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Biomedical subjects

T Ban

Publications and source records attributed to T Ban.

At least 271 records · Page 15Linked to original sources
PubMed1978

Subcortical afferents to the prefrontal cortex in rabbits.

The origins of cells projecting to the prefrontal cortex of the rabbit were studied, using horseradish peroxidase (HRP) technique. HRP injected into the prefrontal cortex labeled cells in the basal forebrain, lateral hypothalamus, raphe nuclei and locus coeruleus area on both sides. Labeled cells appeared also in the nucleus medialis dorsalis of the thalamus and other thalamic nuclei on the injection side.

Afferent Pathways↗
PubMed1975

Metabolic and structural recovery of left ventricular canine myocardium from regional complete ischemia.

The capacity for recovery of the normothermic left ventricular myocardium from a regional complete ischemia (RCI) was investigated using changes in the myocardial metabolic status (ATP, ADP, AMP, creatine phosphate (CrP), free creatine, glycogen, glucose, lactate) and alterations of the morphology as parameters. In dogs, an area of the anterior wall of the left ventricular myocardium was temporarily deprived completely of its blood supply by 5--7 overlapping ligatures extending into the heart cavity. The metabolites of the adenylic acid-CrP system returned to normal tissue levels after 30 and 60 min of RCI within 14 and 35 days of recovery, respectively; restoration averaged 82% after 100 min, 74% after 140 min, and 38% after 180 min of RCI after 5 weeks of recovery. At the same time glycogen amounted to 163% after 100 min, 114% min, and 65% after 180 min of RCI. The biochemical data correlated well with the structural changes in the affected myocardium, especially with the amount of de- and regenerating heart muscle cells. These obviously were functionally defect and were not comparable with normal structured and functioning heart muscle cells.

Adenosine Diphosphate↗
PubMed1975

Fiber connections in the hypothalamus and some autonomic functions.

The ventromedial hypothalamic nucleus (VMH) belongs to area sympathica B of the SPH-System and its fibers to make up the dorsal longitudinal fasciculus and the hypothalamicotegmental tract. Blood glucose increases by rapid increase in active phosphorylase and G-6-Pase activities in liver following VMH stimulation. From the changes of ultrastructure of the hypophysis and the pancreas, GH, ACTH, LH, FSH and TSH (but not prolactin) are thought to be released by each releasing hormone increased by VMH stimulation. D cell hormone is also released and insulin is not released following VMH stimulation. The lateral hypothalamic nucleus (LH) belongs to area parasympathica C of the SPH-System and its fibers to make up the medial forebrain bundle and stria medullaris. Blood glucose decreases by relatively rapid increase in glycogen synthetase activity in the liver following LH stimulation. From the changes of ultrastructure, GH, ACTH, LH, FSH, TSH and D cell hormone are not released, but prolaction and insulin are released following LH stimulation. By giving lesions in VMH or LH, some degenerated fibers could be seen in LH or VMH, homolaterally.

Adrenocorticotropic Hormone↗