Seroepidemiological studies of transfusion-associated hepatitis.
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Adult↗
Biomedical subjects
S Wood
Publications and source records attributed to S Wood.
Mechanisms of establishment of tumor metastases.
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Animals↗
Effects of ultraviolet irradiation on heterozygous diploids of Aspergillus nidulans. I. UV-induced mitotic crossing over.
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Aspergillus↗
Rehabilitation of the mental patient.
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Humans↗
In vivo studies on the intravascular changes produced by antilymphocyte serum.
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Animals↗
Mechanisms of absorption of catgut sutures in vivo.
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Absorption↗
Motility of granulocytes during wound healing in the rabbit ear chamber.
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Animals↗
Inhibition of locomotion of cancer cells in vivo by anticoagulant therapy. I. Effects of sodium warfarin on V2 cancer cells, granulocytes, lymphocytes and macrophages in rabbits.
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Animals↗
Twin spots as evidence for mitotic crossing-over in Aspergillus induced by ultraviolet light.
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Aspergillus↗
Failure of low molecular weight dextrans to alter the frequency of lung metastasis. Report on the V2 carcinoma of the rabbit.
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Animals↗
History and development of occupational therapy.
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Nursing↗
A bioassay of circulating tumor cells.
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Animals↗
Effects of DMSO on blood and vascular endothelium.
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Animals↗
DMSO precipitates tissue destruction.
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Animals↗
A long-term study and correlation of lymphocyte and neutrophil function in the patient with burns.
Twenty-five patients were studied to determine the effects of thermal injury on neutrophil bactericidal function and superoxide release and on lymphocyte proliferation. Neutrophils in patients with burns had depressed killing of Staphylococcus aureus for more than 150 days after burn injury, but killing of Escherichia coli returned to normal. FMLP-stimulated superoxide release by neutrophils in patients with burns was depressed for over 100 days after burn injury, whereas superoxide release by neutrophils in patients with burns stimulated with serum-opsonized zymosan was depressed for 42 days after burn injury. In patients with burns lymphocyte proliferation, with phytohemagglutinin as a mitogen, was suppressed for up to 85 days after injury, then returned to normal. The mixed lymphocyte response was suppressed up to 170 days after injury.
Adolescent↗
The increased release of prostaglandin E2 by Kupffer cells from burned guinea pigs.
Prostaglandin E2 (PGE2) is a very important immunosuppressive substance that is synthesized and released by all macrophages including Kupffer cells (KCs). In this study the changes of PGE2 released by KCs were evaluated in a burned guinea pig model. Prostaglandin E2 was released by KCs from burned guinea pigs at a consistently and significantly high level when stimulated with endotoxin. On postburn day 8, KCs that were cocultured with hepatocytes released significantly less PGE2. There was also a significant decrease in antibody-dependent cell-mediated cytotoxicity on postburn day 1. Kupffer cells can mediate immune suppression in burn injury by a prolonged increased production of PGE2 that has immunosuppressive effects on other cells and by a decreased cytotoxic effect soon after burn injury.
Animals↗