriatics].
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Acid Phosphatase↗
[On the problem of identity of the epidermal barrier and the intermediary zone of human skin].
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Esterases↗
[On the histotopography of the phosphomonoesterases in focal psoriasis and its periphery. 3. 5' nucleotidases].
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Adolescent↗
[Histochemical findings in psoriasis focal areas and their immediate vicinity during healing by Cignolin treatment].
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Acid Phosphatase↗
Induction of antiproliferative effects in HaCaT cells by a synthetic hydroperoxide.
The effects of decylhydroperoxide (DHP) on proliferation were assessed in HaCaT keratinocytes. DHP, a model compound of lipid peroxidation, reduced proliferation dose dependently. The analyses of cell number, DNA synthesis and cell cycle distribution suggest a delayed progression through S-phase of DHP-treated HaCaT cells. It has been proven that necrotic and apoptotic cell death were also causes for the DHP-induced decrease in proliferation. Moreover, in HaCaT cultures treated with the highest DHP concentration, apoptosis occurred to a much greater extent thus playing an overproportional role in the decline in living cell number. Results with the radical scavenger ascorbic acid and the iron chelator deferoxamine support the conclusion that an iron-catalyzed radical formation from DHP may induce the DHP-mediated reduction in proliferation.
Apoptosis↗