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Biomedical subjects

S Pattin

Publications and source records attributed to S Pattin.

At least 55 records · Page 3Linked to original sources
PubMed1984

[Dissecting osteochondritis of the patella. Apropos of 12 cases].

The authors report 12 cases of osteochondritis dissecans of the patella. The relative rarity of this condition (one hundred published cases) is perhaps only apparent, as the diagnosis is sometimes difficult early in the disease. Drawing from their personal cases and from data in the literature, the authors recall the clinical and radiological signs, describe the place of complementary investigations and therapeutic indications and discuss the various pathogenic theories, none of which are fully satisfactory.

Adolescent↗
PubMed1983

[Recurrent familial hyperparathyroidism. A propos of 7 adenomas in 3 members of the same family. Review of the literature].

Over a period of 23 years, 3 members of a family of 5 presented with 7 parathyroid adenomas (4 in the first case, 2 in the second case, 1 in the third case). Excision of each adenoma, with systematic pre-operative assessment of the remaining parathyroid tissue, led to complete clinical and laboratory cure of each episode. The delay in the appearance of recurrence was between 3 and 9 years. After reviewing the literature, the authors stress the importance, in any case of hyperparathyroidism, of routinely investigating the serum calcium levels in members of the patient's family, especially if the patient is young and if he has had several episodes or multiglandular involvement in one episode. The authors discuss the literature concerning recurrent familial hyperparathyroidism with endocrine polyadenomatosis and the "hypercalcaemia-hypocalciuria" syndrome.

Adenoma↗
PubMed1983

[Involvement of the thoracic was in algodystrophy. Apropos of a case report].

With reference to an unusually severe and extensive case of algodystrophia, the sternal and costal localizations of this disorder are described. These fit the definition of the painful anterior thoracic wall syndrome, described i 1955 by Printzmetal and Massumi, which may complicate chronic coronary insufficiency or myocardial infarction or occur as a primary disease.

Bone Diseases, Metabolic↗
PubMed1983

[Clinical aspects of reactive arthritis caused by Chlamydia].

The authors compare a multicentered study of chlamydial reactive arthritis with their own personal series of 99 cases consisting of 58 cases of chlamydial reactive arthritis and 41 cases of non-chlamydial reactive arthritis. In the group with chlamydial reactive arthritis, mono-arthritis is more frequent and involvement of the knees, hands, wrists, the sausage appearance of the fingers and toes and heel pain are rarer than in non-chlamydial reactive arthritis. However, the genetic predisposition, in particular the presence of the HLA-B27 antigen, has an essential influence on the clinical features of reactive arthritis.

Adolescent↗
PubMed1983

[Familial and recurrent hyperparathyroidism. Apropos of 7 adenomas in 3 members of the same family. Review of the literature].

Over a period of 23 years, 3 members of a family of 5 presented with 7 parathyroid adenomas (4 in the first case, 2 in the second case, 1 in the third case). Excision of each adenoma, with systematic pre-operative assessment of the remaining parathyroid tissue, led to complete clinical and laboratory cure of each episode. The delay in the appearance of recurrence was between 3 and 9 years. After reviewing the literature, the authors stress the importance, in any case of hyperparathyroidism, of routinely investigating the serum calcium levels in members of the patient's family, especially if the patient is young and if he has had several episodes or a multiglandular involvement in the one episode. The authors discuss the literature concerning recurrent familial hyperparathyroidism with endocrine polyadenomatosis and the "hypercalcaemia - hypocalciuria" syndrome.

Adenoma↗
PubMed1983

[Value of dapsone in the treatment of Horton's disease and rhizomelic pseudopolyarthritis].

The authors report two cases of Horton's disease and one case of rhizomelic pseudopolyarthritis treated initially with delta-cortisone with a very rapid favourable result. However, while under relatively high dose steroid therapy, there was recurrence of the disease in the three cases and the development of complications of steroid therapy prohibited the use of higher doses of delta-cortisone and even required its rapid weaning. In these three cases, the addition of 100 mg of dapsone per day led to a rapid control of the disease, with return to normal of the erythrocyte sedimentation rate and reduction of the doses of delta-cortisone. The authors discuss the place of dapsone in the treatment of Horton's disease and rhizomelic pseudopolyarthritis in the light of these three cases.

Aged↗
PubMed1982

[Immunologic aspects of ankylosing spondylarthritis].

The immunological profile of 63 men, 53 of whom were carriers of the HLA B27 antigen, and 10 of whom were not, all of whom suffered from ankylosing spondylarthritis (ASP) which was either quiescent or subject to exacerbations, were studied: lymphocytic colonies, quantity determination of serum proteins, investigations of auto-immune antibodies. Following a discussion of the techniques, the results are presented and compared with those obtained in healthy subjects. No significant difference was revealed between the averages obtained for the results on the patients and the controls, nor as regards the B and T lymphocytes, the IgG, IgA, IgM immunoglobulins, the C3 fraction of the complement, or orosomucoid. The haptoglobin and alpha-antitrypsin rates increased significantly in the patients. Tests for the auto-immune antibodies were always negative. The results are compared to other, often contradictory, studies which have already been published. These authors conclude that the ASP in question does not seem to be an immunological disease.

Adult↗
PubMed1981

[A new clinical entity: "the algodystrophic intermittent claudication of the lower limbs syndrome" (author's transl)].

A new clinical entity is proposed by the authors, "the algodystrophic intermittent claudication of the lower limbs syndrome", characterised by diffuse pains in the foot, having neither a constrictive nature, nor a radicular distribution. The pain appears after walking a certain distance, forcing the patient to stop, and reappears when he starts to walk again. Bone isotopic examinations and repeated radiographic investigations should be conducted when confronted with such a clinical syndrome in order to confirm the diagnosis of algodystrophy. In view of the polymorphic nature of this affection, it is not surprising that new clinical aspects can still be described at the present time.

Arteritis↗