Health care is plugging in new relationships and data connections are the future.
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Capitation Fee↗
Biomedical subjects
R L Reece
Publications and source records attributed to R L Reece.
Standardized interpretive reporting of clinical laboratory results.
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Chemistry, Clinical↗
Impact of managed care on physician organizational behavior.
This article examines how physicians act, react, and organize when managed care forces them to consolidate into larger groups and business corporations. Physicians have experimented with ownership by hospitals or business corporations to gain capital, management skills, and information systems. Now they're moving toward physician-owned groups with "outsourcing" of administrative and information system functions. The mood, movement, and momentum of physicians, in short, is toward integrated physician organizations bound together by information that amplifies on their core competencies and capacities to deliver care.
Career Mobility↗
Newspapers foretell health care's future.
This article is based on a two-months snapshot (November 1998 to January 1999) of newspaper articles addressing various health care issues. Newspaper contents reflect the changing market share of competing societal concerns. Health care issues, particularly cost and choice, now preoccupy the American people. Health care trends percolate bottom-up through the pages of newspapers, not top-down from Washington, D.C, policymakers, or health care executives. By reviewing these articles, the author provides a big picture view of the prevailing and emerging health care trends. From the new thrust of consumerism and the public backlash against managed care organizations to the demise of HMOs and PPMCs, these observations signify not only the concerns that are bubbling to the surface but also the direction that health care is headed. Consumers are in the driver's seat and physician executives need to provide them with evidence of the value they desire--and understand what they perceive as value.
Choice Behavior↗
Experimental factors affecting mortality following inoculation of chickens with avian nephritis virus (G-4260).
Groups of approximately 20 one-day-old chickens were inoculated with G-4260, the reference strain of avian nephritis virus (ANV), or saline. Based on mortality rates from severe nephritis in comparable experiments, light Sussex chickens generally were more susceptible than Rhode Island red (RIR) chickens. Mortality was greater in those given broiler starter than those given other feeds, and was greater when light Sussex chickens were given broiler starter feed and cold-stressed at 15 +/- 1 C for 2 hr daily during the first week rather than brooded normally. Inoculation with G-4260 either orally or by intraperitoneal injection produced similar results in RIR chickens. Thirty-three inoculated chickens died of severe nephritis between 4 and 12 days postinoculation, and 24 (73%) of them had visceral urate deposits. Inoculated inbred white leghorn Line 15 chickens with maternal antibody to ANV were brooded normally and given broiler feed: they were susceptible to infection as evidenced by subsequent histological lesions in the kidneys and serology, but mortality was not a feature. There were no deaths from nephritis in inoculated non-inbred white leghorn chickens free of maternal antibody to ANV that were given broiler feed and brooded normally. These results have implications in standardizing experimental conditions for the study of mortality induced by G-4260 and similar viruses.
Animal Feed↗
Absence of a role for selenium deficiency in the runting syndrome of broiler chickens in Australia.
The role of selenium deficiency in the etiology of the runting-stunting syndrome (RSS) of broiler chickens in Australia was investigated. Commercial broiler chickens maintained on selenium-deficient developed signs consistent with selenium deficiency of exudative diathesis and markedly reduced plasma glutathione peroxidase activity, but they did not develop pancreatic atrophy and fibrosis or elevated plasma amylase activity, which are the other lesions associated with RSS. Supplementation of the diets of birds from a RSS-susceptible flock with a mixture of selenium, vitamin E, cysteine, and sulfate had no effect on the incidence of runting in the treated birds. In field outbreaks of RSS there were no observable differences between affected and unaffected birds in the concentration of selenium in tissue samples. Furthermore, evidence is presented which suggests that in cases of RSS, pancreatic atrophy and elevations in plasma amylase precede reductions in plasma glutathione peroxidase activity.
Aging↗
An unusual case of inclusion body hepatitis in a cockerel.
This report describes inclusion body hepatitis in a 12-week-old leghorn-type cockerel. The cockerel had come from a specific-pathogen-free flock and was reared in a positive-pressure isolator until 6 weeks old. Grossly, the cockerel was jaundiced, and the liver was swollen and had small white foci throughout. Histologically, there were foci of hepatic necrosis and basophilic inclusion bodies in the nuclei of numerous hepatocytes. A fowl adenovirus, isolated from the liver, was found to belong to serotype 2/12. The bursa of Fabricius had some degenerative changes, but these were not consistent with infectious bursal disease, nor was there evidence of seroconversion to infectious bursal disease virus in the remaining chickens.
Adenoviridae Infections↗
Investigations of toxicity episodes involving chemotherapeutic agents in Victorian poultry and pigeons.
This series of case reports details observations on toxicity episodes in poultry due to a variety of chemotherapeutic agents. These problems arose owing to overdosage, variation in species susceptibility, potentiation of the toxic effects of one substance by the presence of another substance, and particular disease or other on-farm factors. Ignorance and accident were responsible for some of these situations. The episodes involved monensin, salinomycin, nicarbazin, sulphaquinoxaline, dinitolmide, dimetridazole, nitrofurans, streptomycin, and 3-nitro-4-hydroxyphenylarsonic acid.
Animals↗