Biomedical subjects
Publications and source records attributed to R Kent.
Myelolipoma of the adrenal gland is a rare, benign tumor of which only 17 clinical cases have been reported previously in the literature. We document 1 additional case and review the pathogenesis of the tumor, with emphasis on the theory of the tissue necrosis as the major stimulus of the myelolipomatous changes in the adrenals.
This report presents cinefluorographic data on the articulation of isolated vowels, VCV nonsense utterances, and short sentences by five subjects with athetoid cerebral palsy. Articulatory abnormalities were identified from tracings of vocal tract shapes and from displacement-by-time plots of articulatory events. The most frequent abnormalities were large ranges of jaw movement, inappropriate positioning of the tongue for various phonetic segments (especially because of a reduced range of tongue movement in the anteroposterior dimension), intermittency of velopharyngeal closure caused by an instability of velar elevation, prolonged transition times for articulatory movements, and retrusion of the lower lip. The speech disorder associated with athetosis is considered with respect to a model of motor learning.
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Cineradiographic and spectrographic analyses were performed to study the speech production of a subject who presented the classical neurologic signs of cerebellar lesion and who had speech characteristics like those that have been reported for ataxic dysarthria. These analyses were conducted with special attention to the deviant perceptual dimensions that have been described for ataxic speech. Examination of the cineradiographic and spectrographic records revealed conspicuous abnormalities in speaking rate, stress patterns, articulatory placements for both vowels and consonants, velocities of articulator movements, and fundamental frequency contours. In general, our physiological and acoustic observations of ataxic dysarthria were compatible with existing perceptual descriptions of this condition. The data for the subject are discussed in the light of current hypotheses concerning cerebellar participation in the regulation of skilled movement. Particular suggestions are made concerning the nature of the neuromuscular abnormalities that may underlie the aberrant motorics of ataxic dysarthria.
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