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Richard E Tremblay

Publications and source records attributed to Richard E Tremblay.

At least 37 records · Page 2Linked to original sources

Youth gangs, delinquency and drug use: a test of the selection, facilitation, and enhancement hypotheses.

BACKGROUND: Three different explanations have been given for the observation that adolescent gang members report more delinquent behaviour than their counterparts who do not affiliate with gangs: a) adolescents who commit more crimes join gangs (selection hypothesis); b) gang membership facilitates deviant behaviour (facilitation hypothesis); c) selection and facilitation work interactively (enhancement hypothesis). The aim of this study was to test these hypotheses, while controlling for self-reported delinquency, friends' delinquency, and individual as well as family characteristics. METHOD: The sample included 756 boys first assessed when they attended kindergarten in disadvantaged areas of Montreal. Gang membership was assessed at the ages of 14, 15 and 16 years. Delinquency and drug use data were collected from self-reports and court files at the same ages. RESULTS: Gang members displayed far higher rates of delinquent behaviour and drug use than non-gang members. The results support the facilitation model for transient gang members (i.e., youths in a gang during only one of the three periods considered) and the enhancement model for stable gang members (i.e., youths in a gang for at least two of the periods considered), for person and property offences. The association between gang membership and delinquency persisted after introducing the control variables. Additional analyses showed that the effect associated with belonging to a gang was beyond that of simply having delinquent friends. CONCLUSION: Preventing the creation and participation in such gangs should reduce the frequency of antisocial behaviour during adolescence.

Adolescent↗

Individual change in methylphenidate use in a national sample of children aged 2 to 11 years.

OBJECTIVES: To determine methylphenidate use in children aged 2 to 13 years. To provide age- and sex-specific estimates of methylphenidate initiation and cessation during a 2-year period. METHOD: Data from 2 cycles of a Canadian household survey yielded a sample of over 10 000 children aged 2 to 11 years at Cycle 1 who continued to participate at Cycle 2. We used logit modelling to estimate Cycle 2 methylphenidate use, methylphenidate use over a 2-year period, and methylphenidate initiation and cessation from Cycles 1 to 2. RESULTS: In 1996 and 1997, methylphenidate use ranged from 0.32% to 6.31% among children aged 4 to 13 years. School-aged boys were more likely than girls to use methylphenidate. Odds were greater for boys aged 6 to 7 years than for boys aged 4 to 5 years; they were also greater for boys aged 10 to 11 years than for boys aged 12 to 13 years. Almost 1% of children used methylphenidate at both data cycles. Odds of Cycle 2 methylphenidate use were 135 times greater for children using methylphenidate at Cycle 1, compared with nonusers. Methylphenidate initiation ranged from 0.20% to 3.34%, and school-aged boys had higher initiation rates than girls. Cessation rates ranged from 18% to78%, and there were no statistically significant differences by age and sex. CONCLUSIONS: Methylphenidate prevalence findings are consistent with past studies. We found an age-by-sex interaction on methylphenidate use. We also found both continuity and discontinuity in methylphenidate use.

Age Factors↗

Sex and informant effects on diagnostic comorbidity in an adolescent community sample.

OBJECTIVE: To investigate sex and informant effects on comorbidity rates for anxiety disorders, depressive disorders, attention-deficit hyperactivity disorder (ADHD), and conduct-oppositional disorder (CD-ODD) in an adolescent community sample. METHOD: The Diagnostic Interview Schedule for Children-2.25 (DISC-2.25) was administered to 1201 adolescents and their mothers. RESULTS: The highest comorbidity risk found was between ADHD and CD-ODD, with odds ratios (ORs) of 17.6 for adolescent reports and 12.0 for mother reports. The second-highest comorbidity risk, with ORs of 13.2 for adolescent reports and 11.0 for mother reports, was between anxiety and depressive disorders. There was not much overlap between internalizing and externalizing disorders. Adolescent girls had higher rates of coexisting anxiety and depressive disorders, whereas adolescent boys had higher rates of coexisting ADHD and CD-ODD. There was partial support for the hypothesis that adolescent-reported comorbidity rates would exceed mother-reported rates. CONCLUSIONS: There is a greater cooccurrence of within-category, compared with between-category, disorders. Adolescent girls are more likely to have coexisting internalizing disorders, while adolescent boys are more likely to have coexisting externalizing disorders. Mothers tend to report more externalizing disorders (that is, ADHD), while adolescents generally report more internalizing disorders.

Adolescent↗

Construct validity of an instrument to assess major depression in parents in epidemiologic studies.

