Biomedical subjects
R Salcido
Publications and source records attributed to R Salcido.
Recurrent traumatic brain injury.
This article reviews, analyses and provides commentary on the most recent literature concerning recurrent traumatic brain injury (RTBI) case histories. It is revealed that a significant proportion of TBI sufferers survive and recover most of the functions of daily living. However, relatively little is known about the epidemiology, effects, or causes of a new phenomenon: recurrent TBI.
The postacute continuum: consumer participation & outcomes research.
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Cost-effectiveness of subacute rehab.
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Providing rehabilitation care in rural areas.
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Rehabilitation outcomes in subacute care.
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Mild traumatic brain injury.
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Certification and competency in the year 2000 ... and beyond.
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Complementary and alternative medicine in wound healing.
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The future of wound measurement.
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Enzymatic debridement: a tried and tested method.
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Jill Kinmont Boothe: the obstacles and triumphs of living with an SCI.
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Good wound care: what is it?
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The language of wound care: taxonomy of discourse.
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Evaluation of ibuprofen for pressure ulcer prevention: application of a rat pressure ulcer model.
This blinded study describes the effect of ibuprofen (IBU) on experimental pressure ulcers (PU) induced in the fuzzy rat model. Ibuprofen's fibrinolytic activity has been found effective in preserving dermal vasculature in rats following burn injury. Experimental pressure ulcers were generated on the hips of fuzzy rats with the aid of computer control, using five daily, 6-hour pressure sessions. In the first study, 17 rats received intraperitoneal (I.P.) injection of IBU or saline control after each pressure session. In the second study, 44 rats received IBU or control, before, during, and after application of pressure by I.P. or intramuscular (I.M.) injections. Quantitation of histology is based on five prominent lesions, including: ulceration, infarction, panniculus carnosus necrosis, fat atrophy, and deep muscle necrosis. The following hypothesis was tested: Reperfusion injury and vascular occlusion in pressure ulcers may be similar to that which occurs in burns and, therefore, is responsive to IBU intervention. The results indicate that IBU does not have significant effects on the development of pressure ulcers. The mechanisms of IBU action in other organ and tissue systems are discussed in relation to the effect of IBU on the pressure ulcer model.
Where's the evidence for HCFA's policy?
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