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Biomedical subjects

S Kimata

Publications and source records attributed to S Kimata.

At least 73 records · Page 4Linked to original sources
PubMed1985

[Digital perfusion images for evaluating patients with mitral valve replacement].

To determine whether the distribution of pulmonary perfusion can be applied as a noninvasive means of evaluating patients with mitral valve replacement (MVR), computerized Tc-99m MAA pulmonary perfusion images (digital perfusion images: DPI) were obtained for 32 patients in the preoperative, early postoperative and late postoperative (18 months mean) periods. DPI consisted of isocount areas, and a 100-70% area was defined as a hyperperfusion area. The distribution of pulmonary perfusion was evaluated using patterns of hyperperfusion area in anterior DPI. In 32 patients above-mentioned had the hyperperfusion areas in the upper lung fields preoperatively, the perfusion of the lung base was investigated. In 21 patients hyperperfusion area appeared in the lung base in the late follow-up period (group A), and all patients improved clinically. In 11 patients, the DPI improved to nearly normal patterns. In five patients, the DPI improved after one year postoperatively. In 11 patients, hyperperfusion areas did not appear in the lung base in the late follow-up period (group B). Six of the 11 patients did not improve clinically. There were no significant hemodynamic differences between groups A and B except for slight differences in the postoperative pulmonary vascular resistance. These results suggested significant correlations between the clinical improvement and the normalization of the DPI. The use of DPI patterns may facilitate quantitative and objective estimations of postoperative states. Since DPI are easy to perform noninvasively, DPI may comprise a useful graphic diagnostic method for evaluating patients with MVR.

Adolescent↗
PubMed1985

[Myocardial damage (myocytolysis) caused by subarachnoid hemorrhage].

A case of subarachnoid hemorrhage complicated by neurogenic pulmonary edema and neurogenic myocardial damage is reported. A 50-year-old woman was admitted following the sudden onset of headache and disturbance of consciousness due to a ruptured internal carotid posterior communicating artery aneurysm on the right side. She showed respiratory failure due to pulmonary edema, which subsequently improved with the mechanical ventilation. After that, she manifested chest distress and hypotensive episode then occurred. An ECG showed QS wave and ST elevation which suggested the presence of inferolateral myocardial damage. Subsequent rises in serum GOT, GPT, LDH and CPK were noticed. CPK-MB and LDH I and V isozyme levels rose. An echo cardiogram showed hypokinesis of the apical half of the left ventricular septum. The patient died on 5th hospital day due to rerupture of the cerebral aneurysm. Autopsy revealed diffuse myocytolysis with coagulation necrosis of the heart muscle without occlusion of coronary arteries. A small hemorrhagic lesion was found in the hypothalamus. We suggested that a hypothalamic lesion due to subarachnoid hemorrhage stimulated the sympathetic nervous system which in turn discharged endogenic catecholamine. This was probably accompanied by vasospasm of the coronary arteries and systemic peripheral arterioles. Furthermore, myocardial oxygen consumption could have been increased by the increase in catecholamine. Finally, it gave rise to neurogenic pulmonary edema and extensive diffuse myocytolysis of the heart occurred.

Cardiomyopathies↗
PubMed1983

[Unstable angina].

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Angina Pectoris↗