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Biomedical subjects

R Matsuo

Publications and source records attributed to R Matsuo.

At least 109 records · Page 6Linked to original sources
PubMed1980

Corticofugal effects on the activity of thalamic taste cells.

To examine corticofugal influences on afferent thalamic cell responses in the gustatory system, effects of cortical conditioning stimuli on the responses of thalamic taste cells to peripheral stimulation were examined in the rat. Two types of excitability change of thalamic cells were observed; one is inhibitory (for about 60 msec) (20%) and the other is inhibitory (for about 10 msec)--facilitatory (for about 60 msec) (40%) to conditioning stimulus applied to the ventral gustatory area, and half of the cells belonging to the latter type also showed a similar pattern with more marked and prolonged excitability changes to conditioning stimulus applied to the dorsal gustatory area. It was suggested that some of the response characteristics of thalamic and cortical taste cells were attributed to the corticofugal feedback loop.

Afferent Pathways↗
PubMedDate not supplied

A triple coaxial catheter system for subselective visceral artery catheterization and embolization: preliminary clinical experience.

We developed a triple coaxial catheter system (TCCS) which consists of a 6.1 Fr outer, a 4.1 Fr intermediate, and a 3.0 Fr inner catheter, all coated with a lubricant. The TCCS was used in seven patients with hepatic tumors, after several attempts to access a targeted vessel with conventional catheters and guidewires failed to reach the targeted vessel. No complications were encountered with the use of this system. TCCS may be a useful device for selective abdominal arteriography.

Aged↗
PubMedDate not supplied

Spontaneous regression of hepatocellular carcinoma--a case report.

We report a 72-year-old man with hepatocellular carcinoma, which showed spontaneous regression. He was diagnosed as having chronic hepatitis type C five years before admission. In January 1998, a liver mass was found by ultrasonography. In February, computed tomography showed a low-density mass, 3.5 cm in diameter in the S5 region. Although liver biopsy was not performed, findings obtained by computed tomography and ultrasonography indicated that the tumor was hepatocellular carcinoma. The levels of alpha-fetoprotein and PIVKA (protein induced by vitamin K antagonist)-II were increased to 1000 ng/mL and 2000 mAU/mL, respectively. The patient was admitted to our hospital in March 1998. At the time, the size of liver mass was reduced to 2.5 cm in diameter on computed tomography, and the tumor markers, alpha-fetoprotein and PIVKA-II, spontaneously decreased to the normal range. We considered that hepatocellular carcinoma of this patient regressed spontaneously. Because it was hard to exclude the possibility that the mass contained residual malignant cells, we resected the mass on April 28, 1998. Microscopically, the resected mass did not contain any malignant cells. The parenchyma surrounding tumor necrosis, which is reflected by severe inflammatory infiltration with lymphocytes, indicates spontaneous regression. Although the precise mechanism regarding spontaneous regression of hepatocellular carcinoma is not fully understood, either ischemia due to rapid growth of the tumor or some inflammatory mechanism may be involved in regression of hepatocellular carcinoma.

Aged↗