OBJECTIVE: The Longitudinal Study of Child Development in Quebec (LSCDQ) is a community study of children that examines the risk factors, such as parental depression, linked to childhood psychopathology and maladaptive functioning. Our goal was to test the construct validity of an instrument to assess major depression in the parents. METHOD: Parents of a representative sample of 2120 infants born in the province of Quebec in 1998 were selected. The major depression instrument was administered to the parents when the infants were aged 29 months. We tested the construct validity of the instrument by examining 1) sex differences in symptoms and prevalence, 2) the rank order of the prevalence of symptoms, 3) the clustering of symptoms, and 4) the association of depression with functional impairment and treatment-seeking behaviour. RESULTS: Depression was twice as common in mothers as in fathers. Most mothers and fathers who reported ever being depressed for 2 or more weeks had also experienced at least 4 additional depression symptoms. The rank order of symptom prevalence was comparable to that found in other studies. At least 50% of parents reported having talked to a professional and that suffering from depression had interfered with their functioning. CONCLUSIONS: The expected sex differences, the rank order of symptom prevalence, the symptom clustering, and the associations with functional impairment and treatment-seeking behaviour support the construct validity of a brief questionnaire on parental major depression.

Adult↗

A new look at inter-informant agreement on conduct disorder using a latent class approach.

This study examined agreement on aggressive and nonaggressive conduct disorder in a general population sample of 14- to 17-year-old adolescents (n=1165) and their mothers. We collected diagnostic interview data and applied latent class analyses to estimate inter-informant agreement. The preferred model for aggressive conduct disorder for both males and females was a one-latent-variable/two-class model specifying no inter-informant disagreement beyond chance expectations. This model estimated the prevalence of aggressive conduct disorder to be 13% for males and 0.4% for females. For nonaggressive conduct disorder, a one-latent-variable/three-class model specifying asymmetric agreement was preferred for both males and females. This model estimated the prevalence of nonaggressive conduct disorder in adolescents to be 18% according to males and 13% according to mothers. Prevalence estimates were 12% according to females and 7% according to mothers. Symptom sensitivity estimates for all models were poor whereas specificity estimates were near perfect to perfect. Males had higher rates of aggressive and nonaggressive conduct disorder across informants. There was a high level of adolescent-mother agreement on both types of conduct disorder. However, there were some differences, suggesting that aggressive and nonaggressive are two valid subtypes of conduct disorder with different prevalence estimates and agreement levels.

Adolescent↗

Trajectories of gambling during adolescence.

This study aimed at empirically identifying groups of adolescents with distinct longitudinal trajectories of gambling involvement and validating these groups by comparing them with respect to correlates. 903 low SES boys were followed annually from age 11 to 16 years. Three groups were found: an early-onset high-level chronic group, a late-onset high-level group, and a low gambler group. The Chronic group and the Low group consistently differed on teacher-rated inhibition (i.e., anxiety) during childhood and early adolescence. They also differed on concurrent teacher and self ratings of disinhibition (i.e., impulsivity), while the Late Onset group appeared to lie in between these groups. Compared to the Low group, both high groups subsequently had elevated scores on later gambling related problems.

Adolescent↗

Cognitive-neuropsychological function in chronic physical aggression and hyperactivity.

Histories of violence and of hyperactivity are both characterized by poor cognitive-neuropsychological function. However, researchers do not know whether these histories combine in additive or interactive ways. The authors tested 303 male young adults from a community sample whose trajectories of teacher-rated physical aggression and motoric hyperactivity from kindergarten to age 15 were well defined. No significant interaction was found. In a 1st model, both histories of problem behavior were independently associated with cognitive-neuropsychological function in most domains. In a second model controlling for IQ, General Memory, and test motivation, none of the three Working Memory tests (relevant to executive function) remained associated with physical aggression or hyperactivity. These results support an additive model but no specificity to executive function [corrected].

Adolescent↗

Adolescent gambling: relationships with parent gambling and parenting practices.

This study explored the possible links between family risk factors (i.e., parent gambling and parenting practices) and adolescent gambling. A community sample of 938 adolescents (496 females and 442 males) completed the South Oaks Gambling Screen Revised for Adolescents (SOGS-RA; K. C. Winters, R. Stinchfield, & J. Fulkerson, 1993b) along with a questionnaire assessing parenting practices. Both parents completed the SOGS (H. R. Lesieur & S. B. Blume, 1987). Results showed that adolescent gambling frequency was related to both parents' gambling frequency and problems. However, adolescent gambling problems were linked only to fathers' severity of gambling problems. Low levels of parental monitoring enhanced adolescents' risk of getting involved in gambling activities and developing related problems. A higher level of inadequate disciplinary practices was also related to greater gambling problems in youth. These links were significant after controlling for socioeconomic status, gender, and impulsivity-hyperactivity problems.

