Biomedical subjects
Publications and source records attributed to R J Spinner.
We present a patient with translocation (snapping) of a portion of the triceps over the lateral epicondyle with elbow flexion. This condition is in many ways analogous to its counterpart at the medial aspect of the elbow, snapping of the medial head of the triceps, a clinical entity that is being increasingly recognized.
Five patients with cubitus varus deformities from malunited childhood fractures had dislocation (snapping) of both the medial portion of the triceps and the ulnar nerve over the medial epicondyle. In addition to snapping, these patients had medial elbow pain or ulnar nerve symptoms. Cubitus varus shifts the line of pull of the triceps more medial, which can cause anteromedial displacement of the medial portion of the triceps during elbow flexion. The ulnar nerve is concomitantly pushed or pulled anteromedially by the triceps, and ulnar neuropathy may result from friction neuritis or from dynamic compression by the triceps against the epicondyle. Recognition of both the dislocating ulnar nerve and the snapping medial triceps is crucial in the successful treatment of this pathologic finding. In symptomatic individuals, we recommend either corrective valgus osteotomy of the distal humerus or partial excision or lateral transposition of the snapping medial triceps, or a combination of both. Alternatively, medial epicondylectomy can also eliminate the snapping. Transposition of the ulnar nerve can be performed for ulnar nerve symptoms and/or ulnar nerve instability. Using this approach, correction of the snapping and/or ulnar nerve symptoms was achieved in all cases.
We describe a patient with a preexisting posttraumatic brachial plexopathy who had a complete high median nerve palsy due to rupture of the pectoralis major to biceps transfer near its distal insertion at the elbow region.
We describe two patients who had episodic elbow snapping and ulnar nerve dysesthesias only after weightlifting. These symptoms would disappear soon afterward. The episodic nature of their complaints and findings led to misdiagnosis. We documented by repeated clinical examinations and magnetic resonance imaging that the presence of these symptoms correlated directly with the finding of intermittent, activity-related snapping of the medial triceps. In both patients, the symptoms disappeared when the medial portion of the triceps migrated medially but did not dislocate over the medial epicondyle with elbow flexion. Thus, a minor change in the configuration of the medial portion of the triceps (fluid accumulation) in the same individual at different times can cause intermittent dislocation of the medial triceps. Previous papers dealing with patients with snapping of the medial triceps describe symptoms exacerbated by athletic activities, but the constant finding of snapping on sequential examinations.
Seven patients presented with an isolated extensor digitorum communis (EDC) palsy immediately after undergoing surgery in which the posterior (Thompson) approach to the proximal radius was used. All had normal neurologic examination findings documented prior to surgery. In an attempt to localize this lesion, the authors studied the arborization of the terminal motor branches of the posterior interosseous nerve (PIN) at the distal edge of the supinator. A common innervation pattern to the superficial extensor muscles was observed in 29 of 30 cadaveric limbs. In 10 of 10 specimens, when the EDC was subdivided into its individual bellies, a reproducible pattern emerged: the proximal EDC muscles of the middle and ring fingers were supplied primarily by the recurrent nerve branch(es) and the EDC muscles of the index and little fingers, by separate nerve branches. Consistent with our anatomic findings, perioperative stimulation of the recurrent branch in 1 neurologically intact patient resulted in middle and ring finger extension. Electromyography in 8 normal limbs showed that the middle and ring fingers could be activated together without the index and little fingers in all cases. We believe that these patients with isolated EDC nerve palsy may have sustained an iatrogenic injury to EDC motor branches, distal to the supinator rather than to a PIN fascicle near the proximal supinator.
We describe seventeen patients (twenty-two limbs) who had snapping (dislocation) of both the ulnar nerve and the medial head of the triceps over the medial epicondyle. Two patients (two limbs) were seen because of painless snapping, four patients (five limbs) had snapping and pain in the medial aspect of the elbow, three patients (three limbs) had symptoms related to the ulnar nerve only, and six patients (seven limbs) had snapping and symptoms related to the ulnar nerve. In addition, snapping was identified incidentally on routine screening in five asymptomatic limbs in four patients, one of whom was seen because of snapping and symptoms related to the ulnar nerve on the contralateral side. The diagnosis was confirmed with magnetic resonance imaging or computerized tomography, or both, in all but the first three patients, in whom the operative findings were confirmatory. Only six patients (seven limbs) were sufficiently symptomatic to be managed operatively. Of these six patients, five (six limbs) who had symptoms related to the ulnar nerve had lateral transposition or excision of the dislocating medial head of the triceps in addition to decompression and transposition of the ulnar nerve. Two of these patients had had persistent symptoms immediately after a previous transfer of the ulnar nerve performed at another institution for symptoms related to, and well documented dislocation of, the ulnar nerve; we performed the index procedure to correct the postoperative snapping, which was the result of an unrecognized dislocation of the medial head of the triceps in one patient and the result of an accessory triceps tendon in the other. One patient who had pain in the medial part of the elbow, snapping (without symptoms related to the ulnar nerve), and cubitus varus had a valgus osteotomy of the distal aspect of the humerus that corrected the line of pull of the triceps and relieved the snapping. All of the patients who were managed operatively had an excellent result (no snapping, no symptoms related to the ulnar nerve, and a full range of motion), at an average of 4.5 years postoperatively. Non-operative treatment provided control of symptoms related to the ulnar nerve in four limbs and control of pain from the snapping in four limbs. Snapping on the medial side of the elbow, even if it is associated with symptoms related to the ulnar nerve, is not necessarily caused by dislocation of the ulnar nerve alone. Patients who have a transposition of the ulnar nerve, especially those who have dislocation of the ulnar nerve, should be examined intraoperatively with the elbow in flexion and extension so that the surgeon can be certain that the medial head of the triceps does not snap over the medial epicondyle. Failure to recognize concurrent dislocation of the ulnar nerve and the medial head of the triceps can result in persistent, symptomatic snapping after an otherwise successful transposition of the ulnar nerve.
