[X-ray television in dentistry].
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Biomedical subjects
Publications and source records attributed to S Ota.
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Twenty-five years after a blunt chest trauma, sudden expansion of a traumatic aortic aneurysm induced a dysphagia and chest pain in a 57-year-old man. Resection of the aneurysm and patch repair of the rupture site utilizing a thoracoabdominal temporary bypass was successful. The location of aortic laceration at the thoracoabdominal junction was most unusual as compared with traumatic aneurysms usually seen in the thoracic aorta.
In the present study, we demonstrated that hypertensive left ventricular hypertrophy can be divided angiographically into symmetrical (SH) and asymmetrical septal hypertrophy (ASH) groups. In the SH group, the dynamics of the septal wall and the free wall were almost the same as those of the control group. On the other hand, the ASH group rather resembled the HCM group in the thickness and dynamics of the septal wall and the free wall as well as in the septal configuration. The most characteristic difference of the ASH and HCM groups from the SH group was in the significantly greater thickness of the septal wall at end diastole. However, in view of the developmental mechanism of hypertensive heart, it is interesting to note that the more the septal-free wall ratio increases, the more the thickness of the septal wall increases in the SH group.
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An instance of mitral valve prolapse syndrome demonstrating early diastolic click was reported. From the phonographic analysis of the cardiac sounds with simultaneous recording of apexcardiogram or echocardiogram, the production mechanism of the diastolic click was discussed.
Supravital excitability to electrical stimuli (for convenience, supravital electrocontractility (S.Ec) of the gastrocnemius muscle of rats and bullfrogs was examined after "somatic death" and the time during which S.Ec could be detected (S.Ec duration) was measured. S.Ec of the gastrocnemius muscle of rats and bullfrogs depended on temperature. The maximum S.Ec duration of the muscle of rats and bullfrogs were 110 min and 96 hr, respectively, at a low temperature (5 degrees), and 60 min and 9 hr, respectively, at a high temperature (30 degrees). The time course of rigor mortis at 5 degrees was slow and it was rapid at 30 degrees. In rats, the S.Ec disappeared completely before the onset of rigor mortis, and in bullfrogs, S.Ec existed at progressive stages of rigor mortis and it disappeared completely when rigor mortis reached about the maximum.