Adolescent↗

Cortisol levels and conduct disorder in adolescent mothers.

OBJECTIVE: To examine the association between cortisol levels and conduct disorder (CD) in adolescent mothers. Past research has shown that low levels of cortisol were associated with CD, particularly with its aggressive symptoms. The authors tested the hypothesis that adolescent mothers with CD would show lower levels of salivary cortisol compared to mothers without CD at 4 and 9 months postpartum. METHOD: Midmorning salivary cortisol levels were measured in 228 adolescent mothers (age at delivery 16.9 +/- 1 years [mean +/- SD]) during a laboratory visit at 4 and 9 months postpartum. CD was diagnosed during pregnancy according to the CD subsection on the criteria for antisocial personality disorder (DSM-III-R). RESULTS: Results did not confirm the hypothesis. Lower cortisol levels were not significantly associated with a CD diagnosis, the number of CD symptoms, or aggressive symptoms. CONCLUSIONS: Despite valid measures and strong statistical power, this study failed to find an association between cortisol levels and CD in a sample of adolescent mothers. The results may have been influenced by the fact that participants were 4 and 9 months postpartum and by comparisons of mothers with CD to mothers living in stressful circumstances.

Adolescent↗

Heightened heart rate response to alcohol intoxication is associated with a reward-seeking personality profile.

BACKGROUND: The psychomotor stimulant theory of addiction posits that sensitivity to the positively rewarding properties of alcohol puts certain individuals at higher risk for alcohol abuse. A valid and reliable index of overactivation in the reward system has been a heightened baseline heart rate (HR) increase on the ascending limb of the blood alcohol curve. The main goal of this study was to investigate the relationship between this HR response and a questionnaire measuring sensitivity to reward and sensitivity to punishment. Additional goals included looking at (1). the association between a high HR response and various personality traits (hopelessness/introversion, anxiety sensitivity, impulsivity, and sensation-seeking) and (2). the relationship between these personality traits and stimulant use. METHODS: A total of 18 low- and 19 high-HR responders completed the Sensitivity to Punishment and Sensitivity to Reward Questionnaire (SPSRQ), the Substance Use Risk Profile Scale (SURPS), and a modified version of the Addiction Severity Index. RESULTS: High-HR responders obtained significantly higher scores than low-HR responders on the sensitivity to reward scale of the SPSRQ, as well as increased sensation-seeking scores on the SURPS. High-HR responders were not at significantly higher risk of having used stimulants, but stimulant use was associated with higher impulsivity scores on the SURPS. CONCLUSIONS: Novelty/sensation-seeking is among the personality traits that have been linked to heavy alcohol use. This study suggests that reward sensitivity might mediate the relationship between this personality profile and drinking behavior.

Adolescent↗

Early childcare and physical aggression: differentiating social selection and social causation.

BACKGROUND: Some research findings have suggested that group day-care may be associated with an increased risk for physical aggression. METHODS: Cross-sectional maternal questionnaire data from a representative sample of 3431 Canadian 2- to 3-year-olds were used to compare rates of physical aggression shown by children looked after by their own mothers and those attending group day-care. A family risk index (using occupational level, maternal education, size of sibship, and family functioning) was created to test whether any difference in physical aggression might reflect social selection rather than social causation. RESULTS: Aggression was significantly more common in children looked after by their own mothers than in those attending group day-care. Strong social selection associated with family risk was found, not only in the sample as a whole, but even within the high-risk subsample. However, after taking social selection into account, physical aggression was significantly more common in children from high-risk families looked after by their own parents. No such difference was evident in the majority (84%) of children from low-risk families. CONCLUSION: Insofar as there are any risks for physical aggression associated with homecare they apply only to high-risk families.

Age Factors↗

Physical aggression during early childhood: trajectories and predictors.