This 4 1-year-old man presented with a 2-year history of symptoms and signs of sciatic nerve compression. Imaging studies revealed a large ossified fragment within the biceps muscle of the thigh abutting the sciatic nerve at the level of the lesser trochanter. The bony fragment resulted from an unrecognized apophyseal avulsion fracture of the ischial tuberosity, which the patient had sustained while sprinting 27 years earlier. External neurolysis of the sciatic nerve and excision of the mass led to a successful outcome.
Variations in the medial triceps in conjunction with bilateral ulnar neuropathy have been identified in three generations of one family also possessing the phenotype of Waardenburg syndrome (a rare autosomal-dominant disorder with clinical features including cochlear deafness, dystopia canthorum, and pigmentation problems). To our knowledge, no other inherited condition with triceps anomalies has been reported. Study of this family provided insight into the relationship between dislocating medial triceps and ulnar neuropathy and demonstrated that a broad spectrum of clinical presentations exists-from being completely asymptomatic to producing symptomatic snapping and ulnar neuropathy.
A partial posterior interosseous nerve (PIN) paralysis developed in a patient more than 2 decades after having childhood osteomyelitis of the metaphysis of the radius. In previous reports of tardy PIN palsy, the radial head was dislocated (frequently in cases of unrecognized Monteggia fracture-dislocations), causing compression at the arcade of Frohse. In the patient whose case is discussed here, the radial head was located; compression of the PIN at the arcade of Frohse, in combination with traction against inflamed periosteum, likely resulted in the late development of clinical symptoms and signs.
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A biomechanical study compared the mechanical properties of hand and craniofacial plating systems commonly used in proximal phalangeal fractures. Two plates of each of the various systems were mounted dorsally on a yellow-birch-dowel model of a proximal phalanx after a transverse cut was made in the middle of the section of the dowel, modeling a midshaft transverse osteotomy or fracture. Torsional rigidity, as well as four-point bending rigidity in apex dorsal, lateral and volar directions, was achieved. Failure testing in apex palmar four-point bending was then examined. Between plating systems, torsion varied 1,600% and results of apex palmar testing varied 1,500%. Apex palmar moment-to-failure testing varied 1,000% and represented a 3.5%-38% range of intact proximal phalangeal strength. This also represented 12%-128% of the maximum calculated in vivo bending moments of the proximal phalanx. The wide variation in plate strengths and stiffness raises questions as to the suitability of certain plating systems with regard to early mobilization. Moreover, some plating systems tested were mechanically weaker than the reported strengths of certain Kirschner wire fixation techniques.
A patient is reported with a lesion of the radial branch of the superficial radial nerve following opposition transfer. The ring finger flexor digitorum superficialis slip was responsible for the neural compression near its thumb insertion.
A patient presented with neurovascular symptoms due to penetration of a proper digital nerve by a common digital artery. To knowledge, our report is the first clinically relevant description of this underrecognized, but relatively common anatomic variation.
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Although uncommon, infection of prostheses with Mycobacterium tuberculosis can be managed successfully if it is diagnosed early and treated correctly. A case of M. tuberculosis infection of a prosthetic knee first diagnosed 4.5 years after initial arthroplasty is described. This case and a review of the literature led to the conclusion that there are two distinct patterns of M. tuberculosis infection following joint implant surgery in patients without a history of tuberculosis. (1) Mycobacterium tuberculosis infection may be an unexpected finding at the time of arthroplasty. These patients generally have favorable outcomes using standard antituberculous chemotherapy, without implant removal. (2) Late-onset M. tuberculosis joint infection may be identified in patients with painful, clinically infected, or malfunctioning prostheses. In these cases, medical treatment alone is usually unsuccessful; prosthesis removal is often required. With recent increases in the incidence of tuberculosis in the United States and the emergence of multidrug-resistant strains of M. tuberculosis, periprosthetic tuberculous infection is likely to become more common.
Two cases of suprascapular neuropathy after excision of the distal clavicle are reported. Both patients were treated successfully with neurolysis of the suprascapular nerve starting at the upper trunk of the brachial plexus. Anatomic dissections revealed that the suprascapular nerve is quite close (<1.4 cm) to the posterior aspect of the distal clavicle, within 2 to 3 cm of the acromioclavicular joint. To avoid the complication of suprascapular neuropathy that could be associated with this close relationship, it is recommended that no more than 1 cm of the distal clavicle be removed posteriorly. It is also recommended that minimal periosteal elevation should be performed on the posteroinferior border of the distal clavicle.
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