[Letter: Successful surgical treatment of type A Wolff-Parkinson-White syndrome].
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Biomedical subjects
Publications and source records attributed to R Slama.
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The external ruptures of the left ventricule occuring in the first twelve postoperative hours and demonstrated by a disastrous haemorrhage or cardiac failure, the other traumatics accidents could be delayed and be observed during the first month, or even later. Essentially two mechanisms are implicated: -- Direct traumatism by a ball valve on the ventricular septrum. -- Sub-annular splitting in connection with the excision of a piece of calcareous valve. Two lines of approch can be offered to avoid these accidents: -- Choose a disc valve when the surgeon finds a small left ventricle below a stenosed and calcified mitral valve. -- Care in the decalcification of the mitral ring and the valvular resection, leaving in case of need a small calcified cuff for safety. The analysis of these findings clarify the main causes of left ventricular rupture after mitral replacement, rare complications, but ones which are very real.
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The interest of permanent electrocardiographic recording in ambulatory patients is not restricted to the demonstration of the cardiogenic origin of some functional disturbances. This method provides the proof of the often non fiable character of anamnesis for the diagnosis of arrhythmias. It shows the close relationship between "minor" (extra-systoles) and "major" (tachycardias) arrhythmias, and consequently the prognostic value of the former in relation with the latter. Continuous ECG follow-up affords in the assessment of anti-arrhythmic drugs efficiency a higher safety than that provided by the conventional simple clinical follow-up.
Report of one unhabitual case of reciprocal rhythm with anterograde conduction through James' fibres and retrograde conduction through the normal nodal pathways. The reality of an anterograde nodal short-circuit is indicated by many arguments, and in particular by the presence of a double ventricular response, after induced atrial premature beats reaching the ventricle once by the rapid pathway and the second time by the slow pathway.
Correlation between the endocardial electrocardiographic recording and the histological findings of the conduction pathways in one case of apparent atrial standstill makes it possible to state the following facts: 1. The presence of a supranodal block through an intraatrial conduction disturbance. 2. The presence of James' fibres by-passing the upper part of Tawara node. 3. The functional character of these James' fivres which constitute, in view of the presence of lesions of the upper part of the Tawara node, the only possible pathway from the atria to the ventricles in this case. 4. The possibility to record the action potentials of James' fibres in that exceptional case. The histological examination made it possible also to recognize the anatomical lesions responsible for the idiopathic dilatation of the right atrium and of the apparent atrial standstill which represented the end-stage of an atrial rhythm disease.
The emergency treatment of septal perforation in a myocardial infarction was markedly improved by pre-operative preparation of the patient by an intra-aortic balloon. If one excludes 7 cases in which the favourable immediate course made possible a delayed operation, 21 cases, severe from the start, were observed in the last two years: 8 patients, were thought to be operable with 6 successes. The type of the patient, the age, the associated diseases, the gravity and extension of the local lesions explain that this complication nevertheless still remains very often beyond the present possibilities of the medical-surgical treatment.
Stimulation of the bundle of His and of the uppermost portion of the interventricular septum gives us an opportunity to make a precise study of capture phenomena in patients with paroxysmal nodal tachycardia. According to whether the capture is correctly timed, delayed, or unusually premature, the inferior junction point of the reentry circuit can be located precisely by reference to the H wave and the onset of the R wave. Out of a series of 65 patients, only 30 of whom had a true WPW syndrome, it was shown that 43 cases had a bundle of Kent which ensured retrograde conduction during the tachycardia, and was therefore the seat of a unidirectional block in 13 cases. In 22 cases (33.8%) the diagnosis of WPW syndrome was excluded, but the reentry circuit was nevertheless not of nodal origin. The inferior junction point of the circuit was effectively situated between H and R in 12 cases, and at H in 5. In only 5 cases (7.8%) might there have been a reciprocal intra-nodal rhythm, which should not necessarily be taken as proof of its existence. The validity of the classical criteria in localising the reentry circuit is discussed.
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