Orbital ganglioneuroma in a patient with chronic progressive proptosis.
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Biomedical subjects
Publications and source records attributed to Steven B Flynn.
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PURPOSE: To document reversible corneal edema caused by amantadine in a pediatric patient. METHODS: A 14-year-old boy with a neurologic tremor was referred for bilateral visual loss. Our examination disclosed bilateral corneal edema without ocular inflammation. Pachymetry confirmed significantly increased corneal thickness above 900 microm. RESULTS: Review of the patient's medical information revealed recent institution of amantadine as a means to control the patient's tremor. On cessation of this agent, rapid resolution of corneal edema and recovery of visual acuity occurred. Repeat pachymetry measurement revealed normal corneal thickness. CONCLUSION: In cases of corneal edema and in the absence of any identifiable ocular causes, a review of toxic effects of systemic medication should be undertaken. Amantadine can cause corneal decompensation and needs to be considered as part of the differential diagnosis of corneal edema.
A man with advanced malignant disease presented complaining of vision changes in the left eye. Examination revealed an afferent pupillary defect and visual field defect in that eye. Imaging indicated carcinomatous disease of the left optic nerve, and a diagnosis of carcinomatous optic neuropathy was made.
A 29-year-old African American man presented to the emergency department at the University of Arkansas for Medical Sciences with a fishhook perforating the cornea of the left eye (OS) extending into the anterior chamber. The fishhook was removed via its entrance wound and was subsequently repaired. Fishhook injuries to the eye can result in profound visual impairment. Management of these injuries is dependent on the involved ocular structures and location of the hook. Visual prognosis can be profoundly affected by the initial management. Surgical management options are reviewed.