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Steven H Strogatz

Publications and source records attributed to Steven H Strogatz.

8 recordsLinked to original sources

The size of the sync basin.

We suggest a new line of research that we hope will appeal to the nonlinear dynamics community, especially the readers of this Focus Issue. Consider a network of identical oscillators. Suppose the synchronous state is locally stable but not globally stable; it competes with other attractors for the available phase space. How likely is the system to synchronize, starting from a random initial condition? And how does the probability of synchronization depend on the way the network is connected? On the one hand, such questions are inherently difficult because they require calculation of a global geometric quantity, the size of the "sync basin" (or, more formally, the measure of the basin of attraction for the synchronous state). On the other hand, these questions are wide open, important in many real-world settings, and approachable by numerical experiments on various combinations of dynamical systems and network topologies. To give a case study in this direction, we report results on the sync basin for a ring of n >> 1 identical phase oscillators with sinusoidal coupling. Each oscillator interacts equally with its k nearest neighbors on either side. For k/n greater than a critical value (approximately 0.34, obtained analytically), we show that the sync basin is the whole phase space, except for a set of measure zero. As k/n passes below this critical value, coexisting attractors are born in a well-defined sequence. These take the form of uniformly twisted waves, each characterized by an integer winding number q, the number of complete phase twists in one circuit around the ring. The maximum stable twist is proportional to n/k; the constant of proportionality is also obtained analytically. For large values of n/k, corresponding to large rings or short-range coupling, many different twisted states compete for their share of phase space. Our simulations reveal that their basin sizes obey a tantalizingly simple statistical law: the probability that the final state has q twists follows a Gaussian distribution with respect to q. Furthermore, as n/k increases, the standard deviation of this distribution grows linearly with square root of n/k. We have been unable to explain either of these last two results by anything beyond a hand-waving argument.

Journal Article↗

Theoretical mechanics: crowd synchrony on the Millennium Bridge.

Soon after the crowd streamed on to London's Millennium Bridge on the day it opened, the bridge started to sway from side to side: many pedestrians fell spontaneously into step with the bridge's vibrations, inadvertently amplifying them. Here we model this unexpected and now notorious phenomenon--which was not due to the bridge's innovative design as was first thought--by adapting ideas originally developed to describe the collective synchronization of biological oscillators such as neurons and fireflies. Our approach should help engineers to estimate the damping needed to stabilize other exceptionally crowded footbridges against synchronous lateral excitation by pedestrians.

Biological Clocks↗

Chimera states for coupled oscillators.

Arrays of identical oscillators can display a remarkable spatiotemporal pattern in which phase-locked oscillators coexist with drifting ones. Discovered two years ago, such "chimera states" are believed to be impossible for locally or globally coupled systems; they are peculiar to the intermediate case of nonlocal coupling. Here we present an exact solution for this state, for a ring of phase oscillators coupled by a cosine kernel. We show that the stable chimera state bifurcates from a spatially modulated drift state, and dies in a saddle-node bifurcation with an unstable chimera state.

Journal Article↗

Modeling a synthetic multicellular clock: repressilators coupled by quorum sensing.

Diverse biochemical rhythms are generated by thousands of cellular oscillators that somehow manage to operate synchronously. In fields ranging from circadian biology to endocrinology, it remains an exciting challenge to understand how collective rhythms emerge in multicellular structures. Using mathematical and computational modeling, we study the effect of coupling through intercell signaling in a population of Escherichia coli cells expressing a synthetic biological clock. Our results predict that a diverse and noisy community of such genetic oscillators interacting through a quorum-sensing mechanism should self-synchronize in a robust way, leading to a substantially improved global rhythmicity in the system. As such, the particular system of coupled genetic oscillators considered here might be a good candidate to provide the first quantitative example of a synchronization transition in a population of biological oscillators.

Bacterial Proteins↗

Synchronization in oscillator networks with delayed coupling: a stability criterion.

We derive a stability criterion for the synchronous state in networks of identical phase oscillators with delayed coupling. The criterion applies to any network (whether regular or random, low dimensional or high dimensional, directed or undirected) in which each oscillator receives delayed signals from k others, where k is uniform for all oscillators.

Journal Article↗

Simple model of epidemics with pathogen mutation.

We study how the interplay between the memory immune response and pathogen mutation affects epidemic dynamics in two related models. The first explicitly models pathogen mutation and individual memory immune responses, with contacted individuals becoming infected only if they are exposed to strains that are significantly different from other strains in their memory repertoire. The second model is a reduction of the first to a system of difference equations. In this case, individuals spend a fixed amount of time in a generalized immune class. In both models, we observe four fundamentally different types of behavior, depending on parameters: (1) pathogen extinction due to lack of contact between individuals; (2) endemic infection; (3) periodic epidemic outbreaks; and (4) one or more outbreaks followed by extinction of the epidemic due to extremely low minima in the oscillations. We analyze both models to determine the location of each transition. Our main result is that pathogens in highly connected populations must mutate rapidly in order to remain viable.

Disease Outbreaks↗