OBJECTIVES: Physical aggression in children is a major public health problem. Not only is childhood physical aggression a precursor of the physical and mental health problems that will be visited on victims, but also aggressive children themselves are at higher risk of alcohol and drug abuse, accidents, violent crimes, depression, suicide attempts, spouse abuse, and neglectful and abusive parenting. Furthermore, violence commonly results in serious injuries to the perpetrators themselves. Although it is unusual for young children to harm seriously the targets of their physical aggression, studies of physical aggression during infancy indicate that by 17 months of age, the large majority of children are physically aggressive toward siblings, peers, and adults. This study aimed, first, to identify the trajectories of physical aggression during early childhood and, second, to identify antecedents of high levels of physical aggression early in life. Such antecedents could help to understand better the developmental origins of violence later in life and to identify targets for preventive interventions. METHODS: A random population sample of 572 families with a 5-month-old newborn was recruited. Assessments of physical aggression frequency were obtained from mothers at 17, 30, and 42 months after birth. Using a semiparametric, mixture model, distinct clusters of physical aggression trajectories were identified. Multivariate logit regression analysis was then used to identify which family and child characteristics, before 5 months of age, predict individuals on a high-level physical aggression trajectory from 17 to 42 months after birth. RESULTS: Three trajectories of physical aggression were identified. The first was composed of children who displayed little or no physical aggression. These individuals were estimated to account for approximately 28% of the sample. The largest group, estimated at approximately 58% of the sample, followed a rising trajectory of modest aggression. Finally, a group, estimated to comprise approximately 14% of the sample, followed a rising trajectory of high physical aggression. Best predictors before or at birth of the high physical aggression trajectory group, controlling for the levels of the other risk factors, were having young siblings (odds ratio [OR]: 4.00; confidence interval [CI]: 2.2-7.4), mothers with high levels of antisocial behavior before the end of high school (OR: 3.1; CI: 1.1-8.6), mothers who started having children early (OR: 3.1; CI: 1.4-6.8), families with low income (OR: 2.6; CI: 1.3-5.2), and mothers who smoked during pregnancy (OR: 2.2; CI: 1.1-4.1). Best predictors at 5 months of age were mothers' coercive parenting behavior (OR: 2.3; CI: 1.1-4.7) and family dysfunction (OR: 2.2; CI: 1.2-4.1). The OR for a high-aggression trajectory was 10.9 for children whose mother reported both high levels of antisocial behavior and early childbearing. CONCLUSIONS: Most children have initiated the use of physical aggression during infancy, and most will learn to use alternatives in the following years before they enter primary school. Humans seem to learn to regulate the use of physical aggression during the preschool years. Those who do not, seem to be at highest risk of serious violent behavior during adolescence and adulthood. Results from the present study indicate that children who are at highest risk of not learning to regulate physical aggression in early childhood have mothers with a history of antisocial behavior during their school years, mothers who start childbearing early and who smoke during pregnancy, and parents who have low income and have serious problems living together. All of these variables are relatively easy to measure during pregnancy. Preventive interventions should target families with high-risk profiles on these variables. Experiments with such programs have shown long-term impacts on child abuse and child antisocial behavior. However, these impacts were not observed in families with physical violence. The problem may be that the prevention programs that were provided did not specifically target the parents' control over their physical aggression and their skills in teaching their infant not to be physically aggressive. Most intervention programs to prevent youth physical aggression have targeted school-age children. If children normally learn not to be physically aggressive during the preschool years, then one would expect that interventions that target infants who are at high risk of chronic physical aggression would have more of an impact than interventions 5 to 10 years later, when physical aggression has become a way of life.

Aggression↗

Alcohol promotes dopamine release in the human nucleus accumbens.

Microdialysis experiments in rodents indicate that ethanol promotes dopamine release predominantly in the nucleus accumbens, a phenomenon that is implicated in the reinforcing effects of drugs of abuse. The aim of the present study was to test the hypothesis in humans that an oral dose of ethanol would lead to dopamine release in the ventral striatum, including the nucleus accumbens. Six healthy subjects underwent two [(11)C]raclopride PET scans following either alcohol (1 ml/kg) in orange juice or orange juice alone. Subjective mood changes, heart rate, and blood-alcohol levels were monitored throughout the procedure. Personality traits were evaluated using the tridimensional personality questionnaire. PET images were co-registered with MRI and transformed into stereotaxic space. Statistical parametric maps of [(11)C]raclopride binding potential change were generated. There was a significant reduction in [(11)C]raclopride binding potential bilaterally in the ventral striatum/nucleus accumbens in the alcohol condition compared to the orange juice condition, indicative of increased extracellular dopamine. Moreover, the magnitude of the change in [(11)C]raclopride binding correlated with the alcohol-induced increase in heart rate, which is thought to be a marker of the psychostimulant effects of the drug, and with the personality dimension of impulsiveness. The present study is the first report that, in humans, alcohol promotes dopamine release in the brain, with a preferential effect in the ventral striatum. These findings support the hypothesis that mesolimbic dopamine activation is a common property of abused substances, possibly mediating their reinforcing effects.

Adult↗

Adolescent pain sensitivity is associated with cardiac autonomic function and blood pressure over 8 years.

