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Biomedical subjects

V Toso

Publications and source records attributed to V Toso.

At least 37 records · Page 2Linked to original sources
PubMed1981

Gliomatosis cerebri diffusa. A case report.

The clinico-pathologic findings in an additional case of gliomatosis cerebri are reported: a 60-year-old woman died 8 months after the onset of a progressive deterioration of both the neurologic and mental conditions. Neuropathologic examination disclosed wide demyelination of both hemispheres, communicating through the corpus callosum, extending downward along the internal capsule to the brainstem structures. Cellular stains showed the presence of elongated astrocytes, multinucleated cells, mitotic and anaplastic figures, involving the demyelinated areas and the neighboring regions, and allowed the diagnosis of gliomatosis cerebri diffusa. The nosologic and pathogenetic aspects of this rare entity are discussed.

Autopsy↗
PubMed1980

Tapia's syndrome caused by a neurofibroma of the hypoglossal and vagus nerves: case report.

Tapia's syndrome is characterized by unilateral paralysis of the tongue and vocal cord, and is caused by a lesion of the 10th and 12th cranial nerves below the nodose ganglion, without involvement of the pharyngeal branches of the 10th nerve. The authors report the case of a 25-year-old man who presented with a 4-year history of progressive glossolaryngeal paralysis. Operation through the right laterocervical region allowed complete removal of a neurofibroma involving the 10th and 12th nerves at their crossing below the nodose ganglion.

Adult↗
PubMedDate not supplied

Executive deficits appearing in the initial stage of Alzheimer's disease.

The presence of executive deficits has been sought at a stage of Alzheimer's disease where currently used neuropsychological batteries could not yet distinguish Alzheimer's patients from normal age- and education-matched controls. This study shows that, at this early stage, those patients that 6 months later are found to show clear signs of Alzheimer's had been significantly worse than normal controls in an executive function task adapted from the Brown-Peterson procedure.

Adult↗
PubMedDate not supplied

A componential analysis of visual object recognition deficits in patients with herpes simplex virus encephalitis.

Five patients with a diagnosis of Herpes Simplex Virus Encephalitis (HSVE) underwent neuropsychological assessment to explore the integrity of their visual perceptual abilities. Selective deficits affecting different levels of the recognition processing were found; impaired recognition abilities were also influenced by selective task requirements, which resulted either in facilitatory or constraining effects on patients' performance. A theoretical model of object recognition (Humphreys & Riddoch, 1987) was taken into account to explain patients' performance. Further, the role of specific components of visual processing was evidenced in explaining the performance of patients affected by HSVE.

Adult↗
PubMedDate not supplied

Memory for/and execution of future intentions: evidence from patients with Herpes Simplex Encephalitis.

Prospective remembering was studied in a group of patients who suffered from Herpes Simplex Encephalitis (HSE). All patients showed a marked deficit in executing intentions for future actions under all the given constraints. The deficit extended to both time- and event-based intentions. The analysis of errors showed somewhat different patterns and some dissociations with evidence for selective preservation or damage to specific components involved in prospective remembering. These patients, in fact, may fail because of a difficulty in taking into account all given constraints or in activating stored intentions and in forming intentions or in remembering the content of the actions. Time- and event-based tasks seem to show different sources of errors also in the same patient.

Adult↗
PubMedDate not supplied

[Four cases of Creutzfeldt-Jakob's disease (author's transl)].

The clinico-pathological features of 4 cases of Creutzfeldt-Jakob disease, a transmissible virus dementia, are reported. The onset of the disease varied between 54 and 81 years of age: the course is dramatic and the demise occurs 1-4 months after the onset of symptoms. The clinical picture includes mental deterioration with amnesia, aphasia, apraxia and neurologic symptoms, mainly consisting in progressive rigidity and myoclonus. All cases show the typical 1/sec pseudorithmic spikes on EEG. Histological changes include severe astrocytosis of the cerebral cortex, status spongiosus and mild neuronal loss. The changes are more marked in the parieto-occipital lobes. Criteria of clinical and pathological diagnosis are reviewed. The precautions to be taken in handling biological as well as bioptic and autoptic specimens are emphasized, in order to avoid accidental transmission of the disease. It seems essential to institute a register of cases of Creutzfeldt-Jakob disease in Italy to assess the incidence and diffusion of the disease in our country.

Aged↗