Myocardial sinusoids in pulmonary valvular atresia.
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Biomedical subjects
Publications and source records attributed to S H Cornell.
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Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
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Suprasellar arachnoid cysts are basal midline masses that represent a rare but surgically remediable cause of hydrocephalus and neurologic deficits. These cysts represent a diagnostic challenge and often go unrecognized for many years. The authors review the computed tomographic (CT) findings in seven patients with documented suprasellar arachnoid cysts and define previously undescribed diagnostic criteria. These cysts usually can be differentiated from cystic midline neoplasms by their CT density, homogeneity, and location as well as by their lack of fat, lack of calcification, and absence of contrast enhancement. Accurate distinction from marked third-ventricular enlargement secondary to obstructive hydrocephalus and from third-ventricular ependymal cysts can be made on the basis of basal cisternal expansion, distinctive mass effect and displacement, the characteristic shape and contour of the apparent "third ventricle," and the appearance of structures at the foramen of Monro. Although metrizamide CT ventriculography and cisternography allow confirmation of the diagnosis and evaluation of cerebrospinal fluid dynamics, these definitive studies will not be obtained unless the cysts are first suspected by their conventional CT appearance.
Suprasellar arachnoid cysts are an infrequent but surgically remediable cause of hydrocephalus and neurologic deficits. The optimal method of treatment varies according to whether the cyst communicates with the subarachnoid space; the presence or absence of associated hydrocephalus; and the exact site(s) and severity of intraventricular and extraventricular cerebrospinal fluid (CSF) obstruction. Eight patients with suprasellar arachnoid cysts are described to emphasize the importance of pretherapeutic investigation of CSF dynamics. The variability of the ventriculocisternal-cyst CSF dynamics and the value of metrizamide computed tomographic cisternography and ventriculography in investigation of the CSF pathways are stressed. A diagnostic approach to the evaluation of the CSF dynamics is outlined, based on whether hydrocephalic or compressive symptoms predominate.
Cerebral angiography in patients with nontraumatic lobar intracerebral hemorrhage may or may not uncover the underlying cause of the disorder. The CT and cerebral angiographic studies of 67 consecutive patients with nontraumatic lobar intracerebral hemorrhage were reviewed to assess the relationship between CT pattern and location of hemorrhage and the frequency of diagnostic angiographic findings. Origins of these hematomas were also determined and correlated with radiographic findings. CT revealed 26 temporal, 18 frontal, 17 parietal, three occipital, and three multiple lobar hematomas. Thirty-three patients had "pure" lobar hematomas, 12 had coexistent intraventricular hemorrhage, 12 had associated subarachnoid hemorrhage, and 10 had both intraventricular and subarachnoid hemorrhage accompanying their lobar hematomas. Angiographic findings were diagnostic in 29 cases (43%). In the presence of accompanying subarachnoid hemorrhage, angiographic findings were diagnostic in 17 (77%) of 22 patients; in its absence, angiography was diagnostic in 12 (27%) of the remaining 45 patients. Diagnostic angiograms were also more frequent in the presence of a frontal or temporal lobar hematoma than with a parietal or occipital lobar hematoma. While CT patterns do influence the frequency of diagnostic angiographic findings, cerebral angiography is recommended in all patients with otherwise unexplained nontraumatic lobar intracerebral hemorrhage.
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