Low pain sensitivity has been reported in hypertensive subjects as well in groups deemed to be at increased risk of development of the disorder. However, it is uncertain whether individual differences in pain sensitivity are associated prospectively with increases in blood pressure. In the current study, 24-hour blood pressure and heart rate variability were recorded in 110, 22-year-old men previously assessed at age 14 years for casual blood pressure and pain sensitivity (mechanical finger pressure). Significant correlations were observed between pain tolerance in 14-year-olds and current 24-hour systolic blood pressure (r=0.37, P<0.01) and diastolic blood pressure (r=0.36, P<0.01). Hierarchical multiple regression analyses indicated that information regarding pain tolerance improved prediction of systolic and diastolic blood pressure at age 22 years beyond that afforded by differences in blood pressure, parental history of hypertension, and body mass index at age 14 years. Similar analyses revealed that average pain sensitivity at age 14 was also associated with 24-hour high-frequency heart rate variability (r=0.28, P<0.01) and low-frequency/high-frequency heart rate variability at age 22 (r=-0.35, P<0.01), suggesting increased sympathetic and reduced parasympathetic tone among individuals less sensitive to pain. These results provide further evidence that blood pressure related hypoalgesia might be related to processes involved in blood pressure regulation as well as in the development of sustained high blood pressure.

Adolescent↗

Developmental trajectories of boys' delinquent group membership and facilitation of violent behaviors during adolescence.

Being part of a delinquent group has been shown to facilitate the expression of an individual's own delinquent propensities. However, this facilitation effect has not been investigated from a developmental perspective within a population heterogeneity model. Using a semiparametric mixture model with data from the Montreal Longitudinal Experimental Study, this article addresses important issues in the developmental trends of membership to delinquent groups. We explore how the rate of violent behaviors follows delinquent peer group trajectories and investigate a differential facilitation effect of delinquent peers on violence across multiple developmental pathways. Results suggest that 25% of males followed a childhood or an adolescence delinquent group affiliation trajectory. These two groups account far most of the violent acts assessed during adolescence. In addition, the rate of violent behaviors follows these developmental trajectories. Controlling for these delinquent group trajectories, we also found that being involved in a delinquent group at any specific time during adolescence is associated with an increased rate of violent behaviors, and that leaving these groups results in a decrease in violent behaviors. This facilitation effect appears homogeneous over time and across developmental trajectories. Results are discussed from a social interactional perspective.

Adolescent↗

Life course turning points: the effect of grade retention on physical aggression.

Our objective is to advance the life course analytical framework by demonstrating a model for testing two of its tenets. The first is whether the individual's developmental history conditions the response to a turning point event. The second is whether the influence of a major life event upon an individual's developmental course depends upon the timing of the event. We test both propositions in an analysis of the effect of grade retention on a child's trajectory of physical aggression. Our analysis is based on data from a longitudinal study of 1,037 boysfrom schools in the lowest socioeconomic areas in Montreal, Canada. We find clear evidence that a developmental history of physical aggression conditions the child's response to grade retention. The evidence on whether the timing of retention affects this response is less clear.

Adolescent↗

Developmental trajectories of childhood disruptive behaviors and adolescent delinquency: a six-site, cross-national study.

This study used data from 6 sites and 3 countries to examine the developmental course of physical aggression in childhood and to analyze its linkage to violent and nonviolent offending outcomes in adolescence. The results indicate that among boys there is continuity in problem behavior from childhood to adolescence and that such continuity is especially acute when early problem behavior takes the form of physical aggression. Chronic physical aggression during the elementary school years specifically increases the risk for continued physical violence as well as other nonviolent forms of delinquency during adolescence. However, this conclusion is reserved primarily for boys, because the results indicate no clear linkage between childhood physical aggression and adolescent offending among female samples despite notable similarities across male and female samples in the developmental course of physical aggression in childhood.

Adolescent↗

Exaggerated ethanol-induced cardiac reactivity as an indicator of increased risk for gambling.

Pathological gambling and alcohol dependence show a high rate of co-occurrence. Some individuals at risk for alcohol dependence display an exaggerated heart rate (HR) increase following alcohol consumption, a characteristic suggesting sensitivity to reward. This study examined whether exaggerated ethanol-induced cardiac reactivity was associated with increased gambling behaviors. One hundred five young men (M = 20.13 years, SD = 1.07) consumed 1 ml of ethanol (95% volume) per kilogram of body weight. HR was measured and participants completed the South Oaks Gambling Screen (SOGS; H. R. Lesieur & S. B. Blume, 1987). Those with higher intoxicated HRs reported significantly greater scores on the SOGS (p = .02). This suggests that ethanol-induced HR increase is a possible marker for addictive disorders.

Adult↗